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The fight against cancer continues to wage, boosted by Harris’ announcement this week of a partnership with Cancer Treatment Services International. The partnership seeks to bring quality, cost-effective cancer treatments to patients across the globe. Founded by Pittsburgh-based physicians and healthcare professionals, CTSI’s provides treatments to patients in areas where cancer treatment is minimal or nonexistent.
Founded in May 2009, GovCon Executive is a news blog published by Executive Mosaic Media that covers the dedicated individuals who serve our government through private industry and the officials who affect the government contracting industry at the federal and state levels. Patients get state-of-the-art cancer therapy from a modern linear accelerator in the Stanford Comprehensive Cancer Center. The first patient to receive radiation therapy from the medical linear accelerator at Stanford was a 2-year-old boy. Henry Kaplan, left, and head of radiologic physics Mitchell Weissbluth, the first physicist Kaplan hired, at the working end of the Stanford accelerator. That patient, a 2-year-old boy suffering from a tumor in his eye, was the first to undergo X-ray treatment from a medical linear accelerator that Kaplan developed with campus physicists. Fifty years and 40 million patients later, medical linear accelerators have become the backbone of radiation therapy for cancer worldwide. In recognition of the 50th anniversary of the medical linear accelerator, the Department of Radiation Oncology is presenting a celebratory symposium on April 19.
The medical linear accelerator is a dramatic example of what can be achieved when researchers from different academic disciplines converge to solve a problem. It all came about because Kaplan thought he could find a way to focus intense X-rays to destroy a tumor without harming surrounding healthy tissues—and he thought the physicists would be the ones to help him do so.
An atom smasher, otherwise known as a particle accelerator, uses electromagnetic fields to propel charged particles to great energies. Kaplan and Ginzton, PhD, professor of electrical engineering and of physics, developed the first medical linear accelerator in the Western Hemisphere, installed at Stanford-Lane Hospital in San Francisco. In the years following this first patient treatment, Stanford radiologists continued to improve on the device's design, spurred by Kaplan's early ambitions of having his department's clinical science meld with the university's basic science departments of physics, engineering and biology.
Peter Fessenden, PhD, professor (teaching) of radiation oncology emeritus, arrived at Stanford as the head of the radiation physics section in 1972, just as the original accelerator was retired to the Smithsonian Institution after 16 years of patient treatment. A linear accelerator was co-developed at Stanford that can continually track the position of tumors in real time during treatment. Stanford also has dozens of clinical trials in progress investigating new ways to use radiotherapy for cancer treatment and immunosuppression. When you look at modern medical linear accelerators, said Hoppe, "they are light-years ahead of that unit built by the physicists at Stanford in the early 1950s." But what is in store for a cancer patient 50 years from now? The next generation of radiotherapy will encompass the techniques of molecular imaging, which allows doctors to differentiate tumor from normal tissue.
In recognition of the contributions that the medical linear accelerator has made to cancer treatment, the Department of Radiation Oncology is hosting a celebration, "A culture of innovation: Celebrating pioneering efforts of the past and envisioning the future," on April 19.
1956: The first medical linear accelerator in the Western Hemisphere is installed at Stanford Hospital in San Francisco. 1959: Stanford medical school and hospital move to the Palo Alto campus, bringing the medical linear accelerator. 1962: Kaplan and Saul Rosenberg begin trials using the linear accelerator with chemotherapy to treat Hodgkin's disease, an approach that dramatically improves patient survival.
1994: First use of the CyberKnife, invented at Stanford, which uses sophisticated computerized imaging to aim a narrow X-ray beam precisely.
1997: Stanford pioneers the use of intensity-modulated radiation therapy, which combines precise imaging with linear accelerators that deliver hundreds of thin beams of radiation from any angle.
2004: Implementation of four-dimensional radiotherapy, which accounts for the motion of breathing during imaging and radiation delivery.
