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Washington, March 16 (QNA) - In a search for new drugs to treat viral infections, scientists at the University of Utah School of Medicine have found that a drug routinely used to treat heart failure spironolactone has the ability to block infection by Epstein Barr virus (EBV), a herpes virus that causes mononucleosis and is associated with several human cancers. The researchers found that the drug's antiviral properties stem from its ability to block a key step in viral infection that is common to all herpes viruses. This is because HSV has an incubation period that can range from a few weeks to over a year. ACV can be taken orally, intravenously (passed directly into the body through an IV), and topically.
Equally troublesome is the potentially challenging feat of finding sexual partners after revealing their condition. Both Acyclovir and Valacyclovir work to decrease the severity, intensity, and duration of outbreaks and are available in a variety of dosing strategies (depending on the preference of the patient). Spironolactone's target is distinct from that of existing drugs, suggesting that it could be developed into a new class of anti-herpesvirus drug, according to the researchers. Most people infected with the virus discover their condition either from an initial outbreak, or the results of a blood test.

Oral ACV spreads widely throughout the body via bodily fluids, topical ACV works in the area of application but has little effect elsewhere in the body, and intravenous ACV is reserved for patients with compromised immune systems (such as those already suffering from HIV).
This is where Valacyclovir provides an exciting and unexpected benefit: when taken in a daily suppressive regimen, Valacyclovir was found in an eight-month study to reduce Herpes transmission by 50% among susceptible partners. It must be stressed again that, while these drugs provide significant improvements in the lives of people infected with Herpes, there remains no cure. Valacyclovir is intended for use in the treatment of both genital and oral Herpes, helping to curb infections by reducing the frequency and severity of outbreaks. When taken in a low dosage twice daily, Valacyclovir has a 44% likelihood of aborting lesions before they fully develop, though the chance of this happening is closely related to the quickness with which treatment is started.
Persons infected with HSV can spread the virus outside of a detected outbreak, creating the risk of transmission even when no symptoms are present; suppressive regimens of Valacyclovir work to decrease asymptomatic shedding.
Therefore, all of us here at SexInfo strongly encourage the use of condoms to prevent the transmission of STIs altogether. These individuals, however, can take comfort in knowing that certain drug regimens control the frequency, duration, and severity of outbreaks and are relatively affordable. It has been approved by the FDA to treat both HSV-1 and HSV-2 during initial outbreaks, recurrent outbreaks, and as a therapy to suppress the virus’ activity for extended periods of time.

The chance of the lesions fully aborting is almost twice as likely when treatment is begun less than six hours after the onset of symptoms.
Under the HoodBehavior, Neuroscience & Your Brain What Brain Hacks Can Psychologists Teach Us? It must be stressed, though, that while drugs to control the infection are available and effective, there currently is no cure for a Herpes infection.
For best results, an Acyclovir regimen should begin at the first sign of Herpes sores or lesions.
People who choose to engage in sexual activity with new and experienced partners are strongly advised to take precautions. Using condoms will not only prevent pregnancy, but will also prevent against the transmission of various STIs (including Herpes).

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