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Por fuerte que pueda ser esta conexion seria prudente que mantenga sobrio a su sistema inmunitario y que lo arme con una dieta bien balanceada, mucho reposo y relajacion, y ejercicio regular. Para el maximo foralecimiento de su sistema inmunitario, comience con una taza de aceite de ricinoo, con el cual se empapan completamente dos pedazos de franela. El autor no es responsable del uso y consecuencia de su empleo, ya que este sitio solo proporciona orientacion. Environmental factors, such as viruses, exposure to chemicals, or sunlight, trigger inflammatory or immune activity. The exact combination of genes that predispose individuals to SLE may differ somewhat from patient to patient, but probably share certain common features which tend to impair the ability of the body to get rid of immune-triggering particles and which tend to prolong or increase the degree of immune responsiveness to these triggers. A major characteristic of lupus is that it is an autoimmune response in which immune factors, called autoantibodies, attack the person's own cells. When an injury or an infection occurs, white blood cells are mobilized to rid the body of any foreign proteins, such as a virus. The masses of blood cells that gather at the injured or infected site produce factors to fight any infections. Under normal conditions, the immune system has special factors that control and limit this inflammatory process. For reasons that are still not completely understood, both the T cells and B cells become overactive in lupus patients.
If overproduced, however, they can cause serious damage, including dangerous levels of inflammation and cellular injury.
SLE is a complex disorder and researchers are still in the early stages of unraveling how genetic factors may alter and affect the immune system. About 90% of lupus patients are women, most diagnosed when they are in their childbearing ages. African-Americans are three to four times more likely to develop the disease than Caucasians and to have severe complications. Rheumatoid arthritis is a systemic autoimmune disease that initially attacks the lining, or synovium, of the joints. In genetically susceptible people, there are various external factors that can provoke an immune response. Premature menopause, and its accompanying symptoms (such as hot flashes), is common in women with SLE.
Pain often occurs in the morning and improves during the day, only to return later when the patient tires.
Children may experience these symptoms as growing pains, and, in all patients, they may be the only symptoms for many years. Fever occurs in 90% of patients with SLE and is usually caused by the inflammatory process of the disease, not by infection. Three-quarters of patients with SLE have skin inflammation and skin lesions (ulcers, rashes, or other injured areas). Lupus, discoid -- view of lesions on the chest: This close-up picture of the neck clearly shows the typical rounded appearance of discoid lupus. In some people, reddish-purple lesions appear on the pads of fingers and toes or near the nails of fingers and toes. Lesions caused by vasculitis may ulcerate or blister if they erupt on mucous membranes in the mouth, nose, or vagina and can be painful if they occur on the throat. Vasulitis can attack blood vessels in almost any other organ, including the brain, the heart, and the gastrointestinal tract. Hair loss or breakage may also occur in about half of patients with SLE during severe flares or after pregnancy or severe illness. Raynaud's phenomenon is a condition in which cold or stress can cause spasms in impaired blood vessels, resulting in pain in fingers and toes. Almost 85% of patients with SLE experience problems associated with abnormalities in the blood. Patients who have APS have a very incidence of blood clots, which most often occur in the deep veins in the legs. This picture shows a red and swollen thigh and leg caused by a blood clot (thrombus) in the deep veins in the groin (iliofemoral veins). The effects on blood vessels have also been associated with confusion, headaches, and seizures.
Patients with APS who become pregnant have a high incidence of pregnancy loss, especially in the late term. Not all patients with APS carry both of the autoantibodies, and they can also wax and wane and so have varying effects.
The risk for cardiovascular disease, heart attack, and stroke is much higher than average in younger women with SLE. Inflammation of the membrane lining the lung (pleurisy) is the most common problem, which can cause shortness of breath and coughing.
A very serious and rare condition called pulmonary hypertension occurs when high pressure develops as a result of damage to the blood vessels of the lungs. The kidneys are a crucial battleground in SLE because it is here that the debris left over from the immune attacks is most likely to be deposited. Nearly all patients with SLE report some symptoms relating to problems that occur in the central nervous system (CNS), which includes the spinal cord and the brain. The most serious CNS disorder is inflammation of the blood vessels in the brain (vasculitis), which occurs in 10% of patients with SLE. About 45% of patients with SLE suffer gastrointestinal problems, including nausea, weight loss, mild abdominal pain, and diarrhea.
Inflamed blood vessels in the eye can reduce blood supply to the retina, resulting in degeneration of nerve cells and a risk of hemorrhage in the retina. In one study, 40% of patients with SLE quit work within 4 years of diagnosis, and many had to modify their work conditions. Women with lupus who conceive face high-risk pregnancies that increase the risks for themselves and their babies.
