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Laparoscopy assisted middle-segment-preserving pancreatectomy for multiple pancreatic neuroendocrine tumors: report of a case. Usui M, Kuriyama N, Uchida K, Kishiwada M, Mizuno S, Sakurai H, Tabata M, Shiraishi T, Isaji S.
Desaki R, Mizuno S, Tanemura A, Kishiwada M, Murata Y, Azumi Y, Kuriyama N, Usui M, Sakurai H, Tabata M, Isaji S.
Paradoxical impact of the remnant pancreatic volume and infectious complications on the development of nonalcoholic fatty liver disease after pancreaticoduodenectomy. Sato R, Kishiwada M, Kuriyama N, Azumi Y, Mizuno S, Usui M, Sakurai H, Tabata M, Yamada T, Isaji S. Thrombomodulin administration attenuates ischemia-reperfusion injury of the remnant liver after 70% hepatectomy in rats: simulated model of small-for-size graft in living donor liver transplantation. Tanemura A, Kuriyama N, Azumi Y, Ohsawa I, Kishiwada M, Mizuno S, Usui M, Sakurai H, Tabata M, Isaji S.
Biliary complications in 108 consecutive recipients with duct-to-duct biliary reconstruction in living-donor liver transplantation. Mizuno S, Inoue H, Tanemura A, Murata Y, Kuriyama N, Azumi Y, Kishiwada M, Usui M, Sakurai H, Tabata M, Yamada R, Yamamoto N, Sugimoto K, Shiraki K, Takei Y, Isaji S.
Hypertension and hepatitis C virus infection are strong risk factors for developing late renal dysfunction after living donor liver transplantation: significance of renal biopsy.
Fujinaga K, Usui M, Yamamoto N, Ishikawa E, Nakatani A, Kishiwada M, Mizuno S, Sakurai H, Tabata M, Isaji S. Negative prognostic impact of renal replacement therapy in adult living-donor liver transplant recipients: preoperative recipient condition and donor factors.
Iwata H, Mizuno S, Ishikawa E, Tanemura A, Murata Y, Kuriyama N, Azumi Y, Kishiwada M, Usui M, Sakurai H, Tabata M, Yamamoto N, Sugimoto K, Shiraki K, Takei Y, Ito M, Isaji S. Clinicopathological factors affecting survival and recurrence after initial hepatectomy in non-B non-C hepatocellular carcinoma patients with comparison to hepatitis B or C virus. Okuda Y, Mizuno S, Shiraishi T, Murata Y, Tanemura A, Azumi Y, Kuriyama N, Kishiwada M, Usui M, Sakurai H, Tabata M, Yamada T, Isaji S. Anterior approach to the superior mesenteric artery by using nerve plexus hanging maneuver for borderline resectable pancreatic head carcinoma. Mizuno S, Isaji S, Tanemura A, Kishiwada M, Murata Y, Azumi Y, Kuriyama N, Usui M, Sakurai H, Tabata M. Incarcerated left diaphragmatic hernia following left hepatectomy for living donor liver transplantation.
Usefulness of monitoring cell-mediated immunity for predicting post-kidney transplantation viral infection.
Gemcitabine-based chemoradiotherapy followed by surgery for borderline resectable and locally unresectable pancreatic ductal adenocarcinoma: significance of the CA19-9 reduction rate and intratumoral human equilibrative nucleoside transporter 1 expression. Kobayashi M, Mizuno S, Murata Y, Kishiwada M, Usui M, Sakurai H, Tabata M, Ii N, Yamakado K, Inoue H, Shiraishi T, Yamada T, Isaji S.
Masuya M, Shiraki K, Sugimoto K, Yamamoto N, Yoneda M, Kanayama K, Nishikawa K, Ino K, Tawara I, Ohishi K, Sakurai H, Usui M, Shiraishi T, Isaji S, Takei Y, Katayama N.
Prolonged thrombocytopenia after living donor liver transplantation is a strong prognostic predictor irrespective of splenectomy: the significance of ADAMTS13 and graft function [corrected]. Nobuoka Y, Wada H, Mizuno S, Kishiwada M, Usui M, Sakurai H, Tabata M, Kobayashi T, Nobori T, Uemoto S, Isaji S.
Immune response following liver transplantation compared to kidney transplantation: usefulness of monitoring peripheral blood CD4+ adenosine triphosphate activity and cytochrome P450 3A5 genotype assay. Nobuoka Y, Mizuno S, Nishikawa K, Nakatani K, Muraki Y, Yamada T, Okuda M, Nobori T, Sugimura Y, Isaji S. Benefit of technetium-99m galactosyl human serum albumin scintigraphy instead of indocyanine green test in patients scheduled for hepatectomy. The long-term effects of splenectomy and subsequent interferon therapy in patients with HCV-related liver cirrhosis. Inagaki Y, Sugimoto K, Shiraki K, Tameda M, Kusagawa S, Nojiri K, Ogura S, Yamamoto N, Takei Y, Ito M, Mizuno S, Usui M, Sakurai H, Isaji S.
