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Author: admin | 13.04.2015 | Category: One Ear Ringing

Sensory presbycusis: This refers to epithelial atrophy with loss of sensory hair cells and supporting cells in the organ of Corti. Mechanical (ie, cochlear conductive) presbycusis: This condition results from thickening and secondary stiffening of the basilar membrane of the cochlea. Sign up for our newsletter and get our top new questions delivered to your inbox (see an example). The rest of my answer is based on this book, some parts of which exist online (which I'll quote). As you can see from the graph, severity of age-related hearing loss depends on sound frequency: older people need high-pitched sounds to be displayed more loudly, in order to hear them.
It's not described much on the website, but the book explains that there is a relationship between how loud a sound has to be to harm you, and which frequency it is. Here's a plot of the connection between loudness and frequency in auditory perception, in which you can see that sounds around 4kHz can most easily harm us. Does this explain that when I was younger, I was more sensitive to sound and slept less well?
Not the answer you're looking for?Browse other questions tagged medical-science or ask your own question. If you suspect that your hearing isn’t quite what it used to be, here is a simple self-assessment you can take at home.
Do you have trouble following the conversation with two or more people talking at the same time? Characteristically, presbycusis involves bilateral high-frequency hearing loss associated with difficulty in speech discrimination and central auditory processing of information.


This process originates in the basal turn of the cochlea and slowly progresses toward the apex.
The thickening is more severe in the basal turn of the cochlea where the basilar membrane is narrow. The question is being unable to hear ranges as we get older, starting from childhood, while presbycusis is specific to post middle-aged people. Sounds that are near our auditory thresholds (under 20 Hz and above 20.000 Hz in the normal, healthy hearing range) can't harm us even if they are extremely loud. Jobs where loud noise is a regular part of the working environment, such as farming, construction or factory work, can lead to damage inside your ear.
Exposure to explosive noises, such as from firearms and fireworks, can cause immediate, permanent hearing loss. Drugs, such as the antibiotic gentamicin and certain chemotherapy drugs, can damage the inner ear. Diseases or illnesses that result in high fever, such as meningitis, may damage the cochlea. If you answer yes to more than two of the following questions, you should have your hearing evaluated further by a health-care professional.
These changes correlate with a precipitous drop in the high-frequency thresholds, which begins after middle age. This correlates with a gradually sloping high-frequency sensorineural hearing loss that is slowly progressive. The wiki page does refer to the issue as presbycusis, yet every reliable source on presbycusis lists it as being limited to post middle-aged people.


We don't hear them because they cause no mechanical change in our ear that responds to those frequencies, and consequently no harm is done. Other recreational activities with dangerously high noise levels include snowmobiling, motorcycling or listening to loud music. The abrupt downward slope of the audiogram begins above the speech frequencies; therefore, speech discrimination is often preserved.
It seems the wiki page is the only source referring to range of audible frequencies in young people as presbycusis?
In the case of age-related hearing loss, the hair cells in the inner ear that should respond to high-frequency sounds, have already stopped responding, so there is nothing left to harm. Personal music players, such as MP3 players, can cause lasting hearing loss if you turn the volume up high enough to mask the sound of other loud noises, such as those from a lawn mower. Histologically, the atrophy may be limited to only the first few millimeters of the basal end of the cochlea. One theory proposes that these changes are due to the accumulation of lipofuscin pigment granules.



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