Women with lupus are 20 times more likely to die during pregnancy than women without the disease. Despite these obstacles, many women with lupus have healthy pregnancies and deliver healthy babies.
Vital organs or systems, such as lungs, kidneys, nervous system, joints skin, and others are affected in over a half of patients with SLE. Because of more effective and aggressive treatment, the prognosis for SLE has improved markedly over the past two decades.
Note: A patient must experienced four of the criteria before a doctor can classify the condition as SLE. Methods for measuring the antibodies involved with SLE vary, and the range of results can be bewildering. Antinuclear antibodies may be strongly present in other autoimmune diseases (such as scleroderma, SjA?A¶gren syndrome, or rheumatoid arthritis).
Some drugs can also produce positive antibody tests, including hydralazine, procainamide, isoniazid, and chlorpromazine. If SLE-like symptoms are present and the ANA test is positive, other tests for SLE will be administered.
If SLE-like symptoms are not present and the test is positive, the doctor will look for other causes, or the results will be ignored if the patient is feeling healthy.
Anti-double stranded DNA (Anti-ds DNA) is more likely to be found only in patients with SLE. When the ANA is negative but the diagnosis is still strongly suspected, a test for anti-Ro (also called anti-SSA) and anti-La (also called anti-SSB) antibodies may identify patients with a rare condition called ANA negative, Ro lupus.
An ELISA test (enzyme-linked immunosorbent assay) is performed to detect the anticardiolipin antibody.
As with the ANA, these antibodies also have a tendency to appear and disappear in a single patient.
If a skin rash is present, the doctor may take a biopsy (a tissue sample) from the margin of a skin lesion. Blood tests that measure creatinine, a protein metabolized in muscles and excreted in the urine. Tests of the cerebrospinal fluid (CSF) for elevated levels of autoantibodies are the most reliable ways to detect CNS complications caused by a faulty immune system. Additional tests, including electroencephalograms (EEGs), magnetic resonance imaging (MRI), computed tomography (CT), or x-rays may be useful when blood vessel blockage in the brain is suspected. If the doctor suspects that CNS symptoms are caused by infection, especially for patients who are receiving immunosuppressant therapy, a lumbar puncture should be performed. The primary approach to treating severe SLE is to suppress the immune factors, most often first with corticosteroids and other immunosuppressant drugs.
The major complications of the disease must be treated as separate problems, keeping in mind the specific aspects of SLE.
Over-the-counter NSAIDs include aspirin, ibuprofen (Motrin, Advil), naproxen (Aleve), ketoprofen (Actron, Orudis KT).
Prescription NSAIDs include ibuprofen (Motrin), naproxen (Naprosyn, Anaprox), diclofenac (Voltaren), tolmetin (Tolectin), ketoprofen (Orudis, Oruvail), dexibuprofen (Seractil).
Patients who have kidney problems associated with lupus (lupus nephritis) should be especially cautious about using NSAIDs. An ulcer is a crater-like lesion on the skin or mucous membrane caused by an inflammatory, infectious, or malignant condition.
Treatment may start initially with high doses in order to accumulate high levels of the drug in the bloodstream. Antimalarials may also be used in combination with other anti-SLE drugs, including immunosuppressants and corticosteroids. Severe SLE is treated with corticosteroids, also called steroids, which suppress the inflammatory process. Some people need to take oral prednisone for only a short time; others may require it for a long duration. No one should stop taking any steroids without consulting a doctor first, and if steroids are withdrawn, regular follow-up monitoring is necessary. Drugs known as immunosuppressants are often used, either alone or with corticosteroids for very active SLE, particularly when kidney or neurologic involvement or acute blood vessel inflammation is present.
Cyclophosphamide (Cytoxan) used to be considered the gold standard of treatment for lupus kidney disease (lupus nephritis).
Azathioprine (Imuran) has the lowest toxicity, but is less effective than other immunosuppressants.
Cyclosporine (Sandimmune) has been used for years, mostly for SLE associated with kidney involvement. In general, immunosuppressants should not be used alone unless corticosteroids are ineffective or inappropriate. Treatments for osteoporosis include calcium, vitamin D, bisphosphonates, parathyroid hormone, and selective estrogen-receptor modulators (SERMs).
Table 2:Treatments for Some Complications of Systemic Lupus Erythematosus The spleen is an organ that helps produce and maintain red blood cells. Patients should be sure they are fully immunized and should minimize their exposure to crowds or people with contagious illnesses.
