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To request an appointment at NYOG by email, please give us your contact information in the form below and we will get back to you shortly with available dates. For over 17 years, the NYOG has specialized in the ear, nose and throat, and ranks among the country's leading diagnostic and treatment centers for otolaryngology-related illnesses. Taking care of your hearing is important, especially since we now live in such a noisy world. There are many things you can do, like wearing earplugs when you know you are going to be in a situation where noise levels are extreme, or avoiding these situations altogether, as well as being responsible with the volume levels of your music devices and television. Continued exposure to these and other excessive environmental noise is a major contributor to hearing loss. If you are experiencing hearing loss in one or both ears, or if you have a constant ringing [also known as tinnitus (tin-NY-tus)] or any pain or ache, come in to see us.
There are two different types of hearing impairments, conductive hearing loss and sensorineural hearing loss. A conductive hearing loss is present when the sound is not reaching the inner ear, known as the cochlea.
Dysfunction of the three small bones of the middle ear, the hammer, anvil and stapes may result in conductive hearing loss. A sensorineural hearing loss is one resulting from dysfunction of the inner ear, the cochlea, the nerve that transmits the impulses from the cochlea to the hearing center in the brain or damage in the brain. Another common reason for hearing loss due to hair cell damage is noise-induced hearing loss. Total or near-total sensorineural deafness may be the result of congenital malformations, head trauma, inner ear infection, genetic factors, or long history of excessive noise exposure. As an overview, low frequency (or tone) sensorineural hearing loss (LF SNHL or LT SNHL) is unusual, and mainly reported in Meniere's disease as well as related conditions such as low CSF pressure.
Le Prell et al (2011) suggested that low frequency hearing loss was detected in 2.7% of normal college student's ears. Oishi et al (2010) suggested that about half of patients with LTSNHL developed high or flat hearing loss within 10 years of onset. The new international criteria for Meniere's disease require observation of a low-tone SNHL.
Gatland et al (1991) reported fluctuation in hearing at low frequencies with dialysis was common, and suggested there might be treatment induced changes in fluid or electrolyte composition of endolymph (e.g.
Although OAE's are generally obliterated in patients with hearing loss > 30 dB, for any mechanism other than neural, this is not always the case in the low-tone SNHL associated with Meniere's disease. The lack of universal correspondence between audios and OAE's in low-tone SNHL in at least some patients with Meniere's, suggests that the mechanism of hearing loss may not always be due to cochlear damage. Of course, in a different clinical context, one might wonder if the patient with the discrepency between OAE and audiogram might be pretending to have hearing loss. Papers are easy to publish about genetic hearing loss conditions, so the literature is somewhat oversupplied with these.


