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admin 07.11.2015

All Stars and Zen Garage made a statement last weekend with the very famous Nissan S15 making an appearance. A Mega meet would not be very mega without a DJ playing the best tunes throughout the evening! As the evening grew older, hundreds of cars and individuals packed into Western Sydney International Dragway. We didn’t get a chance to get up close and personal with this R35 like the boys at The-Lowdown did last year, but as it sat quietly in its designated corner, we could have only imagined what the combination of a Vr38, HKS GT800 turbo, plus a titanium GReddy XL exhaust would sound like.
As the Mega Meet came to a close, Top Secret Imports definitely lived up to their reputation of doing things right and doing them big! Reece Arboleda I'm a Nurse during working hours, but a car fanatic every other time of the day. About The SiteThough many may see our cars as mere inanimate objects, here at Estblshd we understand that our cars are a masterpiece that we have spent countless hours out of our lives to build and it naturally becomes an ingrained part of our memory for as long as we live. When children do not achieve their speech sounds by the expected developmental age, they may have a speech delay.
Child’s ability to communicate is directly correlated with their ability to achieve success in whatever they pursue. Children with ADHD find it very difficult to maintain social relationships at home and at school.
It was observed in a previous study that LAC significantly reduced hyperactive behaviour in FXS boys with ADHD who were treated with it for one year without causing adverse side effects.
With an eye on confirming their findings, the same team of researchers conducted the latest study, which has been reported in the American Journal of Medical Genetics Part A. Each patient followed the treatment for 12 months, which involved 500 milligrams of LAC or a placebo given twice daily.
The effects of the drug and placebo were evaluated using a set of neuropsychological tests to assess behaviour. It was observed that patients treated with LAC demonstrated reduced hyperactive behaviour and increased attention, without exhibiting any side effects. According to the researchers, the patients treated with LAC also had significantly improved social ability compared to the placebo-treated group. They suggested that the findings might be applicable to autistic children, who also do not easily tolerate stimulants. Children and adolescents with overactive and attention deficit problems often cause themselves and others tremendous stress along with a myriad of complications dealing with life matters. There are natural alternative treatments for Attention Deficit Disorder and Attention Deficit Hyperactivity Disorder that don’t involve the use of Ritalin or Concerta. You can always use traditional medication for ADD or ADHD, but talk to your doctor about starting with an alternative ADD treatment.
Attention deficit hyperactivity disorder (ADHD), currently the most common childhood-onset behavioral disorder, is nothing if not controversial. ADHD affects from 5-10% of children and adolescents, with boys 8 times more likely than girls to have it. So far over 10 genes have been suspected to be involved in the manifestation of ADHD, and many of these genes center around dopamine receptors or dopamine transporting molecules. ADHD was found to be the result of a deficiency of a specific neurotransmitter — here, norepinephrine. Its likely that the full spectrum of ADHD symptoms is not solely attributed to the prefrontal cortex, but rather entire pathways which interact together. Other ADHD drugs (like Strattera) are selective norepinephrine re-uptake inhibitors, which means it prevents norepinephrine from being degraded in the synapse. Interestingly, the mystery of ADHD doesn’t end with genes and neurotransmitters, as there is a well-documented environmental effect.
One last mention: an interesting study conducted at Chicago Medical School suggests that children diagnosed with ADHD who do receive Ritalin have a reduced likelihood of developing a drug or alcohol problem in adulthood.
I had a college friend who gave up recreational drug use after having ADHD diagnosed and properly medicated. I had been under the impression that there were no actual objective clinical indicators for ADHD, that the diagnosis was subjective. Since the most common treatment for ADHD (Ritalin) is a stimulant, what effect does caffeine have on ADHD sufferers? Caffiene stimulates the release of epinephrine and norepinephrine (although not as well as Ritalin), so I wouldn’t be surprised if many ADHD sufferers self-medicate with caffiene. Some schools do have specially-trained educators who are adept at meeting the needs of children with ADHD. I am not convinced ADHD is an inherently pathological trait (or collection of traits), although I certainly accept that the more extreme end of that trait spectrum has some serious socialising problems associated with it. As someone who, were he going through the entry levels of the educational mill today instead of six decades ago, would probably have been diagnosed as ADHD, thank you for the informative post. Its more accurate, yet costly, to examine brain activation levels in the areas in question (prefrontal cortex, limbic etc). The cheaper, quicker method may be to put the child on Ritalin for a couple days and observe any behavioral changes. Having better-appreciated behavior after taking Ritalin for a while does not constitute evidence for a diagnosis. Many people view ADHD sufferers as pleasure seekers unwilling or unable to delay gratification (symptom or cause?), and they view medication as another quick fix, an immediate gratification that doesn’t require discipline. Since there’s no physiological definition of ADHD, no amount of physiological exams will produce evidence supporting its diagnosis. No, there’s no definitive test that doctors are willing to apply while the patient is alive. Nothing pisses me off more than parents of children who do not have this problem, or ever worse those with no children at all, who judge the parents of those who do for giving them medication. I am a college student who is currently pursuing a psychology degree and who was diagnosed with ADD in 1992.
