How to stop herpes virus from replicating,degree in mindbody medicine from harvard,energy saving in windows 7,herpes 1 how do you get it meaning - PDF Review

admin 13.02.2016

Harvey Simon, MD, Editor-in-Chief, Associate Professor of Medicine, Harvard Medical School; Physician, Massachusetts General Hospital. The information provided herein should not be used during any medical emergency or for the diagnosis or treatment of any medical condition.
We could just review the data presented by Genocea and share scientists’ opinion on herpes vaccine development in this article, but would herpes patients actually benefit from it? Quick recap: Genocea has just shared the data for the first 6 months of a 12-month research for a phase 2, and the results seem to be very promising.
When checked, Shutterstock's safe search screens restricted content and excludes it from your search results. The pathways researchers and doctors take to get to disorders like FM or chronic fatigue syndrome are nothing but diverse, and it’s worth taking a look at how Dr.
Pridgen saw a pattern emerge in his  treatment of thousands of patients with chronic gastrointestinal issues that intrigued him. Shorter and shorter relapses over time in his patients lead Pridgen to conclude that a virus must be involved.
The problem, he thought, had to be some sort of pathogen that was steadily increasing with every recurrence.
HSV-1 has been found in the esophagus, stomach and duodenum of the gastrointestinal system. Pridgen’s patent application indicates that he believes that stressors and  peptides and hormones released by the sympathetic nervous system and HPA axis  set the stage for herpes simplex-1 reactivation. Once  these neurons and ganglia are damaged, Pridgen believes they send out signals that ultimately muck up the pain processing centers in the central nervous system. One of Pridgen’s patent applications suggests that one of his unique insights has been to combine valacyclovir (valtrex) with an anti-inflammatory, Celecoxib (Celebrex) that has antiviral properties. Celexcoxib (Celebrex) is a non-steroidal antinflammatory (NSAID) COX-2 inhibitor usually used  in the treatment of osteoarthritis, rheumatoid arthritis, acute pain, painful menstruation and menstrual symptoms. Pridgen and Duffy are looking for herpes simplex virus, but other herpes viruses could be affected by this treatment.


Pridgen and his partner, molecular virologist, Carol Duffy will also attempt to develop a diagnostic test for fibromyalgia using cytokine arrays they believe will document high levels of  pro-inflammatory cytokines and low levels of anti-inflammatory cytokines.
Celebrex was shown to reduce stress induced herpes virus reactivation in the nervous systems of mice.  Another study found that reactivation of HSV-1 in mice was associated with upregulation of COX-2 gene expression in their nerve ganglia.
Along with treating the virus, Pridgen and his partner, molecular virologist Carol Duffy, will be using PCR to test for the virus, not in the blood, but in gut tissue samples.
One of the most intriguing aspects of the Pridgen-Duffy study is the search for HSV-1, not in the blood, but in the tissues.
Pridgen and Duffy’s big multi-center antiviral trial in Fibromyalgia is nothing if not exciting in its scope and approach.
If they results are positive, Pridgen and Duffy could usher in an entirely new way of treating both fibromyalgia and chronic fatigue syndrome. A licensed medical professional should be consulted for diagnosis and treatment of any and all medical conditions. The vaccine is actually an immunotherapy which was created to treat genital herpes and other viruses by inducing T cell and B cell (antibody) immune responses. The over-generation of neurotransmitters such as glutamate, Substance P, serotonin, norepinephrine, dopamine, brain-derived neurotrophic factor (BDNF) involved in this process then causes central sensitization. Pummeling the virus with that one-two punch, he believes, will finally stop the virus from reactivating. We know the Chronic Fatigue Initiative’s Pathogen study  failed to find evidence of viral infection in the blood. Then antibodies will be used to determine if an active infection is present.  In subsequent studies, electron microscopy will look for the herpesviruses particles themselves.
Pridgen reported in an email they are still trying to determine  the optimum doses and cautioned everyone to wait until the results of the phase three trial are done before starting this treatment. Pridgen’s ability to come up with over $3,000,000 in startup funding suggests he and Duffy have got some solid data backing their trial up. Links to other sites are provided for information only -- they do not constitute endorsements of those other sites.


We all hope that in the future herpes vaccine will be help minimize the spreading of herpes virus and bring the number of people who get infected each year down. Of course, it is more convenient that most common herpes treatments, but the vaccine can only minimize the viral shedding approximately by 50%, which means that 50% of the time it won’t stop the virus shedding. Then he found that adding an anti-inflammatory (which also had anti-viral properties) reduced their fatigue, gastrointestinal complaints, depression and anxiety markedly and improved their energy. Immunoblot testing indicated that an active infection was present in eight of nine positive biopises.
The last, seventh, group received placebo, and was rolled into an active study after 28 days. Some scientists believe that a number of days the virus sheds depends on a number of outbreaks per year. But we all know that many of FDA approved treatments still cause side affects even when they are considered to be safe.
And the number of outbreaks per year in many cases depends on immune system and the amount of stress. Genocea’s CEO Chip Clark said that herpes vaccine could make more than 1 billion in sales one day. However, many professionals are concerned about the fact that the lower dosage of herpes vaccine in µg hasn’t proven to be very effective. And the company still hasn’t confirmed whether the decrease in viral shedding after receiving the vaccine led to a decrease in a number of outbreaks a patient experienced per year.



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