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Author: admin | 30.11.2013 | Category: Tinnitus Hearing Loss

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Did you know that many prescription and over-the-counter medications we take every day can cause dry mouth?
Many medications have been implicated in dry mouth, such as Viagra, Cialis, and 346 other medications all listed below. Acetaminophen with Butalbital with Caffeine with Codeine Phosphate – Marketed under the brand name Phrenilin.
Albuterol Sulfate with Ipratropium Bromide – Marketed under the brand name Combivent.
Amiloride Hydrochloride with Hydrochlorothiazide – Marketed under the brand name Moduretic. Amitriptyline Hydrochloride Amlodipine Besylate with Atorvastatin – Marketed under the brand name Caduet. Amlodipine Besylate with Benazepril Hydrochloride – Marketed under the brand name Lotrel. Amoxicillin with Clarithromycin with Lansoprazole – Marketed under the brand name PREVPAC. Amphetamine Aspartate with Amphetamine Sulfate with Dextroamphetamine Saccharate with Dextroamphetamine Sulfate – Marketed under the brand name Adderall. Aspirin with Caffeine with Orphenadrine Citrate – Marketed under the brand name Norgesic.
Atropine Sulfate with Benzoic Acid with Hyoscyamine with Methenamine with Methylene Blue with Phenyl Salicylate – Marketed under the brand name Urised. Atropine Sulfate with Hyoscyamine Sulfate with Phenobarbital with Scopolamine Hydrobromide – Marketed under the brand name Donnatal. Belladonna Alkaloids with Hyoscyamine Sulfate with Methenamine with Methylene Blue with Phenyl Salicylate with Sodium Biphosphate – Marketed under the brand name Urimax. Brompheniramine with Phenylpropanolamine with Codeine – Marketed under the brand name Dimetane-DX.
Butabarbital with Fluoxetine Hydrochloride with Hyoscyamine Hydrobromide with Phenazopyridine Hydrochloride – Marketed under the brand name Pyridium.
Carbinoxamine Maleate with Dextromethorphan Hydrobromide with Pseudoephedrine Hydrochloride – Marketed under the brand name Balamine. Cetirizine Hydrochloride with Pseudoephedrine – Marketed under the brand name Zyrtec-D.
Desloratadine with Loratadine with Pseudoephedrine Sulfate – Marketed under the brand name Clarinex-D. Diltiazem Hydrochloride – Marketed under the brand names Cardizem, Dilacor, and Tiazac.
Enalapril Maleate with Hydrochlorothiazide – Marketed under the brand name Vaseretic. Ephedrine Sulfate with Hydroxyzine Hydrochloride with Theophylline – Marketed under the brand name Marax. Fluocinolone Acetonide with Hydroquinone with Tretinoin – Marketed under the brand name Tri-Luma.
Fluticasone Propionate with Salmeterol Xinafoate – Marketed under the brand name Advair. Hydromorphone Hydrochloride – Marketed under the brand names Dilaudid and Dilaudid-HP. Testosterone – Marketed under the brand names Androgel, Testoderm, Striant, and Androderm. Triamterene with Hydrochlorothiazide – Marketed under the brand names Maxzide and Dyazide.
If you know of any other drugs that cause dry mouth or want to share your experiences with any of the above drugs and if it caused your mouth to get dry, please leave a comment below. It goes on to say that medications are probably what cause much of the dry mouth that we see in older adults. As a side note, it also says that there are over 500 medications that have been reported to produce dry mouth as a side effect… Maybe I’ll have to do more research and come up with a bigger list for a future post!
I take 9 of the medicines listed and one more the pharmacist warned might cause dry mouth, trazodone.
Thank you for such a great site with so much useful information– I have posted it on facebook in hopes others will visit and learn from it. I started this site to help you have a better understanding of dentistry so that you can make informed decisions regarding your dental treatment.
Pulpitis: What’s the Difference Between Reversible Pulpitis and Irreversible Pulpitis? Hair loss resources, information, and online support forum for women experiencing all forms of alopecia and hair loss. It’s easy to get lost in the maze of tests and blood panels that your doctor should require to diagnose the cause of your hair loss.
Many dermatologists will recommend Rogaine (minoxidil) immediately without spending time reviewing bloodwork and ordering blood panels. Simple blood tests to be ordered if you think your hair loss may be caused by a lack of nutrients in your diet. A lot of hairloss specialists believe that one needs a level of ferritin higher than 40 in order to maintain hair and ferritin above 70 to regrow lost hair. Blood tests that you should have done prior to and during the administration of anti-androgenic medications such as spirolactone. These tests can also aid your physician in giving you a definitive answer as to what is causing your hair loss. These are tests you should have done to rule out adrenal deficiencies which can cause hair loss as well as fatigue, joint and scalp pain.