Immunological aspects in late phase of living donor liver transplant patients: usefulness of monitoring peripheral blood CD4+ adenosine triphosphate activity. Mizuno S, Muraki Y, Nakatani K, Tanemura A, Kuriyama N, Ohsawa I, Azumi Y, Kishiwada M, Usui M, Sakurai H, Tabata M, Yamamoto N, Yamada T, Shiraki K, Takei Y, Nobori T, Okuda M, Isaji S. An approach to the intra-thoracic inferior vena cava through the abdominal cavity preparing for total hepatic vascular exclusion by sagittal diaphragmotomy. Mizuno S, Yagihara M, Tanemura A, Kuriyama N, Azumi Y, Kishiwada M, Ohsawa I, Usui M, Sakurai H, Tabata M, Miyabe M, Isaji S. Combination assays for evaluation of immune function and CYP3A5 genotype to identify the risk of infectious complications and mortality in living donor liver transplant patients. Mizuno S, Nakatani K, Muraki Y, Tanemura A, Azumi Y, Kuriyama N, Ohsawa I, Kishiwada M, Usui M, Sakurai H, Tabata M, Okuda M, Nobori T, Isaji S. Randomized, multicenter trial comparing tacrolimus plus mycophenolate mofetil to tacrolimus plus steroids in hepatitis C virus-positive recipients of living donor liver transplantation. Takada Y, Kaido T, Asonuma K, Sakurai H, Kubo S, Kiuchi T, Inomata Y, Isaji S, Tsumura H, Teramukai S, Matsubara Y, Sakabayashi S, Uemoto S. Behavior of ADAMTS13 and Von Willebrand factor levels in patients after living donor liver transplantation.
Takahashi N, Wada H, Usui M, Kobayashi T, Habe-Ito N, Matsumoto T, Uemoto S, Nobori T, Isaji S. Margin-negative limited resection of metastatic pancreatic tumors from rectal cancer preoperatively diagnosed by endoscopic ultrasound-guided fine-needle aspiration biopsies: report of two cases. Surgery for chronic pancreatitis decreases the risk for pancreatic cancer: a multicenter retrospective analysis.
Ueda J, Tanaka M, Ohtsuka T, Tokunaga S, Shimosegawa T; Research Committee of Intractable Diseases of the Pancreas. Dual cytoprotective effects of splenectomy for small-for-size liver transplantation in rats. Kuriyama N, Isaji S, Kishiwada M, Ohsawa I, Hamada T, Mizuno S, Usui M, Sakurai H, Tabata M, Yamada T. Effect of portal haemodynamics on liver graft and intestinal mucosa after small-for-size liver transplantation in swine. Tumor enucleation with preoperative endoscopic transpapillary stenting for pediatric insulinoma. Ide S, Uchida K, Inoue M, Koike Y, Otake K, Matsushita K, Hashimoto K, Nagano Y, Inoue H, Isaji S, Kusunoki M. Proposal for a morphological classification of intraductal papillary neoplasm of the bile duct (IPN-B). Kato H, Tabata M, Azumi Y, Osawa I, Kishiwada M, Hamada T, Mizuno S, Usui M, Sakurai H, Isaji S. Intracellular interferon-I? staining analysis of donor-specific T-cell responses in liver transplant recipients. Okanami Y, Tsujimura K, Mizuno S, Tabata M, Isaji S, Akatsuka Y, Kuzushima K, Takahashi T, Uemoto S. Living donor liver transplantation for the patients with portal vein thrombosis: use of an interpositional venous graft passed posteriorly to the pancreatic parenchyma without using jump graft.
Mizuno S, Murata Y, Kuriyama N, Ohsawa I, Kishiwada M, Hamada T, Usui M, Sakurai H, Tabata M, Isaji S. Postoperative liver dysfunction in living donors after left-sided graft hepatectomy: portal venous occlusion of the medial segment after lateral segmentectomy and hepatic venous congestion after left lobe hepatectomy. Kumamoto K, Mizuno S, Kuriyama N, Ohsawa I, Kishiwada M, Hamada T, Usui M, Sakurai H, Tabata M, Isaji S.
Donor age affects liver regeneration during early period in the graft liver and late period in the remnant liver after living donor liver transplantation. Impact of CYP3A5 genotype of recipients as well as donors on the tacrolimus pharmacokinetics and infectious complications after living-donor liver transplantation for Japanese adult recipients. Muraki Y, Usui M, Isaji S, Mizuno S, Nakatani K, Yamada T, Iwamoto T, Uemoto S, Nobori T, Okuda M.
Pancreaticoduodenectomy with resection of the splenic artery and splenectomy for pancreatic double cancers after total gastrectomy. Impact of histological response after neoadjuvant chemoradiotherapy on recurrence-free survival in UICC-T3 pancreatic adenocarcinoma but not in UICC-T4.
Murata Y, Mizuno S, Kishiwada M, Hamada T, Usui M, Sakurai H, Tabata M, Inoue H, Shiraishi T, Isaji S. Human equilibrative nucleoside transporter 1 expression is a strong independent prognostic factor in UICC T3-T4 pancreatic cancer patients treated with preoperative gemcitabine-based chemoradiotherapy. Murata Y, Hamada T, Kishiwada M, Ohsawa I, Mizuno S, Usui M, Sakurai H, Tabata M, Ii N, Inoue H, Shiraishi T, Isaji S. Is it possible to predict the development of an incisional surgical site infection and its severity after biliary tract surgery for benign disease? Radiofrequency ablation combined with chemoembolization: treatment of recurrent hepatocellular carcinomas after hepatectomy. Lethal hepatic infarction following plasma exchange in living donor liver transplant patients.
