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In addition to his position as Associate Investigator at The Feinstein Institute for Medical Research, Dr.
Argyelan M, Ikuta T, DeRosse P, Braga RJ, Burdick KE, John M, Kingsley PB, Malhotra AK, Szeszko PR. Szeszko PR, Narr KL, Phillips OR, McCormack J, Sevy S, Gunduz-Bruce H, Kane JM, Bilder RM, Robinson DG. Szeszko PR, Hodgkinson CA, Robinson DG, Derosse P, Bilder RM, Lencz T, Burdick KE, Napolitano B, Betensky JD, Kane JM, Goldman D, Malhotra AK. She also directs the Recognition and Prevention (RAP) program, which she founded in 1998, at Zucker Hillside Hospital. From a research perspective, the RAP program is focused on identifying biologically based risk factors and understanding the biological mechanisms leading to illness. The RAP program is funded by the NIMH and several private foundations, including Robert Wood Johnson and the International Mental Health Research Organization. Cannon TD, Cadenhead K, Cornblatt B, Woods SW, Addington J, Walker E, Seidman LJ, Perkins D, Tsuang M, McGlashan T, and Heinssen R. We have also discovered that in addition to triggering uncontrolled systemic inflammation, bacterial infection also suppresses the expression of multiple “housekeeping genes”, whose products are critical for the maintenance of normal organ functions, and impairs our body’s anti-inflammatory and protective mechanisms.
Philip Szeszko is an Associate Professor of Psychiatry and Molecular Medicine at the Hofstra Northwell School of Medicine. Szeszko has received grant funding from the National Institute of Mental Health, the DANA Foundation, Brain and Behavior Research Foundation (Young and Independent Investigator Awards), Obsessive-Compulsive Foundation and American Foundation for Suicide Prevention and was a prior recipient of a Mentored Research Scientist development Award (K01). Szeszko’s lab is examining the brains of patients with psychiatric disorders using magnetic resonance imaging. Szeszko’s work has also focused on the role of the anterior hippocampus in the pathophysiology of schizophrenia, and he has demonstrated that structural alterations in this region have a unique set of neuropsychological correlates, which implicate a defect in connectivity between the frontal and temporal lobes. Barbara Cornblatt is a professor of psychiatry and molecular medicine at the Hofstra Northwell School of Medicine. RAP is dedicated to treating early warning signs of mental illness in adolescents and young adults and is one of the longest running centers of its kind in North America.
Cornblatt has authored or co-authored over 150 scientific articles and book chapters and has given many scientific talks nationally and internationally to a variety of prestigious scientific organizations, hospitals and consumer groups. Cornblatt focuses on the neurocognitive roots of mental illness and has developed the Continuous Performance Test, Identical Pairs (CPT-IP) version, considered internationally to be one of the cutting-edge measures of working memory and attention.

Cornblatt’s research focus is on the early identification and treatment of adolescents and young adults (ages 12-22) who are considered to be at-risk for developing serious psychiatric illness as adults, especially schizophrenia and other psychotic disorders.
Mechanisms and predictors are studied on multiple levels of development, starting from the earliest genetic factors up through a number of cognitive and behavioral problems. Cornblatt is also concerned with potential environmental triggers of illness and has extensively studied the impact of cannabis on emerging symptoms.
Shu Fang Liu graduated from Shanghai Second Medical University in China and received his PhD in Molecular Pharmacology from Imperial College, University of London, England. Liu’s laboratory focuses on molecular mechanisms of septic shock, and septic multiple organ injury (MOI), conditions that claim thousands of life each year.
We are investigating how bacterial infection represses housekeeping gene expression and impairs the anti-inflammatory and protective mechanisms, and are trying to find the ways of boost these anti-inflammatory mechanisms. NF-?B-to-AP-1 switch: a mechanism regulating transition from endothelial barrier injury to repair in endotoxemic mice.
An Obligatory Role of NF-?B in Mediating Bone Marrow Derived Endothelial Progenitor Cell Recruitment and Proliferation Following Endotoxemic Multiple Organ Injury in Mice.
He is currently the Principal Investigator of a program project for the Zucker Hillside Hospital Center for Interventional Development and Applied Research (CIDAR).
The goal of this work is to identify the structural and functional abnormalities that play a role in the pathogenesis of disorders including schizophrenia, bipolar disorder and obsessive-compulsive disorder and to identify neuroimaging predictors of treatment response and outcome and translate these findings from “bench to beside.”  Prior work by his group suggests that magnetic resonance imaging can be used to predict treatment response in patients experiencing a first-episode of schizophrenia and an ongoing study is examining the use of diffusion tensor imaging to predict which patients with schizophrenia may be most likely to benefit from omega-3 treatment.
In addition, his work has shown that this part of the hippocampus is particularly affected by stress, thus making it a potentially important target for environmental insults, which could lead to the manifestation of schizophrenia.  Along these lines an additional focus of Dr. For more than 20 years, she has focused her research on the causes and treatment of serious mental illness. More than 500 young people have participated in both the research and treatment components of this program. She is the co-director of the International Prodromal Research Network, which sponsors international collaborations and prevention studies.
The major goal of the RAP program is to prevent illness from developing in at-risk youngsters. Most recently, the RAP program and eight other top academic centers have formed a multi-site collaboration to expand prodromal research. He is currently an investigator at the Feinstein Institute for Medical Research and professor of medicine at Hofstra Northwell School of Medicine.

Using modern molecular biology techniques, we are unveiling the molecular and cellular events that take place when bacterial infections initiate the inflammatory process and lead to the development of septic shock and septic MOI. Szeszko is examining how abnormalities in crossing white matter pathways contribute to the neurobiology of schizophrenia. Early treatment in the RAP Clinic has been consistently associated with a high rate of clinical improvement. She is also a member of the North American Prodrome Longitudinal Study (NAPLS) consortium, one of the largest and most successful prevention collaborations worldwide. Since the onset of the RAP program in 1998, over 500 young people have participated in both treatment and research components of the program. Some of the new areas introduced into the RAP research program include studies of the structure and function of the brain (MRIs), electrical activity on the brain (EEG), impact of cortisol and stress and possibly blood analytes, proteins and other biomarkers to improve accuracy of prediction.
Our goal is to discover the key molecules that can be new targets for developing new therapies for treating these life-threatening conditions. Szeszko has authored or co-authored over 80 peer-reviewed publications and sits on the Editorial Boards of Psychiatry Research: Neuroimaging, Progress in Neuro-psychopharmacology and Biological Psychiatry and Schizophrenia Research and Treatment. While a high rate of clinical improvement has already resulted from the RAP intervention procedures, new treatment strategies, both pharmacological and psychosocial, continue to be evaluated, with a particular emphasis on family involvement.
Liu has been investigating the molecular and cellular mechanisms of multiple organ injury caused by sepsis, a condition that claimed thousands of lives each year for 20 years. We have discovered that activation of endothelium, a monolayer of cells lining the inner surface of all our blood vessels, by inflammatory mediators is a “rate-limiting” factor determining the severity of organ injury. Prevention of endothelium activation and injury without blocking inflammation is sufficient to mitigate MOI caused by sepsis.
We are currently developing strategies to strengthen the endothelium or to accelerate the repair of the injured endothelium.

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