These are ESSENTIAL blood tests to have done if you believe that you may be experiencing hair loss due to hormonal imbalances. For premenopausal woman the lowest estradiol value should be shown on the test taken on the 3rd day of her cycle (the week of her period).
If the value is above 50, it could potentially cause the situation of adrogen sensitivity, but the more important number is the one for free testosterone. DHEA and DHEAS: Some doctors report finding that a high percentage of patients with autoimmune disorders are also deficient in DHEA, and should be tested. Diabetes is a common cause of hair loss due to the build-up of blood sugar in the body when it is not controlled.
Telogen effluvium (TE) is the second most common form of hair loss most dermatologists see. Sometimes a TE shed, as our forum members frequently call their thinning hair loss, can last for months or even years. Sometimes skipping a shampoo for a day will make it seem as though more hair comes out the next time you wash. Apply a light conditioning cream to your hair after towel drying to protect it from unnecessary breakage. Visit a professional hair salon professional in your area and request a cut that will give your hair more bounce and move lightly (generally just below the chin and lightly touching the shoulders). Loosely piling your hair up on top of your head and then piecing random pieces of hair with a good hair texturizer makes thinning hair look healthy and thick. It’s important to remember that one supplement that worked for one woman may not work for you.
A female alopecian named Chrissy has her eyelashes and eyebrows done by a professional makeup artist. I did NOT include BCPs or HRT on this listing because I’m actively working on a separate listing for these. Hives (or urticaria) are itchy, erythematous (reddish), often elevated skin lesions that blanch with pressure. Hives come in all sizes and shapes, from tiny 2-3 mm to large irregularly shaped hives covering most of the body.
Individual inflammatory skin lesions persisting for days and resulting in lasting discoloration or hyper-pigmentation of the skin are probably not hives, but a result of some other inflammatory disorder (e.g.
Fortunately, even if an underlying cause cannot be identified, the majority of patients with hives can be successfully managed with a combination of medications and other preventive measures. Angioedema often results from a similar release of mediators, involving the skin’s deeper cutaneous and sub-cutaneous tissues.
Hives that occur over limited periods of time, lasting hours, days, or occasionally, a few weeks are known as acute urticaria. Medications can cause hives soon after exposure or several weeks following the introduction of a new drug. Unfortunately, there are no reliable allergy skin tests or laboratory tests specifically designed for identifying drug allergies.


Food-induced hives are often obvious, as they evolve shortly after eating the suspected food. Less commonly, a food eaten on a regular basis may contribute to chronic hives in a more subtle manner. Moreover, the offending allergen may not be the food, food additive or preservative at all, but a contaminant in the preparation process. Hives and swelling triggered by a physical stimulus to the skin are known as physical urticaria.
Cholinergic urticaria consists of tiny wheals (2-3 mm) surrounded by a large border of erythema. Pressure urticaria is characterized by a delayed swelling that occurs 4-6 hours after pressure has been applied. A sling placed over the forearm or shoulder with a 10- or 20-pound weight for 10-20 minutes is an accurate test for this condition. Aquagenic urticaria involves an outbreak of hives following contact with water that is not temperature-dependent. Cold urticaria is characterized by the rapid onset of pruritus, erythema and swelling following exposure to a cold stimulus. Biopsy of hives which last more than 24 hours may reveal a necrotizing vasculitis involving small vessel infiltrates.
Multiple insect bites can lead to a pruritic, local papular rash known as papular urticaria.
For many physicians and patients, the management of chronic urticaria is frustrating because of the lack of an identifiable cause, and the variable response to medical therapy. Patients with chronic idiopathic urticaria usually have normal blood work and no signs of underlying systemic disease.
