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Androstenedione is a type of Androgen Sexual Steroid Hormone produced by the Hair Follicles, male Testes and (in much lesser quantities) by the female Ovaries. The ability of Androstenedione to cause Muscle Growth is limited (rate-limited) by the availability of the necessary enzyme that catalyzes this conversion of Androstenedione to Testosterone. Removal of the Ovaries causes a reduction in endogenous Androstenedione production and this causes subsequent Bone loss.
Supplemental Androstenedione may improve Memory - especially Short Term Memory (this effect occurs via the conversion of Androstenedione to Testosterone, a known enhancer of Memory). Supplemental Androstenedione (taken approximately 30 - 60 minutes prior to intended sexual activity) has been reported to increase Sexual Desire in some people (this effect is believed to occur from the conversion of Androstenedione to Testosterone). Supplemental Androstenedione increases the body's Estradiol levels - caution excessive Estrone is associated with various toxic side effects. Small amounts (1 - 5%) of Androstenedione are converted (by the P-450 Aromatase Enzyme) within peripheral tissues (e.g.
Supplemental Androstenedione (300 mg per day) causes a significant increase in the body's Estrone levels - caution excessive Estrone is associated with various toxic side effects. Cortisol is a type of Adrenal Glucocorticoid Hormone (Corticosteroid) manufactured by the (zona fasciculata and zona reticularis of the) Adrenal Glands.
Cortisol decreases the permeability of Capillaries (resulting in less Blood Plasma and fewer Cells entering areas of Inflammation).
Cortisol suppresses the synthesis of several endogenous compounds involved in Inflammation (including Interleukin 1 and Tumor Necrosis Factor-a). DHEAS is chemically similar to Dehydroepiandrosterone (DHEA) except for the inclusion of Sulfate in this molecule - DHEAS is predominantly water-soluble, whereas DHEA is predominantly fat-soluble. DHEAS possesses Life Extension possibilities: The death rate for people who naturally have high levels of DHEAS is lower than for those with reduced levels of DHEAS. DHEAS suppresses the proliferation of the Cancer cells involved in Pancreatic Cancer (by inhibiting the Glucose-6-Phosphate Dehydrogenase (G6PD) enzyme). The body's levels of DHEAS decline in tandem with the progression of the Aging Process - by the age of 70, DHEAS levels in most people have declined to approximately 20% of their peak levels. Dihydrotestosterone is manufactured endogenously in the Sebaceous Glands and then attaches to and shrinks the Hair Follicle, reducing the diameter of the hair shaft and restricting the nutrient supply to the Hair.
Binding of Dihydrotestosterone (DHT) to Androgen Receptors on Hair Follicles stimulates the production of excessive amounts of Sebum. DHT is five times more potent in stimulating Prostate Cancer cell growth than Testosterone).
Ethocyn (applied topically) is claimed by its manufacturers to block receptors for Dihydrotestosterone. Progesterone inhibits the conversion of Testosterone to Dihydrotestosterone (DHT) (by inhibiting the activity of 5-Alpha Reductase, the enzyme that catalyzes the conversion of Testosterone to DHT). Enterodiol inhibits the binding of Dihydrotestosterone to Sex Hormone Binding Globulin (SHBG). Enterolactone inhibits the binding of Dihydrotestosterone to Sex Hormone Binding Globulin (SHBG).
Matairesinol inhibits the binding of Dihydrotestosterone to Sex Hormone Binding Globulin (SHBG). Secoisolariciresinol inhibits the binding of Dihydrotestosterone to Sex Hormone Binding Globulin (SHBG). Zinc inhibits the conversion of Testosterone to DHT (by inhibiting the activity of the 5-Alpha Reductase enzyme that is responsible for this conversion). Vitamin B2 inhibits the conversion of Testosterone to DHT by inhibiting the activity of 5-Alpha Reductase. Flax Seeds inhibit the binding of Dihydrotestosterone to Sex Hormone Binding Globulin (SHBG) (due to the Plant Lignans content of Flax Seeds). Approximately 6 - 10% of the body's free Testosterone is converted (via the 5-Alpha Reductase enzyme). Estrogens are a group of Female Sexual Steroid Hormones produced in the Ovaries and (to a lesser extent in males) the Testes. Estrogens improve Blood Circulation to the Brain (and thereby alleviate Cerebral Insufficiency).
Estrogens (preferably natural Estrogens identical in chemical structure to those produced endogenously) help to prevent Alzheimer's Disease in post-menopausal women and improve the Mental Function of persons afflicted with Alzheimer's Disease.
Estrogens stimulate the production of and increase the Brain's sensitivity to the effects of Nerve Growth Factor (NGF). Estradiol (one of the Estrogens) inhibits the ability of Amyloid-Beta Protein to destroy Neurons.
Estrogens improves Blood Circulation to the Frontal Lobe and Precentral Gyrus areas of the Brain.