Effect of a prostaglandin I(2) analog on the expression of thrombomodulin in liver and spleen endothelial cells after an extensive hepatectomy.
Usui M, Kato H, Kuriyama N, Azumi Y, Kishiwada M, Mizuno S, Sakurai H, Tabata M, Hayashi T, Suzuki K, Isaji S. Monitoring peripheral blood CD4+ adenosine triphosphate activity after living donor liver transplantation: impact of combination assays of immune function and CYP3A5 genotype. Mizuno S, Hamada T, Nakatani K, Kishiwada M, Usui M, Sakurai H, Tabata M, Sakamoto Y, Nishioka J, Muraki Y, Okuda M, Nobori T, Isaji S. Azumi Y, Isaji S, Kato H, Nobuoka Y, Kuriyama N, Kishiwada M, Hamada T, Mizuno S, Usui M, Sakurai H, Tabata M.
Indocyanine green (ICG) fluorescence imaging-guided cholangiography for donor hepatectomy in living donor liver transplantation.
Prospective evaluation of three different diagnostic criteria for disseminated intravascular coagulation.
Takemitsu T, Wada H, Hatada T, Ohmori Y, Ishikura K, Takeda T, Sugiyama T, Yamada N, Maruyama K, Katayama N, Isaji S, Shimpo H, Kusunoki M, Nobori T. Chemopreventive anti-cancer agent acyclic retinoid suppresses allogeneic immune responses in rats.
Total vascular hepatic exclusion for tumor resection: a new approach to the intrathoracic inferior vena cava through the abdominal cavity by cutting the diaphragm vertically without cutting the pericardium. Mizuno S, Kato H, Azumi Y, Kishiwada M, Hamada T, Usui M, Sakurai H, Tabata M, Shimpo H, Isaji S. The cytoprotective effects of addition of activated protein C into preservation solution on small-for-size grafts in rats. Kuriyama N, Isaji S, Hamada T, Kishiwada M, Ohsawa I, Usui M, Sakurai H, Tabata M, Hayashi T, Suzuki K.
Prospective Study on the Outcome of Patients With Hepatocellular Carcinoma Registered for Living Donor Liver Transplantation: How Long Can They Wait? Mizuno S, Yokoi H, Shiraki K, Usui M, Sakurai H, Tabata M, Sugimoto K, Takei Y, Yamakado K, Takeda K, Uemoto S, Isaji S.
Takada T, Hirata K, Mayumi T, Yoshida M, Sekimoto M, Hirota M, Kimura Y, Takeda K, Isaji S, Wada K, Amano H, Gabata T, Arata S, Hirota M, Yokoe M, Kiriyama S, Nakayama T, Otomo K, Tanaka M, Shimosegawa T. Amano H, Takada T, Isaji S, Takeyama Y, Hirata K, Yoshida M, Mayumi T, Yamanouchi E, Gabata T, Kadoya M, Hattori T, Hirota M, Kimura Y, Takeda K, Wada K, Sekimoto M, Kiriyama S, Yokoe M, Hirota M, Arata S. Wada K, Takada T, Hirata K, Mayumi T, Yoshida M, Yokoe M, Kiriyama S, Hirota M, Kimura Y, Takeda K, Arata S, Hirota M, Sekimoto M, Isaji S, Takeyama Y, Gabata T, Kitamura N, Amano H.
Development of nonalcoholic fatty liver disease (NAFLD) and nonalcoholic steatohepatitis (NASH) after pancreaticoduodenectomy: proposal of a postoperative NAFLD scoring system.
Elevated Fibrin-Related Markers in Patients with Malignant Diseases Suspected of Having Thrombotic Disorders. Nomura H, Wada H, Mizuno T, Yamashita Y, Saito K, Kitano S, Katayama N, Yamada N, Sugiyama T, Sudo A, Usui M, Isaji S, Nobori T. Intraductal papillary neoplasm of bile duct detected on fluorodeoxyglucose-positron emission tomography performed for cancer scanning. Kobayashi T, Wada H, Usui M, Sakurai H, Matsumoto T, Nobori T, Katayama N, Uemoto S, Ishizashi H, Matsumoto M, Fujimura Y, Isaji S. Tissue factor expression demonstrates severe sinusoidal endothelial cell damage during rejection after living-donor liver transplantation. Usui M, Kuriyama N, Kisawada M, Hamada T, Mizuno S, Sakurai H, Tabata M, Imai H, Okamoto K, Uemoto S, Isaji S. The impact of total bilirubin on plasma micafungin levels in living-donor liver transplantation recipients with severe liver dysfunction. Assessment of liver graft function and regeneration by galactosyl-human serum albumin (99mTc-GSA) liver scintigraphy in adult living-donor liver transplantation. Iida T, Isaji S, Yagi S, Hori T, Taniguchi K, Ohsawa I, Mizuno S, Usui M, Sakurai H, Yamagiwa K, Yamakado K, Uemoto S. Efficacy of procalcitonin in the early diagnosis of bacterial infections in a critical care unit. Nakamura A, Wada H, Ikejiri M, Hatada T, Sakurai H, Matsushima Y, Nishioka J, Maruyama K, Isaji S, Takeda T, Nobori T.