The information provided herein should not be used during any medical emergency or for the diagnosis or treatment of any medical condition. Los expertos no saben con exactitud que hace que el virus del herpes permanezca adormecido durante largos periodos y luego abruptamente despierte para crear un caos; pero mucho consideran que un sistema inminitario debilitado, invita al herpes a volver. Su primera intencion al descubrir las llagas en sus genitales puede ser bombardearlas con todo lo que tenga en su gabinete medico.
Durante el primer ataque o los molestos ataques secundarios, un bano con agua caliente en la zona genital de tres a cuatro veces diarias, puede resultar calmante. El aire es fundamental para la cicatrizacion, por ello, use solo ropa interior que permita a su piel respirar, es decir, algodon, no fibras sinteticas. La miccion para gente con su primer brote de herpes puede producir intenso dolor cuando la orina acida para por las llagas abiertas, lo cual es particularmente aplicable a las mujeres. Aunque a esta enfermedad se le llama herpes genital, es posible contagiar el virus a otras partes del cuerpo si se toca una llaga abierta y luego se toca otra parte con los mismos dedos, por ejemplo la boca o los ojos. Algunas personas recomiendan el cinc, que vienen en presentaciones de unguento o capsulaas, el aminoacido lisina o el aditivo para alimentos hidroxitolueno butilado tomado como complemento, pueden contrarrestar los ataques de herpes, sin embargo, si decide probar cualquiera de estos productos, serpa que las dosis elevadas pueden ser peligrosas y que solamente deben tomarse bajo la supervision de un profesional. La gente que se halla mas tensa, deprimida, que se encuentra abrumada por la hostilidad y que se encoleriza mas facilmente parece sufrir erupciones mas frecuentes. Lea acerca del herpes, hable con su doctor, trate de entenderlo lo mas que pueda y aprenda a dominar la situacion lo mas posible.
Las tecnicas son muy varias, entre ellas la meditacion, terapia de relajacion, visualizacion y retroalimentacion biologica. Dichos remedios no deben sustituir los tratamientos medicos, ni se recomienda que la gente los use sin antes consultar a un profesional. BackgroundSystemic lupus erythematosus (SLE) is a chronic, often life-long, autoimmune disease. It refers to the rash that extends across the bridge of the nose and upper cheekbones and was thought to resemble a wolf bite.
CausesSystemic lupus erythematosus is a complex disorder that most likely results from a combination of processes and factors. This immune activation may begin as an appropriate response to an unwanted "invader." But, because of a combination of genetic factors, an individual with lupus develops an ongoing immune response that does not shut itself off appropriately. Some autoantibodies are normal in a well-balanced immune system, and serve various roles to help the body dispose of wastes, protect from infectious invaders, and to keep blood vessels clear. The immune system is then called upon to repair wounds by clotting any bleeding blood vessels and initiating fiber-like patches to the tissue.
B cells and T cells are two important components of the immune system that play a role in the inflammation associated with lupus.
Antigens are foreign bodies (such as bacteria and viruses) that stimulate the immune system to produce autoantibodies. In lupus, a complex interaction between activated immune cells and an impaired antigen-elimination process leads to a greater than normal range of what the antibodies recognize. An autoantibody called anti-double stranded DNA (anti-ds DNA) may play an important role in some lupus patients. This antibody is found most often in lupus patients of African descent and is almost never detected in people without lupus. Most immune cells secrete or stimulate the production of powerful immune factors called cytokines. Specific cytokines called interferons and interleukins play a critical role in SLE by regulating the secretion of autoantibodies by B cells.
A brother or sister of a patient with the disorder has 20 times the risk as someone without an immediate family member with SLE.
When it does occur, it is often associated with thrombotic thrombocytopenia purpura, a condition resulting from abnormally low levels of blood platelets.

Studies have investigated the relationship among hormones, SLE, and rheumatoid arthritis, another autoimmune disease. These include high blood pressure (hypertension) medications, including hydrochlorothiazide, angiotensin-converting-enzyme inhibitors, and calcium-channel blockers. Smoking may be a risk factor for triggering SLE and can increase the risk for skin and kidney problems in women who have the disease. Possible SLE triggers include colds, fatigue, stress, chemicals, sunlight, and certain drugs. Patients with SLE may be more likely to have been exposed to certain viruses than the general population. Clusters of SLE cases have occurred in populations with high exposure to certain chemicals.
Cytokines, major immune factors that are active in SLE, are directly affected by sex hormones.