This is a form of periodic fever, a rare form of cold intolerance inherited as an autosomal dominant trait. When upper airway angioedema does not respond to medical treatment, a tracheotomy is the procedure of choice to restore a patient's airway. Angioedema, or deep swelling of the soft tissue, may be a result of drug allergy, food allergy, bee sting allergy, complement disorders, or autoimmune disease as well as other uncommon or rare disease processes (see table). A family history of angioedema might point to a unique disorder known as CI-INH deficiency, or hereditary angioedema (HAE). Non-sedating antihistamines such as loratadine (Claritin), desloratidine (Clarinex) and fexofenodine (Allegra) are best administered in the morning, in order to avoid drowsiness and impairment of function as seen with first-generation antihistamines. Some patients are fortunate and their urticaria can be controlled with a single, daily dose of the non-sedating or minimally sedating antihistamines, however, many patients with chronic urticaria require multiple antihistamines, often at higher than conventional doses. Doses of hydroxyzine or diphenhydramine between 25 and 50 mg up to four times per day may be required in order to provide adequate control of symptoms. Allergic angioedema resulting from mast cell mediator release and inflammation often responds well to corticosteroids. An infusion of C1-INH concentrate will quickly resolve active symptoms of HAE by restoring the proper balance of inhibition of this system. In summary, chronic urticaria and angioedema are varied in their mechanisms and presentations and will often require comprehensive investigations in order to identify a cause. Some of the older tricyclic antidepressants (such as imipramine or amitriptyline) may contribute to heartburn by slowing the speed at which the stomach empties, Dr. The material in this site is intended to be of general informational use and is not intended to constitute medical advice, probable diagnosis, or recommended treatments. It’s important to speak to a doctor about possible side effects of medication and before you stop taking any prescriptions. When I got home I looked up my medications on the internet and found out that most of my medications causes dry mouth. It’s okay to remain assertive with your doctor and request a full series of blood tests to rule out any underlying conditions that could be causing your hair loss.
Even though there’s no direct link to hairloss, a high cortisol value means the body is under a lot of stress and the adrenal gland is working overtime. Occasionally, it will appear as if the shedding occurs along with your menstrual cycle (cyclical shedding). Many women who have recovered from telogen effluvium agree that you should steer clear of unnecessary supplements unless you have had blood tests to diagnose any vitamin deficiencies that can contribute to your hair loss.
Our bodies are unique and unnecessary supplements and medicines may do more harm than good. In this case, more edema (fluid accumulation and swelling) is present than that seen with hives.
By definition, acute urticaria persist for six weeks or less, whereas chronic urticaria persist for greater than six weeks. In either case, a thorough history and comprehensive laboratory studies offer the patient the best chance of defining the cause of the hives. Drugs commonly associated with urticaria include penicillin and their derivatives, sulfonamides, analgesics (aspirin, NSAIDs), radiocontrast media, sedatives, tranquilizers and diuretics.
Although drug-induced angioedema is often temporally linked to the drug triggering the reaction, ACE inhibitors are the exception to the rule. Non-immunologic induction of hives occurs by direct mast-cell degranulation and does not appear to be IgE-mediated. Diagnostic studies including allergy skin testing, lab testing (RAST or Immunocap™), elimination of the suspected food, or the double-blind food challenge (the gold standard) may be necessary to confirm the diagnosis. In any case, a careful food history in combination with diagnostic testing or challenge performed by an allergy specialist can often isolate the triggering food product.
Examples include systemic lupus erythematosus, cancer, vasculitis, Sjogren’s Syndrome, rheumatoid arthritis, and cryoglobulinemia. Examples include viral infections such as infectious hepatitis and mononucleosis—both of which are self-limited and fungal infections including thrush, tinea pedis, and tinea capitis. It is thought that physical stimulation (light, heat, cold, pressure) leads to a change in local IgE that then stimulates mast cells to liberate mediators that cause the itching and hives. The diagnosis can be confirmed by applying a test tube of warm water (44 degrees Fahrenheit) to the arm for 4-5 minutes to check for the evolution of hives. Hives result from exposure to certain wavelengths of light and typically are limited to sun-exposed areas. The diagnosis is confirmed by placing a compress with distilled or tap water against the skin for a few minutes (the water should be tepid).
Individual hives resolving within 24 horus without inflammation are rarely a sign of vasculitis. This condition is often observed on the skin of the lower extremities and may be due to mosquitoes, fleas, chiggers, and other insects that attack the exposed skin. The rash consists of slightly elevated brownish macular lesions resulting from concentrations of mast cells on the surface of the skin. Less than 25% of individuals with chronic urticaria have an identifiable cause of their rash. Some studies have shown that up to 40% of such patients appear to have an autoimmune cutaneous disorder. Cold exposure causes a burning rash, fever, chills, arthralgia, myalgia, headache, and leukocytosis. It doesn’t respond to epinephrine, steroids, antihistamines or leukotriene antagonists, usually effective in treating allergic angioedema. The good news is that C1-INH concentrate infusion had been available in Europe for decades and is now approved for use in the United States.