Bespalova et al (2001) reported that low frequency SNHL may be associated with Wolfram syndrome, an autosomal recessive genetic disorder (diabetes insipidus, diabetes mellitus, optic atrophy, and deafness). Bramhall et al (2008) a reported that "the majority of hereditary LFSNHL is associated with heterozygous mutations in the WFS1 gene (wolframin protein). Eckert et al(2013) suggested that cerebral white matter hyperintensities predict low frequency hearing in older adults. Fukaya et al (1991) suggested that LFSN can be due to microinfarcts in the brainstem during surgery. Ito et al (1998) suggested that bilateral low-frequency SNHL suggested vertebrobasilar insufficiency.
Yurtsever et al (2015) reported low and mid-frequency hearing loss in a single patient with Susac syndrome.
There are an immense number of reports of LTSNHL after spinal anesthesia and related conditions with low CSF pressure.
Fog et al (1990) reported low frequency hearing loss in patients undergoing spinal anesthesia where a larger needle was used. Waisted et al (1991) suggested that LFSNHL after spinal anesthesia was due to low perilymph pressure. Wang et al reported hearing reduction after prostate surgery with spinal anesthesia (1987). We have encountered a patient, about 50 years old, with a low-tone sensorineural hearing loss that occured in the context of a herpes zoster infection (Ramsay-Hunt). LTSNHL is generally viewed as either a variant of Meniere's disease or a variant of sudden hearing loss, and the same medications are used.
Classic stigmata of congenital syphilis SNHL, interstitial keratitis, Hutchinson teeth (notched incisors), mulberry molars, Clutton joints (bilateral painless knee effusions), nasal septal perforation & saddle deformity& frontal bossing.
NYOG physicians have consistently been recognized among the city's foremost doctors within their areas of specialization. Whether you live in a big city, like New York City, the suburbs or a rural area, technology can provide many opportunities for damaging your hearing. Whether environmental, health-related or genetic, our docs will get to the root of the problem and provide solutions. The mobility of these bones, or ossicles, may be impaired for different reasons and disruption of the ossicular chain due to trauma, infection or other process may also result in hearing loss. Chronic ear infection can result in a defective eardrum or middle ear cavity, and often in addition to the conuctive hearing loss the individual may have a sensorineural hearing impairment. To us, this seems a little overly stringent, compared to the earlier 1995 AAO criteria that just required any kind of hearing loss, but criteria are better than no criteria. That being said, genetic conditions should not be your first thought when you encounter a patient with LTSNHL. One would think that diabetes insipidus is so rare that hearing loss is probably not the main reason these individuals are seeking attention.


These patients are extremely rare because there isn't much microsurgery done close to the brainstem. We are dubious about the conclusion of this paper because it is different than nearly every other paper in the literature. We are dubious that this has any applicability to humans and we have never seen a patient with lithium toxicity with any LFSNHL.
We have observed this occasionally ourselves, but of course, the usual pattern is a conductive hyperacusis. We find this somewhat plausible as pregnancy and delivery might be associated with spinal anesthesia, with changes in collagen, and with gigantic changes in hormonal status.
Skeletal findings osteochondritis& periostitis of long bones SNHL, interstitial keratitis, Hutchinson teeth (notched incisors), mulberry molars, Clutton joints (bilateral painless knee effusions), nasal septal perforation & saddle deformity& frontal bossing. Staffed by an exceptional team of board-certified physicians and specialists, the NYOG adheres to a comprehensive disease management approach to ensure its patients receive the best possible care of their sinus, head and neck, hearing and balance, speech and swallowing, and sleep apnea conditions.
We are here to answer any and all of your questions because at NYOG…we are here to clear things up.
Hearing impairments are categorized by their type - conductive, sensorineural or both, by their severity, and by the age of onset. This will often interfere with speech understanding, as it is in the high frequency range that we find the consonant sounds that are most important especially in noisy surroundings. There is a large literature about genetic causes, mainly in Wolfram's syndrome ( diabetes insipidus, diabetes mellitus, optic atrophy, and deafness), probably because it is relatively easier to publish articles about genetic problems.
He defined LTSNH as that the sum of the three low frequencies (125, 250, 500) was more than 100 dB, and the sum of the 3 high frequencies (2, 4, 8) was less than 60 dB.
In other words, Wolfram's is probably not diagnosed mainly by ENT's or audiologists, but rather by renal medicine doctors, as DI is exceedingly rare, while LTSNHL is not so rare in hearing tests.
That being said, it is very rare to encounter any substantial change in hearing after pregnancy. As well, its facial plastics practice offers a broad array of facial reconstructive surgeries and cosmetic procedures.
Furthermore, a hearing impairment may exist in only one ear (unilateral) or in both ears (bilateral). Head trauma, ear infections, tumors and toxic medications such as certain strong antibiotics and chemotherapeutics are other reasons for sensorineural hearing loss. Roughly speaking, the frequency patterns of hearing loss are divided up into: Low-frequency, mid-frequency, high-frequency, notches, and flat. Depending on the definition it could be estimated that more than 50% of the population over the age of 70 has impaired hearing.



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