Hormone replacement therapy (HRT), which is used to relieve these symptoms, increases the risk for blood clots and heart problems. Female patients with lupus used to be cautioned against taking oral contraceptives (OCs) due to the possibility that estrogen could trigger lupus flare-ups. About half of these lesions are photosensitive; that is, they are aggravated by ultraviolet (UV) radiation from sunlight, even from light coming through a window.
This rash causes little scarring, although spidery, branching lines of swollen capillaries (the tiniest blood vessels) may appear. Subacute cutaneous lupus erythematosus (SCLE) can cause skin lesions on parts of the body that are exposed to sunlight. It occurs as part of the inflammatory response in blood vessels, which can narrow them and reduce circulation. ComplicationsSystemic lupus erythematosus (SLE) can cause systemic complications throughout the body. Blood clotting, in turn, puts patients at higher risk for stroke and pulmonary embolism (clots in the lungs). A rare blood complication of SLE that occurs primarily in Asians is called acute lupus hemophagocytic syndrome. The immune response in SLE can cause chronic inflammation and other damaging effects that can cause significant injury to the arteries and tissues associated with the circulation and the heart. If kidney injury develops, it almost always occurs within 10 years of the onset of SLE, rarely after that.
CNS involvement is more likely to occur in the first year, usually during flare-ups in other organs. The immune system is indeed overactive in SLE, but it is also abnormal and reduces the ability to fight infections. Severe inflammation of the intestinal tract occurs in less than 5% of patients and causes acute cramping, vomiting, diarrhea, and, rarely, intestinal perforation, which can be life-threatening. Significant factors that predicted job loss included high physical demands from the work itself, a more severe condition at the time of diagnosis, and lower educational levels. Doctors strongly recommend that women wait to conceive until their disease state has been inactive for at least 6 months. Antiphospholipid and anticardiolipin antibodies can increase the risks for preeclampsia, miscarriage, and stillbirths.
Women with active disease may need to take low-dose corticosteroids, but women with inactive disease should avoid these drugs. Women with lupus have a 6 times greater risk for developing deep vein thrombosis (blood clots) than women without the disease.
Blood infections (sepsis), pneumonia, and urinary tract infections are more common in pregnant women with lupus. Women with lupus are three times more likely than healthy women to develop preeclampsia (pregnancy-related high blood pressure), which can be potentially life threatening. Women with SLE have an increased risk of having a pre-term birth, stillbirth, or Caesarean section. To increase the odds of a successful pregnancy, it is important for women to plan carefully before becoming pregnant. More commonly, SLE is a chronic, life-long disease, alternating between periods of symptom relapse, (called flares), and remission. These criteria, proposed by the American College of Rheumatology, are not to be relied upon solely for diagnosis, however. A primary test for SLE checks for antinuclear antibodies (ANA), which attack the cell nucleus. High or low concentrations of ANA also do not necessarily indicate the severity of the disease, since antibodies tend to come and go in patients with SLE. It may play an important role in injury to blood vessels found in SLE, and high levels often indicate kidney involvement.
These autoantibodies may be involved in the sun-sensitive rashes experienced by patients with SLE and are also found in association with neonatal lupus syndrome, in which a pregnant mother's antibodies cross the placenta and cause inflammation in the developing child's skin or heart. An advance in diagnosing SLE has been the detection of antibodies to molecules called SR proteins, which are carried by most patients. Antiphospholipid antibodies increase the risks for blood clots and may be responsible for narrowing of (and irregularities in) blood vessels.
A longer than normal blood clotting time indicates a higher chance for clotting in the body and, therefore, the presence of lupus anticoagulant. Patients who have these autoantibodies as well as blood clotting problems or frequent miscarriage are diagnosed with antiphospholipid syndrome (APS), which often occurs in SLE but can also develop independently. Blood tests of patients with SLE often show low levels of serum complement, a protein in the blood that aids the body's infection fighters. The first blood test ever used for SLE called LE (lupus erythematosus) cell test is positive in only about half of patients with SLE and is no longer used that often.
White and red blood cell and platelet counts are usually lower than normal and, depending on severity, are used to determine complications, such as anemia or infection. A test known as a lupus band detects antibodies known as immunoglobulin G (IgG), which are located just below the outer layer of the tissue sample. High levels suggest kidney damage, although kidney problems can also be present with normal creatinine levels. This may be performed to determine if lupus nephritis is present when less invasive tests indicate kidney involvement. SLE occurring in the central nervous system (CNS) can be difficult to diagnose because its symptoms are easily confused with other psychiatric and neurologic conditions. NSAIDs block prostaglandins, the substances that dilate blood vessels and cause inflammation and pain. Regular, long-term use of NSAIDs can cause ulcers and gastrointestinal bleeding, which can lead to anemia. Patients with lupus who take NSAIDs on a regular basis should have their liver and kidney function tested every 3 - 4 months. Patients can take certain medicines to suppress the acid in the stomach causing the erosion of the stomach lining.