Finally, low C1-INH levels and resulting angioedema may be the result of an underlying autoimmune disease or cancer in which the underlying disease inactivates C1-INH and effectively creates the same clinical symptoms as seen in hereditary angioedema. In contrast to urticaria, angioedema is often not pruritic (itchy), but rather, can be painful and tender. Zyrtec (Cetirazine) and Xyzal (Levocetirizine) are minimally sedating and appear to be well-tolerated by most patients. Physicians and patients must be mindful of the possible sedating and anticholinergic side effects of these older agents and take the necessary precautions. Leukotriene receptor antagonists such as Singulair (montelukast) have proven effective in some patients with chronic urticaria and angioedema, apparently by blocking the leukotriene pathway, which plays a role in mast cell-induced inflammation. Alternative treatments are available, however, their efficacy is highly variable, and they are not approved for general use. When upper airway angioedema of any type does not respond to treatment and the airway is significantly obstructed, then a tracheotomy is the procedure of choice. Fortunately, even those with chronic conditions without a clear cause often respond to carefully planned maintenance programs and emergency backup, which will ultimately lead to a better quality of life. See the Terms of Service and Privacy Policy (Your California Privacy Rights) for more information. I have polymyalgia rheumatica, giant cell arteritis, chiari malformation type 1, elhers-danlos syndrome type 3, obstructive sleep apnea, chronic migraine, essential tremor, chronic vitamin b-12 deficiency, nephrolithiasis, pednisone-induced hyperlipidemia, diabetes, and ocular hypertension.
Not one of my pamplets about my medications said one word about dry mouth let along anything that dry mouth will do to your teeth!!


Often times, a simple hormonal imbalance or vitamin deficiency can be discovered early with a blood test and you will be well on your way to restoring your hair. One thing to notice is that TIBC above the norm (above 400) means the lack of iron in the iron stores. Values above 3 are still considered normal by many labs (the upper level of normal is 5) however it usually indicates an overactive thyroid and should prompt for future evaluation.
I decided last year I was tired of the wigs and showed the world my bald head with a little peach fuzz. Women with TE never completely lose all their scalp hair, but the hair can be noticeably thin in severe cases. For example, if you are iron deficient or anemic, you should take a doctor recommended amount of iron supplements. Such rashes include erythema multiforme, insect bites, cutaneous lymphoma, dermatitis herpetiformis, atopic dermatitis, porphyria, contact dermatitis, and dozens of other skin conditions. In contrast, chronic hives recur over weeks, months and even years, but they can usually be controlled with continuous use of medication. The cause of acute urticaria is often obvious because of the rapid onset of hives following exposure to a specific trigger. Drug-induced urticaria are often identified by elimination of the offending drugs with resolution of symptoms within days. If it is medically necessary to continue a suspect medication, consideration should be given to replacing the medication with a suitable alternative of an unrelated drug class. Angioedema resulting from ACE inhibitors can occur months or years following the initiation of treatment and may only occur sporadically, thus obscuring the relationship between cause and effect. Medications such as Vancomycin or opiates (morphine, Demerol, codeine) can cause hives by directly stimulating the mast cell to release histamine and other mediators without IgE or complement involvement. Such additives include: food coloring agents, preservatives (antioxidants, anti-browning agents), emulsifying agents, stabilizers, gums, acidulants, enzymes and leavening agents. A skin biopsy of a persistent hive can help diagnose underlying vasculitis by revealing the presence of necrosis of blood vessels, leukocyte infiltration, and deposition of immunoglobulins and complement.
Many helminthic infections (roundworms) can cause chronic urticaria and include infestation of: ascaris, ancylostoma, strongyloides, filariae, echinococus, schistosoma, trichinella and toxocara. However, a biopsy reveals perivascular mononuclear cell infiltrates and dermal edema commonly seen in urticaria. Pressure urticaria often affects the soles of the feet after standing for long periods of time, especially on hard, irregular surfaces. When these macules are stroked, mast cells release mediators which trigger the local appearance of hives. These patients suffer from an autoimmune disease in which auto-antibodies are directed at the IgE receptors on mast cells causing release of mediators which induce hives. A decreased quantitative C1-INH level is diagnostic, however 15% of these patients may have normal quantitative C1-INH levels that are defective, requiring a functional assay for C1-INH to confirm the diagnosis.
Instead, the stimulation of the hepatic production of C1-INH by the use of anabolic steroids such as Stanazole is used to prevent attacks.
