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admin 18.02.2016

Oral herpes is easy to diagnose when a patient has visible sores or ulcers, but early stages of herpes on the face and mouth as well as on the genitals are harder to diagnose. Herpes should be distinguished from herpes zoster, chicken pox, impetigo, and drug eruptions.
Herpetic sores are classified as heat sores in TCM because the lesions are red and follow febrile disease conditions.
As with all treatment protocols, be sure to tailor and modify the basic treatments below to fit your patient's specific constitution, root cause, and symptoms. Treatment principle: Drain Spleen and Stomach damp heat, nourish yin, drain deficient heat . Cash, checks and credit cards are also gratefully accepted if you prefer to donate in person. Pain, emotional upheaval, fatigue, people who are burned out, folks who want to try esoteric acupuncture. Thirdly, this is an oversimplified but essentially accurate explanation of how several antiviral drugs work. A herpes simplex virus attaches to a susceptible host cell (seen on the right), fusing its envelope with the cell membrane, and releasing naked capsids that deliver viral DNA into the nucleus of the host cell, where it initiates the synthesis of viral DNA.
Acyclovir molecules entering the cell are converted to acyclovir monophosphate by virus induced thymidine kinase enzyme. Host-cell enzymes add two more phosphates to form acyclovir triphosphate, which is transported to the nucleus. Shown in red is the cleavage of phosphate from the acyclovir triphosphate by the herpes simplex's own enzymes, the herpes simplex's DNA polymerase enzyme incorporates the acyclovir monophosphate into the growing DNA strand as if it were 2-deoxyguanosine monophosphate (a "G" base). Further elongation of the chain is impossible because acyclovir monophosphate lacks the attachment point necessary for the insertion of any additional nucleotides.
During active disease phases there are blisters on the skin which contain the infectious virus. This is mainly due to the way the virus is transmitted between males and females.Herpes is transmitted from the sores which are caused by the virus, but additionally it is also transmitted from skin outbreaks which show no symptoms of the virus and do not appear to be broken or to have a herpes sore. The acyclovir (as do other antiviral drugs) is mistakenly incorportated into the growing DNA strand.


The acyclovir does not have the proper structure to add any additional "A"'s, or "G"'s, or "C"'s, or "T"'s to it, and so the DNA strand cannot continue to elongate or grow.
If I swallow my acyclovir drugs, isn't acyclovir going into all of my cells, and blocking the growth (or replication) of all my cell's DNA, not just the cells infected with virus? Accompanying symptoms may include high fever and swelling of tenderness of the lymph nodes. Crusting of the lesions begins after three to four days of onset and complete healing within three weeks. This means that even if you have no signs of a herpes outbreak, it is possible to infect your partner with the herpes virus.
Viruses are transported along the sensory nerves to the nerve cell bodies where they go dormant and reside as long as the host is alive. They may come out of dormancy because of a variety of triggers (one of which is immunosuppressant drugs). Therefore, only those cells that are infected with the virus will have this specific enzyme and convert the original acyclovir into the acyclovir that can be incorporated as a "G" base. When it becomes active the virus multiplies, travels along the axon of the neuron to the nerve terminals in the skin and releases the virus particles. Type 1 virus is the usual cause of cold sores around the mouth, and herpes simplex infection in the eye. It is commonly passed on by close contact such as kisses from a family member who has a cold sore.) In many people the primary infection does not cause any symptoms, although in some cases symptoms do occur. A minor and temporary inflammation of the conjunctiva (conjunctivitis) or eyelids (blepharitis) may occur with active infection, often at the same time as the cornea is infected.
Often people who get active eye infection will have had previous cold sores during their lifetime.
They may also put some stain on the front of your eye to show up any irregular areas on the cornea. If your doctor suspects a herpes eye infection you will usually be referred urgently to an eye specialist. This is to confirm the diagnosis and to determine whether the infection is in the top layer of the cornea (epithelial keratitis), or if the deeper layers are involved (stromal keratitis).


These do not kill the virus but stop it from multiplying further until the infection clears. You are likely to be kept under review, until the infection clears, to check that the cornea does not become infected.
Note: if you have herpes simplex eye infection, you should stop wearing any contact lens until 24 hours after your symptoms and the infection have completely gone away. As mentioned above, these occur if the virus reactivates from time to time - similar to cold sores. A recurrent infection may occur any time between a few weeks and many years after the first active infection. At least half of people who have one episode of active infection will have a recurrence within 10 years of the first. If recurrences are frequent or severe, then your eye specialist may advise that you take antiviral tablets each day to prevent episodes of active infection. Studies have shown that, on average, the number of recurrences is roughly halved in people who take regular antiviral tablets. Some people say that episodes of active herpes infection may be triggered by strong sunlight. It is also possible that active infection may be triggered if you are run down or unwell for another reason.
Some women find that they get recurrences around the time of their period but again there is limited evidence to support this. However, severe and recurrent herpes simplex eye infections may lead to serious scarring, impaired vision and even blindness in some cases.



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