Herpes breakthrough formula,get rid herpes scars,herpes transmission hsv 1 8.0 - For Begninners

admin 02.09.2013

Early clinical trials in the United States have found the vaccine can prevent infection in three out of four women.
An estimated 100m people around the world are infected with what is known as herpes simplex virus type 2. The virus can cause painful itching and sores that in some cases reappear years after infection. However, many people do not have symptoms and can therefore pass on the infection without knowing it. A vaccine could be of real benefit to the thousands of infants born every year with the virus.
Dr Lawrence Stanberry and colleagues at the University of Texas Medical Branch tested the vaccine on 978 women and 1,736 men whose partners had genital herpes.
Each received three injections containing the vaccine or injections containing a placebo over the course of six months. The researchers found that the vaccine did not work well in women who had been infected with a related virus - herpes simplex virus 1. GlaxoSmithKline is now recruiting 7,550 women between the ages of 18 and 30 across the US for final stage clinical trials. The UK Herpes Viruses Association said most new cases of genital herpes are actually caused by herpes simplex virus 1 - the virus that causes cold sores.
Marian Nicholson, its director, said there was a need to tackle the stigma around the virus.


Using fire to fight fire seems like a fairly bad idea when it comes to curing the human body of fatal diseases. According to a study published on March 27 by the Journal of Clinical Oncology, the drug, called T-VEC, tested positive in the phase 3 trial, making it the first “virotherapy” treatment to make it this far in testing. Part of what makes T-VEC such an amazing breakthrough for cancer treatment is its ability to attack and potentially overtake cancer when it has advanced throughout a person’s body and into their organs, a condition which is generally very grim.
The results are particularly promising considering the state most of the patients were in when treatment began. The revolutionary drug works by first attacking the cancer cells directly and then drawing the attention of the immune system to come in and finish off the remaining cancer. After the immune system is drawn by the initial T-VEC attack on cancer cells, it quickly launches an assault on all the cancer cells in the body, even those that haven’t been touched by the virus. When used to treat patients with stage III and early stage IV melanoma, the treatment increased survival rates dramatically. However, half of those infants who contract the virus die unless they receive powerful antiviral drugs. But a new drug that uses a modified form of the herpes virus to combat advanced cancer has shown extremely promising results, and may herald a new generation of cancer treatments.
During the trial, one in four of the 400 patients treated with the experimental drug responded to the treatment.
The backbone of the treatment is the modified version of herpes that has been ‘neutered’ so that it can no longer produce the protein that causes it to attack ordinary healthy cells.


Patients who took the drug survived an average of 41 months, compared to the average 21.5 months in the 66 earlier-stage seen in patients who only received the control treatment. Enabling JavaScript in your browser will allow you to experience all the features of our site.
The unconventional treatment is predominantly used to treat patients with advanced and aggressive forms of skin cancer. It’s the first time a virotherapy has been shown to be successful in a phase 3 trial,” Kevin Harrington, professor of biological cancer therapies at the Institute of Cancer Research London, who led the work told the Guardian. All of the patients had aggressive melanoma, and after the drug was administered, 16 percent of them were still in remission after six months and 10 percent were in complete remission, with no discernible trace of cancer remaining. Cancer cells, however, create a different version of the protein that was cut out of the herpes virus, causing the virus to attack and thrive within the regions affected by cancer.
The drug is close to market ready, with scientists claiming that if it is approved T-VEC could be an option for cancer patients as soon as next year.
After infecting the cancerous tissue, the herpes virus multiplies rapidly within the cancer cells causing them to burst open while drawing the attention of the otherwise blind immune system. In this case we are harnessing the ability of an engineered virus to kill cancer cells and stimulate an immune response,” said Professor Paul Workman, Chief Executive of The Institute of Cancer Research.




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