Allergic triggers of angioedema are typically IgE-mediated and are often easily identified based on the demonstrated temporal relationship between exposure and development of symptoms. Histamine released from mast cells is the primary chemical mediator involved in the development of urticaria. Some of the alternative treatments presented in medical literature include: intravenous gammaglobulin, methotrexate, Plaquenil (hydroxychloroquin), cyclosporine, (omalizumab, Xolair) and UV light therapy.
I have been told that I need all my teeth removed and I have extra jaw bone that will have to be removed by surgery as well as my three broken teeth.
Angioedema of the GI tract can cause severe abdominal pain, while angioedema of the larynx or tongue can lead to respiratory distress.
These infections are usually associated with significant eosinophilia and elevated levels of IgE. An intradermal injection of methacholine reproduces these tiny hives and is diagnostic of this disorder. Treatment consists of low-dose corticosteroids (15-25 mg of prednisone every other day), since antihistamines and other common urticaria medications have proven ineffective. Hives occur often occur along common areas of physical pressure (waist, sock line, bra line, etc.). Systemic vasculitis with hives often has elevated sedimentation rates, arthralgia, fever, etc. Uritcaria pigmentosa may consist of a few isolated lesions or dozens of macules over large areas of the body.
Biopsy shows no cellular infiltrate and the edema results from the release of vasoactive factors such as kinins. Episodes of angioedema related to this disorder can be severe and life-threatening and respond poorly to conventional therapy.
Antihistamines competitively bind histamine receptor sites in the skin, thus suppressing the development of hives and the related pruritus (itching).
However, I was taking Chondroitin and Glucosamine which I researched and found one of the side effects is dry mouth.
I had the decompression surgery in September and despite complications am doing pretty well now!
I think the medication companies should be held responsable for not informing people what their products and that they cause dry mouth and what dry mouth will do to your teeth!!!!
I hope they come up with a treatment that works, but if not, I have come to grips with the fact that I may never have a full head of hair again. Consequently, food challenges are often the only means of identifying the role of these hidden additives. There are different types of solar urticaria, which are classified according to the wavelength of light that induces the eruption. Therefore, individuals with cold urticaria should always swim with a buddy, and an Epi-pen™ should be kept readily available. Cutaneous vasculitis is often associated with low complement levels (hypocomplementemic vasculitis), in which the vasculitis is limited to the skin without other systemic involvement. In the event that a cause can be identified, the trigger can either be eliminated or managed more effectively with a focused, therapeutic approach.
Further, the skin biopsy reveals an increase of CD4+ cells, neutrophils, and eosinophils, similar to that seen in late-phase allergic reactions. Most patients will experience significant relief of their hives with an oral corticosteroid. HAE and acquired C1-INH deficiency-related angioedema is often difficult to treat with poor a response to epinephrine and other corticosteroids.
After being off all over the counter medications for a month and still experiencing dry mouth and eyes, my eye doctor prescribed cevimeline which has greatly helped my mouth and eyes! However, the condition may be classified as idiopathic when no identifiable cause can be determined. Many cases of cold urticaria are IgE-mediated, but cold urticaria may be associated with other diseases including: cryoglobulinemia, cold agglutinin disease, cryofibrinogenemia, and paroxysmal cold hemoglobinuria. The treatment of choice is with the antihistamine cyproheptadine (Periactin) for cold urticaria. When lesions cover most of the skin surface, it is referred to as diffuse cutaneous mastocytosis. Some patients will require low-dose daily treatment (5-10 mg daily) while others can benefit from alternate-day treatment in which a low dose is given every other morning. By the middle of the night my cheeks are stuck to my teeth, my lips can’t form a seal on a straw, and my throat is so dry I literally can’t swallow!
Sunblock containing zinc oxide, titanium oxide, or both are particularly effective in avoidance treatment.
Alternate-day treatment with corticosteroids allows control of hives without the potential for steroid dependency associated with long-term use of daily steroids.
My lips are parted about a quarter of an inch and air from the c-pap machine is flowing out when I wake.
Extreme caution must be exercised with the administration of short courses or chronic use of oral corticosteroids. Symptoms may be limited to intense itching or may evolve into generalized hives, which ultimately subside following cessation of exercise. A severe form of this phenomenon is that of exercise-induced anaphylaxis in which the patient becomes hypotensive and breaks out in hives. This latter form of exercise-induced hives is preventable in some patients by avoiding meals for 4 hours prior to exercise, which indicates that it may be a food-dependent reaction. Pre-treatment with an antihistamine is recommended, along with maknig sure an Epi-pen™ is on hand.



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