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Successful Cancer surgery at We Care India partner hospital allows Henry Stolz to live a normal life despite a microendoscopic Decompression. Radiation therapy uses high-dose X-rays or other types of radiation to destroy bladder cancer cells. Internal (interstitial) radiation therapy (brachytherapy) uses radioactive material contained in tiny tubes, wires, or beads. Both men and women may have sexual problems following radiation therapy for bladder cancer. Radiation therapy may cause a decrease in your white blood cells (leukopenia), which increases your risk of developing an infection. This information is not designed to replace a physician's independent judgment about the appropriateness or risks of a procedure for a given patient. We Care Health Services - India a€“ Mumbai (Bombay), Chennai (Madras), Hyderabad, Kolkata, Delhi, Bangalore. Thomas Seyfried PhD, a brain cancer researcher with over 25 years of experience in the field, gave a groundbreaking presentation about cancer at the Ancestral Health Symposium held at Harvard Law School this past August.
After attending his lecture, which challenged everything I thought I knew about cancer, I was inspired to read his new book, entitled Cancer as a Metabolic Disease: On the Origin, Management, and Prevention of Cancer. I was taught in medical school that cancer is about genes going haywire—something evil comes along, like a toxic chemical or a beam of radiation, attacks your DNA, and poof—you’ve got cancer (unless you are a cartoon character, in which case you develop superpowers). The company line is that cancer is caused by mutations (changes) in DNA that transform healthy, well-behaved cells into reckless, ravenous, immortal renegades. Hundreds of thousands of different mutations have been discovered in cancerous cells, but it is actually rare to find genetic mutations in healthy cells because healthy cells have stable DNA. Yes, most tumor cells are growing faster than most of their healthy neighboring cells, but this is not because they are speedier.
Just because cancer cells have lots of mutations doesn’t necessarily mean that mutations cause cancer. A huge variety of things in the world—from viruses to radiation to chemicals to oxidation—can damage DNA and cause mutations.


The transformation of a healthy cell into a cancerous cell (malignant transformation) happens in the very same specific way every time. How can all of these different and unpredictable events leading to all of those random mutations always cause exactly the same outcome? Mutated genes thought to be strongly associated with cancer (“oncogenes”) sometimes do promote tumor growth, but sometimes they inhibit tumor growth, and sometimes they even do both. Here’s the thing: if you transplant mutated cancer cell DNA into a healthy cell, the healthy cell almost never becomes cancerous.
Just think about it: if cancer is a genetic disease, based on hundreds of thousands of mutations, what are we supposed to do, create hundreds of thousands of different drugs to treat it? Seyfried argues that the reason why we are making so little progress is because we are fighting the wrong enemy.
In the next article in the series, What Causes Cancer: Part II, I explain the role of mitochondria in our cells and the significant link between damaged mitochondria and cancer. Sign up to be notified of my latest posts!Signup now and receive an email once I publish new content. Tripping Over the Truth: The Return of the Metabolic Theory of Cancer Illuminates a New and Hopeful Path to a Cure  by Travis Christofferson. In it, he says “cancer is a metabolic mitochondrial disease caused by multiple different things in the environment damaging respiration. I read an interview with Loren Cordain (author of The Paleo Diet), who cites an obscure publication on autopsies of centuries-old Inuit mummies—who apparently ate diets of 100% meat or extremely close to it—that showed that they had significant atherosclerosis. I understand that your body does not tolerate fruit well, and you do seem to agree with me that fruit is generally healthier than other plant parts. But why is it that regardless of what you do, once you’ve metastasized, you almost always die of the cancer? I’m sorry to hear that you are dealing with cancer, and sincerely hope that you will be able to find a nutritional approach that will help the body to remove cancer cells. Hello Vanessa, I just wanted to let you know that I have written an eBook on implementing a ketogenic diet to treat cancer that you may find helpful. I wish I knew ersonally of clinicians who could help you; this is the most common question I get from readers. I have been a fan of Thomas for a few years, interviewing him and writing about his theories in papers for graduate school. Thank you very much for taking the time to both read the synopsis and to tell me what you thought of it. So many readers have written in to ask me if I know of physicians who use ketogenic diets to treat cancer and I have been at a loss to guide them in this regard.