Modafinil seemed to me to have no affect on my ability to process and analyse information, but did cause me to be a little paranoid and my heart to start racing. This is one of the areas where lumping vaguely similar symptom profiles into a single theoretical and diagnostic category is harmful. As per usual you have absolutely no idea what you are talking about Caledonian so please shut the fuck up. As a (prescribed) adult ritalin user for ADHD, I’ve wondered if ritalin could increase your likelyhood of becoming addicted to, say, gambling, if you engage in gambling while under the influence of ritalin. Sooner or later, you need to do the massive problem sets, or practice the scales, or write the story, if you want to become a physicist, or a concert pianist, or the next JK Rowling.
There’s a test called the TOVA test (test of variability of attention), which is like a very boring 15 minute video game, in which the patient is told to watch the screen and to click a button on certain circumstances, but not click it under other circumstances.
Caledonian likes to troll anything psychiatrically related to try to deligimize any diagnosis he cannot see under a microscope.
Just because some illness, especially psychological ones, suffer from not always having a completely clear anatomic correlate does not invalidate their use. I was under the impression that it was pretty well assumed that genetics is a big part of ADHD.


I can believe there is a genetic component to it in that I was tied into my desk in second grade by the teacher, tired of me walking around the room. My wife and I, along with my son’s teachers, tried everything we could think of before resorting to meds.
If you don’t see these issues as problems, discussing medical ethics with you would seem to be problematic.
Before we can ask that second question, we have to possess a definition of ADHD other than the diagnostic guidelines. It’s a shame people are more interested in legitimizing existing practice rather than attempting to understand and analyze them, or we might be able to answer your questions. ADHD is not inherently pathological in anything like the same sense as a fractured femur, an aneurism, or a plasmodium infection.
I do agree that ADHD type behaviours can be socially and hence personally problematic and difficult, sometimes extremely so. But the real sticky question is, does the problematic component of these behaviours arise primarily due to a failure of society to provide an atypical but otherwise non-pathological (and indeed, potentially highly productive) neurotype with the appropriate environment?
Where is it written in the human genotype that sitting in a class for several hours a day, for several years on end, engaged in often abstract and rote based learning, is the ‘normal’, let alone optimal, developmental path? None of my points are new, this is well trodden ground, and we still don’t have a clear answer. Their name is synonymous in the Sydney Automotive scene and if you’re not aware of who they are, I am sure you have seen images from inside their headquarters posted up by our good friends The Lowdown in recent times. Reassuringly, the weather held up until one hour before show time as the heavens then opened. Everything from the track fitment to the paintwork definitely had individuals coming back for another good look. We have, however, noticed a subtle change the owner has done since Alfas private cruise awhile back. Sydney is definitely leading Australia in the fitment game showcasing to the rest of the world the high quality fitted rides we have hidden away.
It was time for the big boys to play, and it’s amazing what a change of light can do to a car. Japanese built rides weren’t the only types of rides that were of attendance last weekend. This R35 may look familiar to most of you as our good friends over at The-Lowdown had featured this ride owned by Heasman Steering¬† November last year. Due to the finite amount of hours in the day, we don’t think any photographer captured pictures of ALL of the cars in attendance. Stimulant medication like Ritalin is often successfully used to treat children with the condition, but while it is effective in children with mental retardation, it also causes side effects such as increased irritability, decreased verbalization and social withdrawal. Giulia Torrioli and Giovanni Neri, who led the project, involved in the study 51 boys between 6 and 12 years old with FXS and ADHD, who were treated in one of eight centres in Italy, France and Spain. Patients were evaluated by an interdisciplinary team of child neuropsychiatrists and psychologists at the start of the study, after one month, six months and 12 months. The patients treated with the placebo also showed reduced hyperactive behaviour, though not nearly to the extent as the LAC-treated patients. Both groups took intelligence tests, but LAC did not improve overall intellectual functioning. Watching a child with learning problems (either through a learning disorder or attention deficit disorder) struggle through school and social situations can be one of the most challenging things a parent can face. These conditions are dysfunctions of the Central Nervous System and affects attention and concentration abilities, memory, learning, and difficulties in processing and sorting out stimuli coming from a person’s subjective and objective realms.