Liver retransplantation with external biliary diversion for progressive familial intrahepatic cholestasis type 1: a case report. Usui M, Isaji S, Das BC, Kobayashi M, Osawa I, Iida T, Sakurai H, Tabata M, Yorifuji T, Egawa H, Uemoto S. The effectiveness of measuring for fragmented red cells using an automated hematology analyzer in patients with thrombotic microangiopathy.


Abe Y, Wada H, Yamada E, Noda M, Ikejiri M, Nishioka J, Kobayashi T, Matsumoto T, Masuya M, Isaji S, Usui M, Uemoto S, Katayama N, Nobori T. Kuriyama N, Isaji S, Hamada T, Kishiwada M, Ohsawa I, Usui M, Sakurai H, Tabata M, Suzuki K, Uemoto S. Primary non-Hodgkin's lymphoma of the gallbladder diagnosed by laparoscopic cholecystectomy. Regulatory mechanisms of C4b-binding protein (C4BP)alpha and beta expression in rat hepatocytes by lipopolysaccharide and interleukin-6. Hepaticojejunostomy by interventional radiology with an expandable metallic stent after extended right hepatectomy for hilar cholangiocarcinoma: case report. Successful laparoscopic splenectomy after living-donor liver transplantation for thrombocytopenia caused by antiviral therapy. Early-stage hepatocellular carcinoma: radiofrequency ablation combined with chemoembolization versus hepatectomy. Yamakado K, Nakatsuka A, Takaki H, Yokoi H, Usui M, Sakurai H, Isaji S, Shiraki K, Fuke H, Uemoto S, Takeda K. Survival rates according to the Cancer of the Liver Italian Program scores of 345 hepatocellular carcinoma patients after multimodality treatments during a 10-year period in a retrospective study. Yamagiwa K, Shiraki K, Yamakado K, Mizuno S, Hori T, Yagi S, Hamada T, Iida T, Nakamura I, Fujii K, Usui M, Isaji S, Ito K, Tagawa S, Takeda K, Yokoi H, Noguchi T.
Use of an interpositional venous graft posterior to the pancreas for LDLT patients with portal vein thrombosis.
Usefulness of breast-volume replacement using an inframammary adipofascial flap after breast-conservation therapy. Experiences and problems pre-operative anti- CD20 monoclonal antibody infusion therapy with splenectomy and plasma exchange for ABOincompatible living-donor liver transplantation.
Primary liposarcoma of gallbladder diagnosed by preoperative imagings: a case report and review of literature. Impact of venous drainage on regeneration of the anterior segment of right living-related liver grafts. The role of living-donor liver transplantation in surgical treatment for hepatocellular carcinoma.
Are the results of surgical treatment of hepatocellular carcinoma poor if the tumor has spontaneously ruptured? Japanese multiinstitutional study of intraductal papillary mucinous tumor and mucinous cystic tumor. Metalic stent placement in the pancreatic duct: an experimental study in the normal dog pancreas. Management of advanced pancreatic cancer: staging laparoscopy and immunotherapy- a new treatment strategy. Changes in serum hyaluronic acid levels and expression of CD44 and CD44 mRNA in hepatic sinusoidal endothelial cells after major hepatectomy in cirrhotic rats.
Refractory biliary leak from intrahepatic biliaryenteric anastomosis treated by selective portal vein emobolization. Clinical and genetic analysis of noncancerous and cancerous biliary epithelium in patients with pancreatico- biliary maljunction. Liver injury in acute pancreatitis and mitigation by continuous arterial infusion of an antibiotic via the superior mesenteric artery. Portal vein stent placement in patients with pancreatic and biliary neoplasms invading portal veins and causing portal hypertension. Pancreatic head resection with second-portion duodenectomy for benign lesions, low-grade malignancies, and early stage carcinomas involving the pancreatic head region.
A different kind of PREbiotic dietary supplement, Good Gut Daily, has recently entered the market. Polyphenols are naturally occurring compounds found in plants – including fruits, vegetables, tea, coffee, and wine. I’ll be writing about Good Gut Daily in more depth in an upcoming post but, in the interest of not overwhelming you with information, I thought it useful to do a preliminary post on some of the causes of increased intestinal leakiness so you can see how your GI problems originated and how poor gut health creates major health problems elsewhere in your body. This post grew out of a phone and email conversations with molecular biologist Rob Wotring, the Chief Scientific Officer at Greenteaspoon. Damage to the gut’s mucosal layer leads to a whole range of serious problems as the body tries to cope with the invaders being released into the bloodstream. For an easy to understand explanation of increased gut permeability, see Leaky Gut Syndrome in Plain English – and How to Fix It. Leaky gut syndrome is not generally recognized by conventional physicians, but evidence is accumulating that it is a real condition that affects the lining of the intestines. Infections (eg, acute viral or bacterial infection, intestinal parasites, HIV, candida, etc)  that damage the integrity of the intestinal mucosal lining are  the most common causes of increased gut permeability.