Estrogens improve Memory (studies have demonstrated the ability of Estrogen Replacement Therapy to improve Short-Term Memory and Long-Term Memory in women). The toxic effects of Estrogens only occur when the sum of Estradiol + Estrone exceeds 50% of the body's total Estrogens .

Breast Cancer (specifically 2-Hydroxyestrone form of Estrone)It is noteworthy that of the three types of Estrogens, Estradiol is 1,000 times more potent in terms of its ability to stimulate Breast tissue growth compared to Estriol. The specific Cell growth that Estrogens are known to stimulate is Trophoblasts which are strongly implicated in Cancer when they are produced in situations other than Pregnancy.
Excessive use of exogenous, supplemental Estrogens by postmenopausal women increases their risk of developing Systemic Lupus Erythematosus (SLE). Estrogens inhibit the mobilization of Adipose Tissue from Cellulite for redistribution to other areas of the body (possibly accounting for the greater incidence of Cellulite in females than in males).
Excessive Estrogens levels can cause Depression (due to them blocking the Serotonin manufacturing properties of Vitamin B6). Excessive Estrogens levels or a high Estrogens:Progesterone ratio (Estrogens Dominance) may be a cause of Multiple Sclerosis. Excessive production of Estrogens is implicated in the PMS-A (Anxiety) form of Pre-Menstrual Syndrome (PMS). During and following Menopause, a significant number of females do NOT require Estrogen Replacement Therapy - the female body still produces a small quantity of Estrogens (from Androstenedione within Adipose Tissue) - Estrogen production merely declines in tandem with the female body's reduced requirement for Estrogens that were formerly necessary to prepare her Endometrium for Pregnancy.
Excessive production of Estrone (and impaired conversion of Estrone to Estriol within the Liver) has been closely linked to Breast Cancer. Excessive production of Estrone (and impaired conversion of Estrone to Estriol within the Liver) has been closely linked to Ovarian Cancer. Excessive production of Estrone (and impaired conversion of Estrone to Estriol within the Liver) has been closely linked to Uterus Cancer.
Impaired conversion of Estrone to Estriol within the Liver causes the PMS-A form of Pre-Menstrual Syndrome (PMS). Synthetic Estrogens differ in their chemical structure to that of the body's natural Estrogens and can produce toxic side-effects that exceed those of natural, endogenous Estrogens. Estradiol is the major (most biologically potent) Estrogen produced and secreted by the Graafian Follicle of the female Ovaries (and produced in smaller quantities in males) - note that Estradiol is not one single compound - there are two forms of Estradiol (of which Estradiol 17-Beta is the most biologically potent). Male Alzheimer's Disease patients have significantly lower levels of Estradiol than healthy control subjects. Estradiol increases (female) Sexual Desire (by stimulating the endogenous production of Nitric Oxide Synthase (NOS) which catalyzes the production of Nitric Oxide (NO)). Estriol inhibits the ability of Estradiol and Estrone (and synthetic Estrogens that mimic Estradiol and Estrone) to stimulate the growth of Cancer Cells involved in Uterus Cancer. Estriol (natural, not synthetic) is the primary natural Estrogen recommended for use in Estrogen Replacement Therapy (ERT) post-menopausal women whose production of Estriol declines significantly following Female Menopause. Topical Estriol decreases the size of Enlarged Pores (as measured scientifically Skin profilometry) by between 61% and 100%. Topical Estriol increases the quantity of Type III Collagen in the Skin and increase the quantity of Collagen Fibers in the Skin.
Melatonin is a Neurohormone (regarded as a Neuropeptide) produced by the Pineal Gland (and to a lesser extent in the Retina of the Eye and by the Enterochromaffin Cells of the Gastrointestinal Tract). Excessive consumption of Alcohol (ethanol) inhibits with the body's production of Melatonin.
Progesterone is a type of female Sexual Steroid Hormone secreted by the Ovaries, Placenta and (in small amounts) by the Adrenal Cortex and (in males) the Testes. Progesterone inhibits the excessive proliferation of the Smooth Muscle of the Endothelium of Blood Vessels (excessive proliferation of the Smooth Muscle of the Endothelium of Blood Vessels can lead to Atherosclerosis).
Progesterone helps to prevent many types of Cardiovascular Diseases (due to its ability to minimize risk factors for Cardiovascular Diseases such as elevated LDL Cholesterol, lowered HDL Cholesterol and elevated Triglycerides levels). Progesterone helps to prevent Hypertension (by facilitating the excretion of excessive Sodium from the body). Progesterone helps to prevent Endometrial Cancer in postmenopausal females (by counteracting Estrogens-dominance that is implicated in Endometrial Cancer). Progesterone inhibits the growth of Prostate Cancer cells (by inhibiting the conversion of Testosterone to Dihydrotestosterone and by opposing the Prostate Cancer stimulating effects of Estrogens). Progesterone may stimulate Prostate Cancer cells to undergo Apoptosis (programmed cell death). Progesterone facilitates the utilization of Adipose Tissue (body fat) in the production of Energy.