Information about ADD is key, and by educating yourself, your family and your child, a comprehensive natural treatment for ADD is attainable.
Also, there seems to be no one gene which guarantees ADHD, but rather certain genes have been identified as denoting susceptibility to ADHD.
Like all neurotransmitters, norepinephrine is synthesized within the brain; however norepinephrine synthesis requires dopamine as an intermediate step.
About 4 million Americans are currently on Ritalin or a similar stimulant, and about 70% of those diagnosed with ADHD respond to this class of drug. A potentially promising new ADHD drug (modafinil) was recently abandoned during clinical trails just this month when one of 933 children taking the drug developed a serious skin condition. For example, babies born prematurely face a significantly greater risk of developing ADHD than full-term babies (socioeconomic status was controlled for). It is thought that the stimulants actually reduce the pleasurable effect that the drug elicits from the brain, making drug-seeking behavior and addiction more unlikely. One might wonder why the education system doesn’t develop a special program for those children. Although I wouldn’t be surprised if that was more the exception to the rule than the rule. If you consider ADHD via the neurochemical problems which cause it (low norepipinephrine), the symptoms are rather unvarying in human populations and can be easily replicated in animal models.
This would confirm the behavioral diagnosis (which, there is one, consists of observation and rating systems of sorts). If theres doubt from the initial symptoms and cognitive tests, brain activation levels could be assessed via MRI etc. I am not one who thinks this, but this seems to be one of the paradigms that conflict with attempts to define a neurological basis for ADHD.
He has a hard time remembering what 2X3 is when he is off his medication but 15 minutes after taking his pill he can reel off all the muliplication tables and is actually a pretty good student.
The way we figure it he would be stigmatized far more for being a 12-year-old third grader which is where he would be eternally stuck (in some subjects anyway). I get hyperfocused on the task at hand to the point hwere I block out everything that is going on around me. I wish I could say it wasn’t real, it would make attacking my school work a lot easier. I have yet to find anything that is comparable, but the necessity of taking it early in the morning and being affected all day is not enjoyable. When combined with Adderal it was more effective but the side effects resulted in its necessary cessation. From my experience, although I haven’t viewed the clinical data, losing modafinil as an ADD medication was no great loss. Goldstein calls a ?self-regulation deficit?, because the main problem lies in executive functions. Suggested implications for diagnosis and intervention: especially for kids and adults with ADHD-Combined, Dr.
Lots of worthwhile tasks require lots of boring repetition in between the exciting moments. When I was diagnosed in 1993, the summer before my senior year of college, I was able to provide old report cards going back to kindergarten, plotting a gradual decline of performance from straight-a to C starting in about 5th grade and extending to college. My father woke up one morning in 2005 having lost large chunks of memory of the last 50 years. All the neuro-anatomical correlates asscoiated with ADHD don’t demonstrate pathology, only difference, which is not inherently pathological.


That is my argument with the diagnostic concept, it assume the primary pathology lies with the individual. It may serve the majority of individuals well (or at least not badly), but the fact that some don’t fit into that model is no demonstration of intrinsic pathology on their part.
All I am saying is a little less haste in pathologising the individual, and a little more humility before our still profound ignorance of the nature of these behaviours. In accordance with typical Melbourne fashion, mother nature decided to clear the skies just in time for the show to start. With an approximate final count of over 3000 cars (you read that correctly!), we can only imagine that the next Mega Meet will be an incredibly important day for the automotive community. Symptoms may include passivity and inattentiveness along with uncontrollable and unruly behavior. Adding Vaxa’s Attend to the natural treatment regimen for ADD is a wonderful way to achieve the desired results.
And honestly, who could blame parents for being hesitant to medicate their young children, especially since medications come with risks? Often these symptoms are paired with poor social skills and difficulties at school, which makes for a very complex situation.
Specifically, the basic building block of each norepinephrine molecule is dopa; this molecule is converted into dopamine, which is then converted into norepinephrine. Since these areas communicate with each other, its likely that neurochemical problems in one area may affect others. Like any drug, there are costs and benefits to treatment which should be weighed with a health provider.