Since intestinal barrier dysfunction is known to predispose the development of intestinal diseases  as well as autoimmune diseases in other parts of the body, it is highly important that infants’ intestinal barriers be allowed to receive the health benefits of breast milk so they mature properly. In the elderly, epithelial stem cells mutate more frequently, leading to thinning of the mucosal lining. The observation that gut and lung disorders commonly occur together has led GI and respiratory researchers to think they share a common cause.
In a 2010 paper appearing in the National Review of Gastroenterology and Hepatology, neurogastroenterologist Nicholas Talley and his colleagues observed that people with asthma and allergic rhinitis have abnormally high levels of eosinophils in both their airways and their intestines. In allergies, these eosinophilic cells start growing in the lungs and airways and the ones in the GI tract stop serving their protective function and instead damage the gut’s mucosal lining, allowing toxins to leak through.
Alcohol damages the delicate lining of the stomach and intestinal tract as it passes through, creating increased permeability.
Emulsifiers are chemicals or natural substances that encourage the suspension of one type of liquid in another – as in the oil and water in margarine, shortening, ice cream, salad dressings, and creamy sauces. Emulsifiers are added to commercial breads and cakes, icings, frozen desserts, soups, mayonnaise, homogenized milk, whipped toppings, non-dairy creamers, chocolate bars, chew candies, bubble gum, extruded snacks, soft drinks, bottled liquid coffees … and many other processed foods. Yet there is evidence that these emulsifiers disturb the colonies of probiotic bacteria living in the colon, increasing the risk of inflammatory bowel diseases and metabolic disorders. Could adding emulsifiers to food products to make them look more appealing and ‘last’ longer possibly be worth ruining our gut linings and increasing our risk for developing one or more autoimmune diseases?
Aspirin, ibuprofen and naproxin are common NSAIDs (non-steroidal anti-inflammatory drugs) available OTC for use as pain relievers. An estimated 16,500 Americans die each year from and 100,000 are hospitalized with NSAID-induced complications. It takes NSAIDs such as asprin, ibuprofen, Advil, Motrin, Aleve only 15-30 minutes to create lesions in the mucosal layer of the GI tract! Japanese researchers found small bowel injuries occurring in 80% of their study participants after only two weeks on aspirin therapy. After many decades of promoting an aspirin a day to prevent heart attacks, the FDA has now reversed its position.
Hopefully this news will change the behavior of the 40 million Americans who take an aspirin every day. Many people experience nausea, heartburn, cramping, and diarrhea while exercising – especially during high-intensity exercise.
When the body is at rest, your heart directs 20-25% of its pumped blood  to your digestive tract. The harder or longer you run or exercise, the less blood gets delivered to your gut, causing digestion to slow. Runners, cyclists and triathletes tend to get diarrhea after 30-60 minutes of intense exercise. Even worse, exercising can damage the gut’s mucosal lining and cause increased gut permeability. Anyone who participates in physical exercise is at risk for injury and illness arising from such activity…. There is a very high prevalence of gastrointestinal (GI) complaints during exercise among long-distance runners, triathletes and athletes involved in other types of strenuous long-lasting exercise.
Damage to the gut and impaired gut function is associated with increased of intestinal permeability after a marathon. When the body is in an overheated state, some of the blood that normally flows to the intestines gets diverted to the skin and the temperature inside the intestines increases.
This combination damages  the intestinal barrier, creating increased intestinal permeability to microbial endotoxins (toxins  present inside a bacterial cell that get released when the cell disintegrates),  leading to endotoxemia (the presence of endotoxins in the blood).
Be careful when exposing yourself to high heat for extended periods of time (eg, while tanning all day at the beach, taking a long sauna, engaging in intense exercise). There’s convincing evidence that polyphenol PREbiotics (as in Good Gut Daily) are able to heal damage in the gut lining. And finally, next to the lamina propria, the MUSCULARIS MUCOSA, a thin, double layer of smooth muscle responsible for local movement of the mucosa. The layer next to the mucosa is the SUBMUCOSA, a loose connective tissue layer containing larger blood vessels, lymphatics, and nerves. The outermost layer is the ADVENTIA (OR SEROSA) consisting of loose connective tissues containing blood vessels, lymphatics, and nerves. DISCLAIMER:  Nothing on this site or blog is intended to provide medical advice, diagnosis or treatment. Aside from being a cool name for a band (“Dude, Gut Flora totally ROCKS!”), gut flora have three important functions: protective, structural, and metabolic.
Structural functions of your intestinal bacteria include barrier fortification, tightening of “tight” junctions, induction of immunoglobulin A and immune system development.
Barrier fortification means that the bacteria provide a physical barrier between your insides (blood vessels of the gut) and the outside (lumen of the gut). Another structural protective function of the bacteria is to make the space between the cells of the gut even smaller (aka tightening junctions).
Induction of immunoglobulin A (IgA) and immune system development by your gut bacteria are inter-related.
The article I’m reviewing this week is hot off the press –- actually it hasn’t been published on paper yet, just online –- and it compares blood metabolites in mice without any bacteria (germ free) and “conventional” mice with an average amount of gut bacteria.
The methods are pretty straightforward: Researchers compared blood serum of conventional or normal mice compared to blood serum of mice without gut bacteria (germ free – GF). The conventional group had 145 additional biochemicals and the bacteria-free had 52 different biochemicals. Of the 197 different biochemicals only 19 were significantly different (statistically speaking). A number of glycine conjugates (products of chemical reactions), phenylpropionylglycine and phenylacetylglycine, were found only in conventional mice, and in amounts 3.8 times higher, respectively.