Having looked at your Parts 1-4 of Thomas Seyfried’s work, to greatly simply this, is it too simple to say or addiction to simple sugar, the sugar in beer and social alcohol beverages, and no exercise to burn this sugar up, is killing us?
Thank you so much for being a truthful, generous and sincere person, willing to help others for no financial benefit. Harris will provide technology for CTSI’s oncology clinics and work to develop healthcare IT and clinical solutions.
The accelerator, which was developed at Stanford, will celebrate its 50th anniversary at an April 19 symposium.
The treatment saved the child's sight and he lived the rest of his life with his vision intact.
Roughly half of all cancer patients receive radiation therapy, primarily from the rays generated by a linear accelerator. Its radiation can quell the rejection of an organ transplant, suppress the immune systems of patients undergoing blood and marrow transplantation, and correct certain neurological and cardiovascular disorders. This 50-year-old achievement was a precursor to the multidisciplinary approach now called translational medicine. The interview appeared in The World of Stanford Radiology, a 2006 book by Otha Linton that chronicled the 100-year history of radiology at the School of Medicine.
By slamming accelerated electrons into a target made of heavy metal, high-energy X-ray beams result. In January 1956 the machine was ready to be used on their first patient, a boy with retinoblastoma in his one remaining eye after surgeons had removed the tumor in the other eye.
Fessenden continued studies he had begun years earlier with the section's previous leader, exploring how to create linear accelerators that delivered their punch to tumor cells through two types of radiation. The heart beating, the lungs inflating and deflating with each breath, the blood flowing through the veins, subtle movements of the skeleton or muscles—all this motion adds up to a moving target for a beam of deadly radiation.
The radiation oncology group has a long history of exploring other sources of radiation instead of X-rays and electrons—such as negative pi mesons, the subatomic particles that Fessenden studied. The invitation-only event will include a symposium, tours of the Stanford Comprehensive Cancer Center, a poster session and reception. It may be used alone, before surgery to shrink the cancer, or after surgery to destroy any remaining cancer cells. It is more effective when it is used in combination with chemotherapy than when it is used alone.
Your doctor will monitor your blood counts regularly and may change your treatment schedule until your white blood cell count returns to normal. In it he lays out 400+ pages of dense scientific evidence—complete with 1,740 references, and dozens of charts, graphs and full-color glossy images to make his case. These blueprints have to be flexible, because cells need different proteins under different circumstances. These mutations hijack the set of instructions encoded in the cells’ DNA, and scientists think these mutations cause cells to go wild. They grow independently, ignoring the anti-growth signals and death cues that would normally keep healthy cells from getting out of control.
DNA is the most important molecule in the body so evolution has made sure it is well-protected. It is, after all, the stability of healthy DNA that allows our cells to adapt to stressful environments.


Seyfried argues that mutations are just red herrings (no disrespect to the herring community intended). That’s like saying no matter how you attack an orc—whether you stab him in the belly with a sword, throw a rock at his head, or push him off a cliff—his left arm always falls off. Even those mutations most strongly associated with certain cancers only cause cancer in certain people. Only 2 out of 24 experiments were successful in transforming normal cells into cancer cells (and scientists couldn’t be sure that viral contamination wasn’t to blame). The mutation theory of cancer has been solidly in place and guiding research since 1981, yet despite the enormous amounts of money, time, and energy that have been poured into cancer research since, we continue to lose the war against this killer disease. It’s a small list, just under 5,000 people, but they are a committed group of people in many areas of life. Folks who adopt ketogenic diets, folks who become vegan, folks who do tons of cancer-fighting herbs under the care of a knowledgeable MD or herbalist, still almost always die of their cancer once they’ve metastasized, even though some of them are well managed for longer than with conventional approaches. Why would that be?And thanks in advance for answering as clearly as you presented your articles. Also he states cancerous cells might fuse with certain immune system ones and this might lead to the cancer spreading. Ede, I understand that you are not able to give medical advice, but could you direct me to someone who can in NYC. Kaplan thought this machine could be harnessed to deliver X-ray therapy that was superior to the unreliable, weak and unfocused radiation therapy approaches of his day. Destroying the tumor while sparing the eye would have been impossible with earlier, less-focused radiation sources.