This drug would have been an alternative to the stimulant variety, and focused more on producing states of wakefulness. Another explanation is that the stimulant conveys improved impulse control which reduces the likelihood of partaking in risky behaviors like drug abuse, and increases the likelihood of performing well in school and developing positive social skills which would deter drug abuse.
There are far too many unresolved problems with the assumptions behind such pathologising labels.
However, as individuals have different tolerance levels (and there’s environmental factors) the behavioral (rather than neurochemical) manifestations of ADHD will differ somewhat from person to person. Its not uncommon to diagnose a neurological disorder from behavioral tests and global brain activation scans. He makes the honor roll now and I think he would feel really really bad about himself if we did not give him the pill (as opposed to just mildly bad about himself).
I have had near panic attacks when forcing myself to concentrate on difficult subject matter (that requires intense analysis and thinking) without medication in my system. Unfortunately, there are some heavy cognitive load activities that I just cannot perform without it, like upper level calculus or some more complicated chemistry. This can be conceptualized as ?a failure of self-control within the context of prefrontal lobe functions? (Dr. Even goals chosen by a person, not dictated by parents or teachers, have this inherent characteristic. It then does some analysis, provides graphs, and places your performance in comparison to normals.
No physiological tests or scans showed anything, and some physicians thought it might be psychological. Despite the heavy downfall prior, this did not stop the thousands of individuals who packed into the Western Sydney International Dragway (Eastern Creek).
That custom wide body has definitely made a big difference to the overall appearance of the ride.
We are loving the owners choice of subtle modifications that make a big difference to the overall look of the car. We look forward to seeing more from Top Secret Imports and the Sydney automotive scene in general.
Affected individuals might get out of control, become over or under stimulated, or go through uncontrollable stimulation patterns weaving through their sensorium, which cause inconsistent and volatile behavioral patterns very hard to control in a household. It is a frustrating condition, causing plenty of stress to individuals which could go through serious emotional and psychological problems.
No one particular approach is always going to work, however, by implementing many of the ideas to naturally help these symptoms there is a likelihood that improvement will occur. However, ADHD is a very real (and prevalent) disorder which has discrete neurochemical and, as more and more research is suggesting, genetic causes. One early study in 1990 discovered that brain activity was 8.1% lower in 30 of 60 brain region in adults who suffered from ADHD (measured by global glucose metabolism). According to the graph below, the United States and Canada have seen sharp increases in Ritalin prescriptions over the past few years. This is also suggested by the number of genes thought to be involved in suspectibility to ADHD (10+). I actually have a hard time with coffee in that it makes me unbearably talkative and controlling. Elkhonon Goldberg, 2001), and the main problem seems to lay on poor behavioral inhibition (Barkley, 1997).
One shrink put him on Ritalin, which was a trip – for a week he was an absolute whirlwind of activity, replaced the planks on the back deck of the house, even got on my old mountain bike and rode down the block, probably his first time on a bike in 60 years.
Some of our team will be in Sydney for the Community Meet held at Garage 88 this Friday so make sure you come and say hi! Attention Deficit affects the entire realm of learning, from socializing with others to performing well academically. Many authorities believe that one of the greatest deficiencies of our times is an omega 3 deficiency. But this is just part of the problem. The regions with the most significant decreases in activity were the premotor cortex and the superior prefrontal cortex (shown below, circled), which are regions which (among other things) mediate impulse control. Theoretically, if this dopa-to-norepinephrine synthesis is altered (say by certain genes), low levels of norepinephrine and ADHD-like symptoms could occur. More dopamine receptors would, in turn, mean less norepinephrine in the brain as the required substrate (dopamine) would have a higher likelihood of being bound or degraded before it could be synthesized into norepinephrine. These genes mostly affect dopamine receptors (as I explain above) but perhaps in different ways, and varying in severity.
I don’t think that the school gives out caffienated beverages but if they did we might even consider going that route. It is generally not considered appropriate or helpful to segregate these children in special classrooms and doesn’t happen except perhaps an hour a day if the child is experiencing delays in, say, learning to read, or organizing written work, or getting assignments completed. Because of depleted nutrients in our soil that has been occurring over the last 100 years, your child is not getting all of the nutrients you did or your grandparents did when they do eat a fruit or vegetable. Conversely, drugs which provide extra levels of norepinephrine relieve the symptoms of ADHD.




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