In recent years, people have been paying more attention to the health of their gut and what organisms are living in there. Give credit for this revolution to Barry Marshall, who discovered the cause for stomach ulcers and gastritis was a bacterium (Helicobacter pylori) in 1984.6 Taking one for the science team, he infected himself with the bacteria -– by drinking a beaker of H. While disease and health have been linked to gut bacteria this study goes a step further and looks at the effect of those bacteria on your metabolism.
Hopefully in the future we will be able to pinpoint which bacteria are responsible for what metabolites and prescribe specific bacterial cultures, but until then make sure that you keep your gut bacteria happy by using probiotics and live culture yogurts, especially during and after rounds of antibiotics and anti-inflammatories. To learn more about making important improvements to your nutrition and exercise program, check out the following 5-day video courses. They’re probably better than 90% of the seminars we’ve ever attended on the subjects of exercise and nutrition (and probably better than a few we’ve given ourselves, too). From staph infections to pneumonia, strep throat to urinary tract infections, we can't help but feel the bacteria around us are alien invaders, designed to hurt, maim or kill us. Our microbiome -- the collection of microbes and their genetic material that live in and on the body -- is packed so full of bacteria, viruses and even fungi that our human cells make up just 10 percent of the cells in our body [source: Yang]. But even with the enormous density and importance of our microbiome, we really know little about it. So gather your bacteria, your fungi, your archaea and protozoa by the fire to learn about how the Human Microbiome Project works. And it’s not just the oil that works wonders – coconut water is getting a great rap, too, in the beauty stakes!! 7.  They say that drinking coconut water makes your skin look younger and more vibrant, as it aids in cleansing toxins naturally and erases any harmful toxic build up.
9.  To make coconut scrub to hydrate your skin, mix 1tbsp of coconut water and lentils into a paste.
About Glam Beauty BarWelcome to Glam Beauty Bar, one of the best, beautiful & most unique Beauty & Make Up Salons in Moonee Ponds. With several treatment rooms and a team of fun loving Beauty Therapists, we waste no time in serving you up an effective treatment that will get you looking and feeling beautiful the moment we are finished with you! Five days after the birth of my third child, Hugo, I was diagnosed with seven different bacterial infections.
Unfortunately, we do not have any naturopathic or holistic hospitals in Vancouver, so I had to check into a regular medical hospital BUT I agreed to do so only if my MD brother would oversee my treatment – thus ensuring my safety.
The first thing he did was to make them culture out the infective bacteria to identify the pathogens and see WHICH antibiotics were effective against the pathogens. Next, my brother Millan researched the drugs they said I should receive for 10 days and discovered plenty of recent research showing a 7-day course was just as effective, so my treatment time was cut to seven days, rather than ten.
Lastly, I brought in my own 100% cotton bedding and used a lambskin (from Ikea) for a mattress pad – this keeps you from breaking out in bed sores or pressure sores, the wool regulates your body temperature and it is just really comfortable. The woman in the bed next to me could not use the toilet, otherwise, I would have had to take extra precautions to ensure I didn’t pick up any infections from her feces.
When I checked out after a week in the hospital on IV antibiotics, my lungs still weren’t completely clear of fluid.
Something to keep in mind: Not only did  the infection clear completely and the fluid drain completely from my lungs, but there is also no residual scarring.


Unless you can find an integrative MD, all these therapies are available through a naturopathic (or perhaps an osteopathic doctor). The only side effect is the IV hydrogen peroxide can sometimes cause a temporary headache in susceptible people. I’m wary about trying hydrogen peroxide orally since my intuitive feel is that it might be too harsh for the GI tract. I had a bad case of mononeucleosis and did one Hydrogen Peroxide IV and one 25 gram Vit C IV 3 days later. Unfortunately we do not have much information about doctors in various localities who do this but any full-service Naturopathic Doctor would be able to.
Preservation of the pancreatic function via the blood supply from the posterior epiploic artery: report of a case. The empty space in the center, just below the villi (the spikes you see in the image of a healthy mucosal membrane in the image to the left above),  is called the lumen, the tube in which food travels through the intestines. This is where our probiotic bacteria live, so degrading it also degrades the strength of our immune systems.
Once this lining has become disturbed, allowing problematic things to flow through it into the blood stream, a cycle of chronic irritation begins, leading to chronic inflammation in the body and a whole series of autoimmune conditions.
The theory is that leaky gut syndrome (also called increased intestinal permeability), is the result of damage to the intestinal lining, making it less able to protect the internal environment as well as to filter needed nutrients and other biological substances. In the mucous membrane lining the stomach, small intestine and colon, their purpose is to prevent pathogenic bugs and toxins from escaping through the gut walls and getting into the body. This increased porosity permits large, incompletely digested food particles to move through the gut walls directly into the bloodstream, where immune cells regard them as foreign invaders and attack them with specially designed antibodies.
Multiple studies have compared the allergic responses of alcoholics, depressive, and schizophrenic patients, and found that the alcoholic group was significantly more allergic to a variety of food allergens. Other studies have noted GI damage in people on low-dose aspirin therapy taken for cardiovascular protection. While even moderate exercise increases your heart rate and therefore the amount of  blood  being pumped from your heart, the amount of blood flowing to the GI tracts gets decreased by as much as 60-70% and is instead diverted to your muscles, heart, lungs, and brain.