Most people who receive external beam radiation therapy for bladder cancer are treated 5 days a week for 6 to 7 weeks as an outpatient.
Cells must be able to adapt to various conditions, such as stress, injury, infection, temperature changes, and food supply. This “genomic instability” is viewed as a strong suit by scientists who believe in the mutation theory. All the healthy cells around them are capable of growing just as fast, but there are checks and balances in place to prevent them from growing willy-nilly. Radiation and chemotherapy are toxic to all cells, cancerous or not, but they are more toxic to tumor cells.
Tumor cells are also more sensitive to heat (fever) and to starvation.  When the body is stressed, the tumor cells are the first ones to go. The sheer number and type of mutations found in cancer cells are so serious that they would cause a healthy embryo to spontaneously abort, yet cancer cells somehow soldier on. Also BRAC1 does not cause cancer by itself – but rather predisposes one for cancer because it inhibits the function of the mitochondria, and therefore the ability to generate energy through oxidative pathways.
Many of the current studies continue to refine how to account for the anatomical changes of a patient with time, from second-by-second to day-by-day, said Keall. The book is expensive, and would be nearly impossible to understand without a strong scientific background, but the secrets inside are so important that I wanted to make them available to everyone—they simply must be shared. So, genes contain lots of special controls that can be turned on and off, depending on what’s going on in and around the cell.
Cancer cells lose many of the physical features of their mother cells; they are usually smaller, and may be disfigured or even shapeless.
They think that the tumor cells keep mutating to improve themselves, and that the ones with the most clever mutations are the ones which survive best and reproduce best (Darwinism—survival of the fittest). If tumor cells were more robust than normal cells, these therapies would kill off all your healthy cells and only the big ugly tumor would survive. Researchers now place hope in the Cancer Genome Project, which they see as the shining future of cancer treatment. I started eating this way for weight loss, like most people, but the current wave of interest in keto eating and cancer (Eugene Fine, for example) has really been gratifying.
I very much appreciate your work and especially look forward to these next two installments! I suspect that the reason that Inuits get aterial plaque (even if it is not a threat to their health) is because of what I am assuming is a relatively high load of PUFA’s in their diet from fish and marine mammals. This series of articles is dedicated to my friends, family, colleagues, and patients who are battling cancer or who have loved ones suffering with cancer. There are even “caretaker genes” that are designed to maintain and repair defects in DNA, because lots of things in the natural environment can injure DNA—even things we think of as healthy, such as sunlight and vegetables. They think of cancerous cells as invincible—as stronger, faster, and smarter than healthy cells.
For example, when the liver is injured and healthy cells need to grow rapidly to replace the injured cells, their growth rate is the same as for liver cancer cells during tumor progression. Instead, people are treated to the brink of destruction with chemicals and radiation while doctors cross their fingers hoping more tumor cells will die than healthy cells, and that patients will survive the therapy.
They have already started using the genetic fingerprints of cancer to design expensive, high-tech drugs that specifically target the unique DNA pattern of individual cancer cells. Because the neurons have been killed, the glutamine now goes into the tumor cells and is fermented.
Some find this cure in an all vegan diet, an all raw diet, a 75% raw eating diet, some even a Paleo diet and of course this kind of diet. More than 700 of these smart bombs have been developed so far, yet none of them have saved a single life. The vast majority of whatever progress we have made against cancer has been due to identification of and education about lifestyle risk factors (such as smoking), not due to advances based on genetic theories.
So you have powerful glucose and powerful glutamine and together they wlll fuel the tumor.This is the demise of the cancer patient.



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