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In our laboratory, we study the central mechanisms involved in the sensory-motor transformations of visual and vestibular information that are relevant to maintain image stability while an animal moves around in a dynamic environment.
We conduct translational research developing pharmacologic otoprotective agents to prevent or procide resuce from noise-induced hearing loss and tinnitus, to prevent cisplatin-induced hearing loss and both central and peripheral neuropathy, radiation-induced oral mucositis, and aminoglycoside induced hearing loss and vestibular disorders. Our laboratory is focused on the pathogenesis of chronic otitis media and cholesteatoma, which is characterized by localized bone resorption and keratinocyte proliferation.
Understand how perceptual and cognitive abilities interact to determine relative benefits of sensory devices for children with moderate to profound sensorineural hearing loss. Our lab is interested in the cellular and molecular mechanisms that modulate sensitivity and temporal acuity in the auditory pathway and how deficits in these mechanisms lead to functional losses. Developmental changes in speed of information processing across the life span and on the role of age-related difference on other aspects of cognition.
Our laboratory is interested in the study of innate immunity in the inner ear and investigates the role of monocytes and macrophages in various models of hearing loss and cochlear injury. My lab studies basic cell biology of the tight junction in the kidney, the inner ear and the brain vasculature. My research focuses on the bone morphogenetic protein (BMP) signaling pathway as it relates to oral cancer.
My major area of interest is directed towards understanding the mechanisms by which Fibroblast Growth Factors (FGFs) regulate organogenesis, tissue homeostasis and response to injury.
In the Rutherford lab, we investigate questions of cellular auditory neurobiology with techniques of molecular anatomy and electrophysiology.
One goal of our laboratory is to better understand the underlying pathophysiology associated with these disorders. Research focus on spoken language processing, with an emphasis on age-related changes in speech perception.
Biochemical characterization of tissues and fluids of the mammalian inner ear; functional role of inner ear proteins in ionic homeostasis, transduction and otoconial formation. Research interests include audiovisual speech recognition and audiovisual integration in both adults and children, auditory training, and communication breakdowns in acute medical care conditions. The initial primary purpose of WUHARL was to create an environment allowing the Division of Adult Audiology at Washington University in St.
A current research focus involves relating our behavioral findings concerning the noncompetitive NMDA receptor antagonist MK-801 with our neurotoxic findings associated with this compound.
Key to this sensory-motor transformation are the cerebellum and its target neurons in the brainstem. The focus of the current laboratory studies include optimizing D-methionine dosing to prevent and provide rescue from noise-induced hearing loss and tinnitus, to determine the maximum length of time that pharmacologic intervention can be effective after noise cessation, and to determine optimal D-methionine dosing in preparation for clinical trials to prevent aminoglycoside induced hearing loss and vestibular disorders.
Pseudomonas aeruginosa is an opportunistic pathogen capable of forming biofilms and is the most common organism isolated from infected cholesteatoma.
Research is focused on developing and testing a model of the sensory, perceptual, and cognitive mechanisms that mediate vocabulary acquisition in hearing-impaired children. We are particularly interested in molecules that have physiologic roles in normal auditory signal transduction but engender pathologic responses due to inflammation, noise and ototoxin exposure and aging.
A structure-function approach which combines electrophysiological measure, gene and protein expression and morphological analysis. Human recombinant BMP therapies have already been FDA-approved as an alternative to bone grafts for orthopaedic and orodental applications. My laboratory discovered that mice lacking FGF receptor 3  are deaf and that FGFR3 is essential for formation of Pillar Cells and Dieters cells within the organ of Corti. Piccirillo is the Director of the Clinical Outcomes Research Office, a research laboratory that uses clinical epidemiology methodology to study the diagnosis, treatment, and prognosis of otolaryngology-related diseases. We aim to elucidate the function of unique protein complexes that underlie mechanosensory perception in mechanisms of synaptic transmission and action potential generation in the ear.
We showed previously that syngeneic bone marrow transplantation (see below) and direct protein replacement can prevent many of the clinical signs of disease in MPS VII mice.

Louis School of Medicine to independently evaluate newly introduced hearing aid technologies.
Our lab is particularly interested in the role of this cerebellum-brainstem loop in the acquisition and consolidation processes that lead to the formation of new motor memories in the vestibulo-oculomotor system.
She has successfully completed clinical trials to prevent cisplatin- induced hearing loss and radiation induced oral mucositis.
Utilizing a wide variety of functional, anatomical and molecular techniques, we are currently investigating the roles of the synuclein family of proteins in peripheral and central auditory processing. The application of BMP therapies for bone regeneration after tumor resection is limited by the unknown effects of these morphogens on oral squamous carcinoma cells (SCCA). In our pursuit of FGF ligands that may be important forinner ear development, we have focused on the FGF9 sub family. Areas of study include: cortical neural network abnormalities among patients with tinnitus, rTMS for tinnitus, chemotherapy-induced cognitive impairment, comorbidity among cancer patients, dizziness, and sleep apnea. Modern industrial wind turbines generate high infrasound levels that affect people living nearby. We have characterized the cognitive deficits using a battery of behavioral assays, the seizure frequency using electroencephalography, the retinal dysfunction using electroretinography and the hearing deficits using auditory-evoked brain stem responses. More recently we showed that injection of an adeno-associated viral (AAV) gene transfer vector directly into the brain can correct some of the cognitive deficits associated with several of these diseases.
This in turn would enhance the services and knowledge of our Audiologists and physicians in providing the best hearing aid healthcare to our patients. Our work has shown that alpha- and beta-synuclein work in a compensatory fashion to modulate hearing sensitivity. As compared to the cochlear base, less is known about the cochlear apex because, in part, conventional electrophysiologic techniques such as otoacoustic emissions and compound action potentials perform adequately only at high-frequencies above 1 kHz or so.
Studies are being performed to determine whether the BMP signaling pathway is functional in oral squamous cell carcinoma. We initiallygenerated a FGF9 knockout mouse and in collaboration with another group showed that these mice have defects in inner ear development that could be attributed to defects in mesenchymal development. We are also pursuing several recent observations that these mouse models of LSD have immune deficits, suffer from chronic inflammation and have profound secondary metabolic abnormalities. We showed that injection of an AAV vector directly into the vitreous of the eye can improve retinal function. In addition, our desire was to share this knowledge with Audiologists and hearing aid manufacturers around the United States and the world via presentations at conferences and submissions of the results of our work to peer and non-peer reviewed journals as well as textbooks or chapters within textbooks.
Further, over-expression of alpha-synuclein can actually enhance the acuity of central auditory processing as well as protect the inner ear from oxidative damage.
Understanding low-frequency hearing and hearing loss is important because speech vowels and many bothersome environmental background noises are of low frequency.
Interestingly, the therapeutic enzyme was also transported axonally from the eye into the CNS and corrected the disease in specific areas of the brain. This research will elucidate the safety of using BMPs for bone regeneration in oral cancer defects, and determine whether these proteins or inhibitors of these proteins may additionally have a therapeutic role in treating head and neck squamous cell carcinomas. Finally, encouraging results from a pre-clinical experiment using a human immunodeficiency virus (HIV)-based gene transfer vector in hematopoietic stem cells from an MPS VII patient have led to a pending clinical trial for this disease.
FGF20 knockout mice have normal inner ear anatomy but have severe cochlear dysmorphology and selective loss of outer hair cells in the organ of Corti.
We are very excited about this finding given the importance of outer hair cells in age related hearing loss.

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