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The shingles vaccine is approved for use in people 60 and older, though younger people may benefit too.
After you get chickenpox, the virus remains inactive (becomes dormant) in certain nerves in the body. If an adult or child has direct contact with the shingles rash and did not have chickenpox as a child or the chickenpox vaccine, they can develop chickenpox, not shingles. The first symptom is usually pain, tingling, or burning that occurs on one side of the body.
The rash usually involves a narrow area from the spine around to the front of the abdomen or chest. You may also have pain, muscle weakness, and a rash involving different parts of your face if shingles affects a nerve in your face. Strong anti-inflammatory medicines called corticosteroids, such as prednisone, may be used to reduce swelling and pain. Caring for your skin by applying cool, wet compresses to reduce pain, and taking soothing baths. Stay away from people while your sores are oozing to avoid infecting those who have never had chickenpox -- especially pregnant women.

Do not touch the rash and blisters on persons with shingles or chickenpox if you have never had chickenpox or the chickenpox vaccine. The information provided herein should not be used during any medical emergency or for the diagnosis or treatment of any medical condition. Cientificos espanoles han descubierto que el virus del herpes produce una proteina que, al entrar en contacto con las neutrofinas, favorece el crecimiento axonal de la enfermedad y facilita su entrada en el sistema nervioso. Un estudio realizado por investigadores del Centro de Biologia Molecular Severo Ochoa (centro mixto del Consejo Superior de Investigaciones Cientificas (CSIC) y la Universidad Autonoma de Madrid) ha demostrado que una proteina generada por el virus del herpes simple tipo 2 une un tipo de neutrofinas, proteinas que favorecen el crecimiento axonal y la supervivencia de las neuronas. Hasta hora se desconocia el mecanismo por el que el virus del herpes conseguia infectar la piel y la mucosa hasta alcanzar las neuronas perifericas, en las que permanece latente durante toda la vida del individuo afectado. El trabajo, que se ha publicado en la revista PLOS Pathogens, indica que la capacidad de potenciar el crecimiento axonal de la proteina generada por el virus del herpes simple tipo 2 esta modulada por la union a la neurotrofina, atrayendo asi las terminaciones nerviosas hacia el sitio de la infeccion. De ser asi, este crecimiento axonal ayudaria a que el virus pasara desde la piel hasta el sistema nervioso, garantizando de este modo el ciclo viral. Antonio Alcami, otro de los cientificos participantes en el estudio, explica: “Descubrir los mecanismos que permiten al virus del herpes acceder al sistema nervioso permitira disenar en el futuro virus especificos y eficientes que puedan usarse en terapia genica”. Ecology, social and environmental sustainability, emphasis on conservation of rivers and ecosystems, denounces impact of megaprojects.

Vyas, MD, PhD, Assistant Professor in Medicine, Harvard Medical School; Assistant in Medicine, Division of Infectious Disease, Department of Medicine, Massachusetts General Hospital. A licensed medical professional should be consulted for diagnosis and treatment of any and all medical conditions.
Este hallazgo podria abrir nuevas vias para terapias de procesos neurodegenerativos y de regeneracion axonal. Ademas, la interaccion entre esta proteina y las neurotrofinas explicaria su habilidad para invadir y vivir en determinados tejidos nerviosos.
Older adults who receive the herpes zoster vaccine are less likely to have complications from shingles. Links to other sites are provided for information only -- they do not constitute endorsements of those other sites. Any duplication or distribution of the information contained herein is strictly prohibited.

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