This drug is effective as maintenance therapy to reduce flares in patients with mild or inactive disease. Eye damage after taking hydroxychloroquine is reversible when caught in time and treated, but it is not reversible if the damage develops after taking chloroquine.
Steroids can help relieve many of the complications and symptoms, including anemia and kidney involvement. Other drugs include methylprednisolone (Medrol, Solumedrol), hydrocortisone, and dexamethasone (Decadron). An intravenous administration of methylprednisolone using "pulse" therapy for 3 days is proving useful for flare-ups in the joints.
Most patients with SLE can eventually function without prednisone, although some may have to choose between the long-term toxicity of corticosteroids and the complications of active disease. Unfortunately, serious and even life-threatening complications have been associated with long-term steroid use. Long-term use of oral steroid medications suppresses secretion of natural steroid hormones by the adrenal glands. Patients should discuss with their doctors measures for preventing adrenal insufficiency during withdrawal, particularly during stressful times, when the risk increases. These drugs suppress the immune system by damaging cells that grow rapidly, including those that produce antibodies. Cyclophosphamide is given intravenously and is sometimes used in combination with corticosteroids or other drugs. Many recent studies have shown that this drug works better than cyclophosphamide and causes far fewer severe side effects (diarrhea is the main side effect). Intravenous immunoglobulins (IVIG) are sometimes used for patients who have not responded to other SLE treatments. Dehydroepiandrosterone (DHEA) is a natural steroid hormone that is produced by the adrenal glands and converted into estrogen and androgen.
Some patients with severe lupus have achieved at least short-term remission after undergoing autologous transplantation of stem cells and high-dose drug therapy to suppress the damaging immune factors. A promising treatment uses ultraviolet A-1 (UVA-1) radiation, long UVA wave lengths that do not promote sunburn and may actually block inflammatory immune factors.
Plasmapheresis is a process in which the fluid part of the blood, called plasma, is removed from blood cells. Immunizations with inactive viruses and preventive antibiotics should be considered for patients with SLE who are at high risk for infection. While inflammation of the lung (pneumonitis) resembles pneumonia, it is not an infection but is a result of the autoimmune process.
Hydroxychloroquine or aspirin may help prevent blood clots in women with antiphospholipid syndrome (APS).
Treatments that may be effective for thrombocytopenia include combinations of a corticosteroid and either danazol (a male hormone) or the antimalarial hydroxychloroquine. Mycophenolate mofetil (CellCept, Myfortic) and intravenous cyclophosphamide, as well as steroids are used to treat lupus kidney disease.
Kidney transplant or dialysis should be considered for patients with SLE with severe kidney damage.
SLE is also accompanied by high cholesterol levels, which requires diet changes and drug therapies. It also aids the body's immune system by producing white blood cells that destroy harmful substances in the body.
Light-to-moderate exercise, interspersed with rest periods, is good for the heart, helps fight depression and fatigue, and can help keep joints flexible. Simple preventive measures include avoiding overexposure to ultraviolet rays and wearing protective clothing and sunblocks. Getting adequate rest of at least 8 hours and possibly napping during the day may be helpful.
Mycophenolate mofetil for induction therapy of lupus nephritis: a systematic review and meta-analysis. A licensed medical professional should be consulted for diagnosis and treatment of any and all medical conditions.
Ya sea por la progresion natural de la enfermedad o por intervencion terapeutica, el herpes dista de ser un estado sin esperanza.
El petrolato y los unguentos antibioticos peuden bloquear este aire y frenar el proceso de cicatrizacion. Cuando termine su bano, seque la zona con un secador de cabello ajustado en baja temperatura o frio, para no quemarse. Si emplea pantimedias de nylon, asegurese de que la zona de la entrepierna este hecha de algodon. Trate de alejar el chorro de orina de las llagas mediante un poco de papel higienico entrollado. Porque un sistema inmunitario que esta fortalecido puede evitar que el virus del herpes actue y las compresas hechas a base de acete de ricino fortifican su sistema inmunitario.
Sobre el plastico aplique un cojincillo electrico calefactor ajustado a la temperatura mas alta posible, y dejeselo durante una hora. This leads to waxing and waning flares of inflammation that can involve various organs of the body, depending on specific features of this self-perpetuating immune response in individual patients.