A serious underperfusion of the gut often leads to mucosal damage and enhanced permeability so as to hide blood loss, microbiota invasion (or endotoxemia) and food-born allergen absorption (with anaphylaxis)…. These GI complaints occur because of the redistribution of the blood flow, that is shunted from the viscera to skeletal muscle, heart, lung and brain…. Some of the symptoms, however, can be life-threatening, such as blood loss in feces in the hours following the running presented by some marathoners and long-distance triathletes. Moreover, vigorous exercise (jogging, aerobics, dancing, tennis, bicycling, racquetball, swimming, and skiing) facilities allergen absorption from the GI tract, leading to a food-dependent exercise induces anaphylaxis (FDEIA). There are usually two sub-layers of smooth muscles in the muscularis propria: An inner circular layer and an outer longitudinal layer. In order to post comments, please make sure JavaScript and Cookies are enabled, and reload the page.
You have hundreds of trillions of micro-organisms in your body, while you have only a measly ten trillion cells. Luckily, all those bacteria only weigh about 2 kg, so just think of it like carrying around a well-loaded purse made up of little wiggly things. Just like a rowdy gang, your gut bacteria crowd out other micro-organisms, take all the food and make biological weapons (anti-microbials) to keep the pathogens out and you healthy. While cells of the gut are already very close to one another (aka tight junctions) the closer they are to each other the fewer unwanted things pass through. IgA is a protein (specifically an antibody) that binds to foreign or bad things like antigens (foreign proteins), food, bacteria and viruses so that these “bad” things aren’t absorbed into the gut cells. This includes helping with the breakdown of non-digestible foods (or parts of foods), metabolizing carcinogens, making vitamins (biotin and folate) and absorbing ions (magnesium, calcium and iron). Metabolomics analysis reveals large effects of gut microflora on mammalian blood metabolites. Using high performance liquid chromatography (HPLC) and mass spectrometry (MS) they identified biochemicals (or metabolites) in the serum of the mice. Oh, and if you’re stressed it wouldn’t hurt to hedge your bets and keep restocking your body with “good” bacteria – there was a reason ulcers flared up when people were stressed. And if germ warfare has been officially declared, we must do nothing short of destroy our bacterial foils, creating a world of sterile peace through Purell. If you gasped to learn that Escherichia coli strains are living on your kitchen counter, you're going to be really upset when you realize they're also happily living in your gut.
While we're aware that something like an abundance of the bacteria Clostridium difficile in the gut can cause fatal health issues, we're not sure what a healthy gut looks like. You can massage coconut oil into your skin to maintain its suppleness, but you should also take a tablespoon each day in its uncooked, raw form.
Used as a skin moisturiser,  coconut oil nourishes and hydrates your skin by replenishing your natural skin oils. A common cause for skin problems and dehydration is a build up of toxins, fungi and bad bacteria.
Coconut oil will make your hair shine and glow by bringing out your natural vibrancy and beauty. At our salon, we offer the best skin treatments, hair removal, lashes, cosmetic injectables, beauty & tanning treatments at a very affordable price!
Since I was on IV antibiotics, I didn’t worry about scheduling (normally you would take probiotics 2 hours after antibiotic dose), but just took 1 tsp. But that was just too hard on all of us and so I had Ian bring them, but from the second they stood outside the hospital door, they had to clasp their hands behind their backs and were not allowed to touch anything (no door handles, elevator buttons, chairs, counters, etc.). This is the heavy-hitter antibiotic and although it gave me headaches on the day of treatment, it was well worth it.
It is now 4 years later and I have also not had pneumonia or any other bronchial infection occur during the winter. All recommendations are based foremost upon an honest belief that the product, service, or site will benefit my site visitors in some way. This kind is polyphenol-based and has  been clinically shown to calm acute digestive symptoms in as little as 30 minutes and enhance immune health. It then travels down through the esophagus to the stomach and from there into the small and large intestines, where it is selectively digested.
On average, the small intestine in adults is 22 feet long and the large intestine is 5 feet.
The probiotics residing in the gut mucosal layer make up 70-90% of the human immune system. Ulcerative colitis occurs when the immune system reacts aggressively against the normal bacteria inhabiting the colon – ie, it is an autoimmune process.
In young babies, the barrier function is underdeveloped so large amounts of big molecules get through the gut mucosal layer and enter circulation in the body.
A similar study compared patients admitted to an inpatient alcoholism hospital with a matched control group of patients with no history or evidence of alcohol abuse who have been admitted to a general hospital for elective surgery. ALL NSAIDs cause injury in the GI tract: erosions, ulcers, bleeding and perforations in the stomach and intestines. The two layers work together to produce peristalsis ((rhythmic waves of contraction) to move food through the gut. Intestinal Barrier Dysfunction, Endotoxemia, and Gastrointestinal Symptoms: The ‘Canary in the Coal Mine’ during Exercise-Heat Stress? Food preservatives linked to obesity and gut disease: Mouse study suggests that emulsifiers alter gut bacteria, leading to the inflammatory bowel condition colitis.