Progesterone facilitates the utilization of Adipose Tissue (body fat) for the production of Energy. Progesterone increases bone mass density (BMD) by 7% after one year of supplementation, by 12% after two years, and by 15% after three years. Progesterone improves Concentration ability in anovulatory premenopausal and postmenopausal women. Progesterone may be an effective therapy for Multiple Sclerosis (due to its ability to counteract estrogens dominance.
Progesterone prevents the premature shedding of the supportive secretory Endometrium during Pregnancy.
Progesterone (cream applied topically) alleviates Uterine Fibroids (by counteracting the estrogens dominance that is the underlying cause of many cases of Uterine Fibroids).

Progesterone (Progesterone cream applied intravaginally) alleviates many cases of Vaginal Atrophy.
Natural, exogenous Progesterone (cream applied topically) alleviates Vaginitis (including the vaginal dryness and atrophy of Mucous Membranes of the Vagina that are associated with Vaginitis). Progesterone (cream applied topically) alleviates many cases of Acne in adult women (by counteracting the excessive production of Androgens that causes most cases of adult Acne in women). Progesterone (as a component of some Cosmeceutical Skin Moisturizers) alleviates Dry Skin (by facilitating the retention of Water in the Skin).
Abnormally large Breasts can occur in women as a result of insufficient endogenous production of Testosterone. Supplemental, exogenous, natural Testosterone often alleviates Male Menopause (by counteracting the decreased free serum Testosterone associated with increased levels of Sex Hormone Binding Globulin).
Lack of Sexual Desire (libido) in post-menopausal females is often caused by insufficient Testosterone production - supplemental Testosterone (administered by a qualified practitioner implanting a Testosterone "pellet" under the skin every six months) often restores the libido of such females. Testosterone exerts its effects on Sexual Desire by influencing (presently unidentified) Receptors in the Brain.
Testosterone is responsible for stimulating the development of Male Sexual Organs and male secondary sexual characteristics (e.g. Testosterone improves (male) Sexual Performance (men with low serum Testosterone levels (i.e. Testosterone (cream applied topically to the Clitoris) improves (female) Sexual Performance (by increasing the sensitivity of the Clitoris). Testosterone is responsible for stimulating the development of female secondary sexual Hair.
Acetyl-L-Carnitine (ALC) increases plasma Testosterone levels (via its influence on Acetylcholine neurotransmission in the Striatal Cortex of the Brain). Melatonin inhibits the production of Testosterone - although low dosage levels of Melatonin (up to 3 mg per day) do not appear to interfere with Testosterone production. Progesterone lowers endogenous Testosterone production (in men) to levels that prevent the proper maturation of Sperm.
Alcohol (ethanol) decreases the production of Testosterone and lowers the body's Testosterone levels. Tobacco smoking inhibits the production of Testosterone (by causing the destruction of Leydig Cells that normally produce Testosterone). Vegetarians have approximately 18% lower Testosterone levels compared to meat eaters (this occurs because vegetarians have a lower intake of Dietary Fats which are precursors for Testosterone production; also the Fiber Polysaccharides in Vegetables bind to Testosterone and other Steroid Hormones). We couldn't find the page you were looking for.Please try another search or go back to the home page.
At second roundabout, which will have your first set of traffic lights, take the fourth exit, (one exit before you double back on yourself). Turn left at point 4 Traffic lights at the top of the hill (seconds after passing petrol station on your left). It is also produced in lesser quantities by (both the male and female) Adrenal Cortex of the Adrenal Glands. It is produced in and secreted by the zona fasciculata and zona reticularis of the Adrenal Glands. For this reason post-menopausal women are advised to consider the use of natural Estrogens in ERT (natural Estrogens are more difficult to procure than Synthetic Estrogens). Because Estriol has minimal ability to stimulate Cell proliferation, it thereby inhibits the ability of other Estrogens to stimulate Breast Cell proliferation (by occupying Estrogens Receptors that would otherwise be occupied by Estradiol or Estrone). Intelligent physicians recommend that natural Estriol comprise 80% of the Estrogens in natural Estrogens Replacement Therapy. Photographs of "old" mice treated with Melatonin reveal that their fur is thick and lustrous and their bodies firm and streamlined, while the untreated mice have flabby bodies and wrinkled necks and scruffy coats that are graying in patches.
Women administered supplemental Progesterone up to the age of 80 following Menopause exhibit strong Bones without evidence of Bone loss while continuing to use natural Progesterone. Seconds later you will pass a petrol station on your left, immediately followed by a post office sorting depot on your right. Shortly before you reach the next set of traffic lights, you will see a car park on your right. As you turn right, facing into this car park, the back of the Osteopathic Practice will be situated to the far left corner of the car park. Androstenedione is regarded as a weak Androgen as it does not exert physiological effects to the same extent as the major Androgens. Here, there is a pedestrian alleyway to access Nolton Street and the front of the Practice.

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