In healthy people, autoantibodies tend to be well-regulated and well "masked," or covered up, until needed. When a T cell recognizes an antigen it will produce chemicals (cytokines) that cause B cells to multiply and release many immune proteins (antibodies). Eventually, antibodies are made that recognize more of the body's own tissues in a stronger or more persistent manner than is healthy, and inflammatory responses are mounted in these tissues.
Such autoantibodies may be present in individuals up to 7 years prior to their developing symptoms of lupus.
This is comprised of more than 30 proteins and is important for defending and regulating the immune response. Risk FactorsThe number of people diagnosed with lupus has more than tripled over the past four decades.
SLE in children may also be caused by certain medications, including minocycline and zafirlukast.
Higher levels of estrogen are associated with SLE, while lower levels are associated with rheumatoid arthritis.
These viruses include the Epstein-Barr virus (the cause of mononucleosis), cytomegalovirus, and parvovirus-B1.
When they bombard the skin, they can alter the structure of DNA in cells below the surface. In general, estrogen enhances antibody production, and testosterone reduces antibody production, although their exact role in SLE may be more complicated than that since there are various ways in which each hormone might influence various immune cells.
However, recent evidence indicates that OCs are safe, at least for women with inactive or stable lupus.
Untreated, the margins gradually extend outward as the center dries out and shrivels, causing severe scarring. This is a specific set of conditions related to the presence of autoantibodies called lupus anticoagulant and anticardiolipin. In such cases, blood clotting is impaired, which causes bruising and bleeding from the skin, nose, gums, or intestines.
Patients with SLE often neutropenia, but the condition is usually harmless unless the reductions are so severe that they leave the patient vulnerable to infections. It is generally of short duration and characterized by fever and a sudden drop in blood cells and platelets.
In addition, SLE treatments (particularly corticosteroids) affect cholesterol, weight, and other factors that can also affect the heart. About 50% of patients with SLE exhibit inflammation of the kidneys (called lupus nephritis).This condition occurs in different forms and can vary from mild to severe.

Central nervous system symptoms are usually mild, but there is little effective treatment available for them. Patients are not only prone to the ordinary streptococcal and staphylococcal infections, but they are also susceptible to fungal and parasitic infections (called opportunistic infections), which are common in people with weakened immune systems.
Fluid retention and swelling can cause intestinal obstruction, which is much less serious but causes the same type of severe pain.
Patients with SLE also commonly experience reductions in bone mass density (osteoporosis) and have a higher risk for fractures, whether or not they are taking corticosteroids (which can increase the risk for osteoporosis).
Anti-SSA and anti-SSB antibodies can increase the risk for neonatal lupus erythematosus, a condition that can cause skin rash and liver and heart damage to the newborn baby. Steroids appear to pose a low risk for birth defects, but can increase a pregnant woman’s risks for gestational diabetes, high blood pressure, infection, and osteoporosis. The risk is highest for patients with antiphospholipid antibodies, active kidney disease, or high blood pressure. Women with lupus are 3 times more likely to need a transfusion during pregnancy than women without lupus. The only symptoms may be the skin rashes of discoid lupus erythematosus (DLE) or subacute cutaneous lupus erythematosus (SCLE) with or without joint aches. The disease may begin in any of the various systems of the body and progress unpredictably to others.
About 85 - 95% of people with lupus survive 10 years, and many people have a normal life span. Although many lupus patients may not have this antibody, its presence almost always indicates SLE. The test accurately detects lupus in 50 - 70% of patients who test positive for these antibodies. Antiphospholipid antibodies are linked with miscarriages and other pregnancy complications, strokes, heart attacks and blood clots in almost any part of the body, including kidneys, legs, lungs, and eyes. TreatmentNo treatment cures systemic lupus erythematosus, but many therapies can suppress symptoms and relieve discomfort.
However, many patients with discoid lupus do not respond to steroids, particularly if they have eruptions that are caused by sun sensitivity.
Patients should always use sunblock creams (not just sunscreens) and always wear hats and clothing made of tightly woven fabrics.
To avoid these problems, it’s best to take NSAIDs with food or immediately after a meal.
Hydroxychloroquine may help protect against blood clots in people with antiphospholipid syndrome, high cholesterol levels, and bone loss. Some researchers believe they inhibit the immune response, and others think they interfere specifically with inflammation. The bone-thinning condition osteoporosis is a common and particularly severe long-term side effect of prolonged steroid use.
After withdrawal from these drugs, this so-called adrenal suppression persists and it can take the body a while (sometimes up to a year) to regain its ability to produce natural steroids again.