If you really want to lose 2 kg, just mentally subtract your bacteria since it really isn’t “you” anyway. The “cardboard toilet paper roll” (or the walls of your GI tract) isn’t solid, but rather slightly permeable, in order to let nutrients through.
The most well known function of your gut bacteria is the breakdown of non-digestible or partially digestible foods, such as beans, by the process of fermentation that leads to socially awkward situations, especially if a dog is not nearby to blame. Now, I know you aren’t a mouse –- unless you’re Brain from “Pinky and the Brain.” But it is really hard to find people without any gut bacteria that are willing to eat exactly the same thing and it’s even harder to find a group of people that are nearly genetically the same –- so we’re stuck with mice. Even then it was ten years before antibiotics became the standard treatment for ulcers and gastritis.
Unidentified curved bacilli in the stomach of patients with gastritis and peptic ulceration.
If you feel squeamish about the live cultures of Lactobacillus in your yogurt, you're probably going to be unhappy to know there's a thriving population colonizing your vagina (or that of someone near and dear to you).
Accordingly, scientists are trying to not just determine how our microbial friends can help or harm us, but also to establish a baseline for a normal microbiome.
Certified organic, one hundred per cent pure and raw virgin coconut oil makes a fantastic do-all beauty product.
It helps to cleanse and balance your healthy gut flora, which is thought to minimise the outbreaks of skin rashes,  and irritations. It contains Medium Chain Fatty Acids (MCFA’s), which are also found in your natural skin oils.
Coconut oil helps to cleanse and neutralise these toxins,  fungi and bacteria on the outer layers of your skin, which not only detoxifies but also helps to build your skins natural immune system and protection.
Simply mix a few drops of coconut water with a few drops of lemon and then apply it to your skin. This means you could end up cycling through numerous antibiotic drugs until they find the one(s) that work.
In retrospect, it probably would have been easier to get them to put on a pair of disposable surgical gloves (available at any pharmacy). Here’s a very detailed instruction manual for all methods of using hydrogen peroxide.
For those of you who, like me, suffer from ongoing digestive health problems and haven’t found a satisfactory solution, the arrival of this new supplement is excellent news.
During this trip, various phases of digestion take place  and nutrients are extracted and absorbed.
This triggers an autoimmune reaction, which can lead to gastrointestinal problems such as abdominal bloating, excessive gas and cramps, fatigue, food sensitivities, joint pain, skin rashes, and autoimmunity. This makes infants susceptible to infectious diarrhea, necrotizing enterocolitis (the lining of the intestinal wall dies and the tissue falls off), and allergic gastroenteropathy. Most alcoholics are allergic to a wide range of foods as well as environmental-mental allergens. It may be hard for you to think of the inside of your intestines as being your outside, but it is -– in the simplest terms you are a tube with your gastro-intestinal (GI) tract running though you.
But these walls can’t be too easily passed; otherwise stuff that you don’t want in your bloodstream may leak through. Much like the Human Genome Project that mapped the entirety of human genes, the Human Microbiome Project is looking to identify the whole of our microbial landscape. Obviously, this is not a good thing since drug antibiotics create a lot of collateral and systemic damage. I also did not touch the hospital food and instead drank Absorb Plus shakes (with all the healing supplements added as recommended in The IBD Remission Diet) and homemade broth for all my meals, along with Emergen-C.
Be VERY careful when handling 35% hydrogen peroxide as it will burn your skin instantly on contact. The liver, gall bladder and pancreas, organs that aid in the digestive process, are located along the length of the GI tract.
The cause of this syndrome may be chronic inflammation, food sensitivity, damage from taking large amounts of nonsteroidal anti-inflammatory drugs (NSAIDS), cytotoxic drugs and radiation or certain antibiotics, excessive alcohol consumption, or compromised immunity. Among foods, grains (the primary ingredient of many alcoholic beverages) are highly reactive. They mostly live in the gut or intestines of your body, while about 10% of them live on your skin.
So, your GI tract is a part of the outside world just like the centre of a toilet paper roll is not toilet paper. Not to mention the extra time you’ll spend in a very unhealthy (infective) environment. The Absorb Plus was doubly needed since my mouth had broken out in painful ulcers from the antibiotics. If it’s in a plastic bottle, then direct the opening away from you when you open it, in case it spits some out from the pressure build-up. The outer bacterial environment on the skin helps prevent the growth of other micro-organisms such as those that produce yeast and fungal infections. Stuff that you ingest must interact with the “inside outsides” of you (the cardboard layer of the toilet paper roll) before getting through to the “inside insides” of you (the inner paper layers). The inner bacterial environment – known as “gut flora” – has a similar function, but also does lots of other things.
Relatively looser spaces between gut lining cells allow large food particles past into your bloodstream. It helps to cleanse and balance your healthy gut flora, which is thought to minimise the outbreaks of skin rashes, and irritations. Used as a skin moisturiser, coconut oil nourishes and hydrates your skin by replenishing your natural skin oils. Coconut oil helps to cleanse and neutralise these toxins, fungi and bacteria on the outer layers of your skin, which not only detoxifies but also helps to build your skins natural immune system and protection. You can also use coconut water as a facemask - thoroughly soak cotton pads in the coconut water and apply them to your face.



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30.10.2015, author: admin


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