About a third of patients take immunosuppressants at some point in the course of the disease. B cell over-activation has been identified as a key component of the lupus disease process. The synthetic equivalent of DHEA, prasterone (Prestara), is being investigated as a potential treatment for SLE.
Small studies have suggested that UVA-1 phototherapy may have some benefits for lowering disease activity in SLE.
However, antibiotic drugs such as penicillin or the sulfa drugs may cause sensitivity rashes that can be confused with SLE rash. This condition needs to be treated with corticosteroids or immunosuppressants, but only if the doctor is sure infection is not present. Drugs known as prostacylins -- which include epoprostenol, iloprost, and treprostinil -- are standard drugs. Report of a Task Force of the EULAR Standing Committee for International Clinical Studies Including Therapeutics.
Muchas personas tienen diferentes factores desencadenantes de los brotes de herpes genital o labial. Como sucede con las llagas, no necesita preocuparse por adquirir una infeccion secundaria, pero lo unico que necesita emplear para mantener la zona libre de germenes es agua y jabon. Jamas emplee una crema de cortisona, puede puede inhibir su sistema inmunitario y de hecho alentar el crecimiento del virus.
El aire del secador tambien sera calmante y tal vez acelere la cicatrizacion al ayudar a resecar las llagas. Si cree que puede rascarse durante la noche, mientras esta dormido, y sin darse cuenta, cubra sus llagas con material protector poroso, como una gasa.
Cuando tiene llagas, es altamente contagioso, por lo tanto debe evitar tener relaciones sexuales.
SLE may affect various parts of the body, but it most often manifests in the skin, joints, blood, and kidneys.
Therefore, it is probably the high activity and high detestability of autoantibodies that makes lupus unique, not the fact that they exist. These antibodies circulate widely in the bloodstream, recognizing the foreign particles and triggering inflammation in order to rid the body of the invasion. Inherited deficiencies in certain complement components (C1q, C1r, C1s, C4, and C2) have long been associated with SLE. Some research suggests that some patients, in fact, progress from one disease to the other, and that such transitions occur during major hormonal shifts, such as the onset of menopause or pregnancy. Anyone diagnosed with cutaneous lupus erythematosus should be sure to tell their doctors all the medications (including herbs and supplements) that they are taking. In particular, some research suggests a strong association between Epstein-Barr virus (EBV) and increased risk of lupus, particularly for African-Americans. The immune system may perceive these altered skin cells as foreign and trigger an autoimmune response against them. Women with SLE may have lower levels of several active male hormones (androgens), and some men who are affected by SLE may also have abnormal androgen levels. If discoid lesions appear on the scalp, they can plug hair follicles and cause irreversible hair loss.
These autoantibodies react against fatty molecules called phospholipids, and so are called antiphospholipids. Rarely, lupus pneumonitis becomes chronic and causes scarring in the lungs, which reduces their ability to deliver oxygen to the blood. They also face an increased risk for urinary tract, herpes, salmonella, and yeast infections. The number and intensity of symptoms in mild cases often decrease over time, as does the likelihood of major organ involvement.
The biopsy will not differentiate between systemic and discoid lupus, but it can rule out other diseases. Electron microscopy (very high-powered electronic microscopes) may be especially important in obtaining critical information on the degree of kidney damage. Genetic research in lupus is progressing very rapidly, and hopefully new drugs will be approved in the future.
A cream derived from vitamin A (Tegison) may help some lesions that do not clear up with steroid creams. Long-term use of NSAIDs (with the exception of aspirin) can also increase the risk for heart attack and stroke.
Medications that can prevent osteoporosis include calcium supplements, parathyroid hormone, alendronate risedronate, or hormone replacement therapy in post-menopausal women. A few cases of severe adrenal insufficiency have occurred when patients switched from oral to inhaled steroids, which, in rare cases, has resulted in death. Side effects are very severe and include nausea, vomiting, hair loss, infertility, and infections. Most doctors now recommend mycophenalate mofetil as a first-line treatment for newly diagnosed patients with mild or moderate lupus kidney disease. Prasterone is still in the drug development stage and it is not clear when, or if, it will be commercially available.
The plasma, which contains the inflammatory antibodies and other immunologically active substances, is discarded and replaced with other fluids. Surgical removal of the spleen may be advisable if bleeding disorders are a serious problem, but this option should be considered carefully, because the spleen provides one line of defense against infection.
Studies are conflicting, however, over whether SLE transplant patients have higher organ-rejection rates than other kidney-transplant recipients. Patients who may benefit from them should discuss risks and benefits with an SLE specialist.
Links to other sites are provided for information only -- they do not constitute endorsements of those other sites. Al prestar atencion usted podra saber cual de estos factores como: exposicion al sol, el estres, o alimentos ricos arginina. De todos modos, nada de lo que tenga matara el virus y en cambio puede contener muchas cosas que empeoren la situacion. Cuando no se presenten llagas tal vez no contagie el virus, pero debera emplear condon para asegurar mayor proteccion y tranquilidad de conciencia.
Continue los tratamientos tres veces por semana, y aumente la aplicacion de los paquetes durante un ataque de herpes.
Guidelines recommend that women who take HRT use the lowest possible dose for the shortest possible time. Their actions have complex effects that include causing narrowing and abnormalities of blood vessels. Corticosteroid and immunosuppressants, treatments used for SLE, also increase the risk for infections, thereby compounding the problem.
Certain medications (aspirin, heparin) and tests (fetal heart monitoring) may be needed to ensure a safe pregnancy.
Methotrexate (Rheumatrex) and cyclophosphamide (Cytoxan) should not be taken during pregnancy.
Complement levels are especially low if there is kidney involvement or other disease activity. Tests for other antibodies will rule out or confirm discoid lupus and subacute cutaneous lupus. There are also different drugs available to treat some of the conditions associated with lupus.
It may not be appropriate for patients with kidney failure or rapidly progressing kidney disease.
Scientists are investigating other treatment options, including autologous stem cell transplantation. Son los causantes de su brote de herpes genital o labial2- Aprenda a reconocer los sintomas predominantes. A proposito, el hecho de que ya tenga herpes no queire decir que no pueda contagiarse de otra manifestacion de la enfermedad. Women with SLE who have active disease, antiphospholipid antibodies, or a history of blood clots or heart disease should not use HRT. For this reason, women should wait until the disease reaches a stable state before taking OCs. SLE can affect so many organs that a cause of death in some people with SLE may not be directly attributed to the condition.
Mycophenolate mofetil should not be used during pregnancy as it can cause miscarriage and birth defects. Plasmapheresis is not useful for routine management of patients but may have some benefits for patients who do not respond to standard treatments or in specific cases, such as lupus patients with hemolytic anemia. The procedure has shown promise in studies for treating lupus-associated APS, but it is still experimental. Any duplication or distribution of the information contained herein is strictly prohibited.
Todo el mundo va a un ritmo tan rapido que es facil pasar por alto las sustancias precursoras de un brote de herpes genital o labial. Aunque esto no sucede frecuentemente, el herpes genital puede recurrir con mas de una variedad del virus. The weight of evidence to date, however, finds no support for this concern.) Some drugs have been associated with a temporary lupus syndrome (drug-induced lupus), which resolves when these drugs are stopped. In addition, women who have a history of, or who are at high risk for, blood clots (particularly women with antiphospholipid syndrome) should not use OCs. A primary cause of death among patients with lupus is atherosclerosis, a disease of the coronary blood vessels resulting from accelerated buildup of plaque. The first symptoms of SLE can resemble one of many syndromes or disorders, including rheumatoid arthritis, Still's disease, rheumatic fever, Lyme disease, multiple sclerosis, thrombotic thrombocytopenia purpura, cryoglobulinemia, Weber-Christian disease, viral infections, vasculitis, psychosis, and other conditions.
Other autoimmune disorders, such as SjA?A¶gren syndrome or scleroderma, may even be present at the same time as SLE. Incluso si usted toma una dieta rica en arginina normalmente le aconsejo abstenerse de consumirla durante situaciones de estres  y asi reducira el riesgo de crisis provocada por arginina. Dentro de este tipo de receptores estA?n los TLR (Toll Like Receptor) que se encuentran en la superficicie o en el interior celular. Symptoms tend to be worse during winter months, perhaps because prolonged exposure to sunlight in the summer causes a gradual build-up of factors that trigger symptoms months later.
La culebrilla puede desatarse por la edad avanzada o por el uso crónico de medicamentos con cortisona.
Todo lo que necesita Saber Infeccion Vaginal por Hongos o Herpes Genital ?Como identificarla?
Las infecciones de culebrilla son mucho menos contagiosas que las infecciones de varicela, porque la varicela se puede contagiar por el aire.
El virus de la culebrilla se contagia sólo por contacto con el fluido de las ampollas. Se puede contagiar por contacto directo con una ampolla o con el fluido, o por contacto indirecto, como con una toalla. Para prevenir este problema puede tomar un medicamento antiviral cuando le diagnostiquen la culebrilla y tomarlo durante 7 días.

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