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admin | Herbal Remedies For Bv | 07.01.2015
Images on this website have been digitally watermarked with ownership and usage information. I'd like to write a meaningful update about Fred, but the truth is I've become incapacitated by this horrific situation.
It feels like a month's worth of time has passed over the last 10 days since Tater Tot first fell ill.
I have to take on the responsibility for ALL decisions for our foster cats and trying to do it from 1000 miles away is grueling. I took a Bottle Baby Bootcamp class at Tabby’s Place a few months ago and it dawned on me that Baby G.
The next 24 hours were spent in a mad dash to see if there was anything we could do to save Tater's life. So we ran tests, even though Polly fussed and screamed, being the sickest, we decided to do the tests on her.
While I waited for test results to come back, I spent time with the kittens, and their mom, Mazie, too.
Sometimes URIs become chronic or can reoccur very frequently with the consequence that bad breath becomes very persistent too. Chronic or recurrent URIs are in fact one of the causes of chronic fetid tonsillitis (or chronic caseous tonsillitis), otherwise known as tonsilloliths, and commonly named tonsil stones.
In this post I will talk about about what tonsil stones are and how they form, and I will explain the relationship between tonsil stones and bad breath. The tonsils are naturally filled with crevasses (called tonsillar crypts) where bacteria, fungi, dead cells, mucous, food particles, and other foreign objects get trapped.
In the case of a lump of food for instance, the process starts with digestive enzymes present in saliva breaking down the trapped food. More layers may be added, such as layers made of dead cells that used to form the lining of the mouth for instance. Tonsil stones are a therefore a concentrically laminated pattern, composed of many different substances and organisms. In fact, several studies show that tonsilloliths are actually a living biofilm (similar to dental plaque). The characteristic pungent smell is mostly due to the volatile compounds produced by the anaerobic bacteria located near the nucleous.
Most people with healthy tonsils have very small tonsil stones forming more or less regularly inside their tonsil crypts (rarely larger than about 1mm in diameter).
These small tonsilloliths can be accidentally released when laughing, singing, talking, coughing or performing any activity that involves squeezing the tonsils.
Although it is quite difficult to determine exactly why some people are prone to the formation of large tonsil stones, there are indeed a few factors that play a significant role.
The same applies when the crypts are not necessarily big but very numerous; the tonsils may have a higher than average amount of pits, folds and tunnels, where debris, mucous, etc can easily get trapped. In most cases, if the tonsils are working adequately and are not scarred with large crypts, foreign bodies and micro organisms will either be eliminated or get killed by the white cells inside the tonsils.
Even when small tonsil stones are formed, these will eventually make their way out of the body when accidentally coughed, swallowed (down the stomach and eventually expelled), etc. In addition, healthy tonsils are quite likely involved in the regulation and control of the amounts and types of bacteria present in the mouth. Unfortunately there is not much research done in this area yet and I would advise you not to make any radical changes in your diet unless you have spoken to your family doctor or a dietician first. These excessive amounts of bacteria will quickly colonise the tongue and the tonsils as well. In the same way, having poor oral hygiene can have the same effect on how healthy your tonsils are. There may be several factors involved that can encourage tonsillolith buildups during pregnancy. However, you may have larger than average tonsil crypts with exposed openings, and perhaps you tend to eat a lot of dairy as well; these two factors may be sufficient to guarantee the frequent formation of tonsil stones and the presence of chronic bad breath as a consequence. Therefore, it is reasonable to think that the more tonsil stones your tonsils contain at any given time and the larger they are, the higher the risk of suffering from bad breath. It is clear that tonsil stones and bad breath go hand in hand, and so it is important that you determine whether your tonsils are prone to gathering debris, so you can establish if you suffer from tonsil stones and bad breath. Hi, I have suffered from tonsil stones and bad breath for about 5 years or so (although I did not know until very recently that my halitosis is caused by tonsil stones). Very interesting post… I have wondered for a long time whether or not there was a correlation between URIs and bad breath, and it seems I was correct. I had no idea what tonsil stones were until my friend showed me her tonsils and how she got them out.
Since I first broke the news a few days ago that Tater may have FIP, his foster mom Maria and I have been working around the clock trying to find out if this diagnosis is accurate.
Opal wasn’t caring for her kittens and we weren’t even sure if she had any milk to give them if she could.
Not only do I emotionally support our volunteers, I have to help them make difficult choices and I have to KNOW what I’m talking about to do that…which would be fine IF I knew what I was talking about. I pushed back, saying the kittens were chronically ill, BUT they were thriving, in some way, too. I've asked so much of everyone that I can't even bear to ask now, so I'll just post this ChipIn and if you can help the kittens, that would be very much appreciated.
If I can get them over this, then I can start the happier task of finally finding them good homes. However, tonsil stones and bad breath are often present in very healthy individuals with no history of chronic or recurrent URIs.
They start maybe with a small lump of food or a bit of mucous which gets trapped in one of the crypts; then successive layers are created on top of this initial lump (stratification).
But what is important to notice here is the fact that tonsil stones are absolutely crammed with bacteria; literally layer upon layer! There are both aerobic and anaerobic layers of bacteria present in them, which calcify over time. However, smaller tonsil stones are very common and they produce the same foul odours as the big ones. Usually these are not numerous nor big in size and are hidden pretty deep somewhere inside the labyrinthine net of nooks and cranies that form the tonsils.
There are factors that may increase the speed at which tonsil stones form and other factors that may not only encourage tonsil stone formation but also impair adequate elimination of the calcified matter before it becomes too large. Pus, inflammation, crypt enlargement, presence of excessive amounts of bacteria, etc, all create an environment more prone to tonsil stone formation.
Mucous attracts bacteria, and so tonsilloliths can rapidly form when large amounts of mucous and bacteria regularly make their way inside the crypts. When saliva levels decrease, either chronically or temporarily, debris in the mouth sticks more easily and it can build up in the tonsils.
They do so by filtering foreign viruses, bacteria, debris, etc, and subsequently attacking them via the white cells. However, there seems to be anecdotal evidence that consumption of dairy products can increase the frequency of build-up of tonsil stones in the crypts.


Sensitivities or intolerances (without being necessarily an allergy as such) can cause the palatine tonsils or adenoids to enlarge and swell, as well as increased mucous production (and congestion), post nasal drip etc. The tonsils are part of the immune system, and so they will not function adequately if your immune system as a whole is weak.
Therefore, an unhealthy oral flora may be a contributing factor to the formation of tonsil stones, since a poor chemical balance in the mouth may make them grow at a faster pace.
Both food particles and bacteria can quickly build up inside the tonsils if the mouth is not properly cleaned after each meal. For instance, you may be very healthy, have excellent oral hygiene, with no allergies, sinus or post-nasal drip problems, nor any chronic throat infections at present. I have kept it hidden from my girlfriend for a long time, but she keeps mentioning the bad breath and I feel too embarrassed to tell her it is because of those little buggers. Asian countries,[18] United States (including Hawaii), Canada ,[19] and Scotland)[20] due to bacterial resistance.
The "Digimarc-Enabled" Web Button is a trademark of Digimarc Corporation, used with permission. I'm focused on providing round-the-clock care for Fred, for arranging his next test or Vet appointment and for finding a way to pay for it all.I'm sick to my stomach. Three days after we began treatment, Tater's temperature dropped and by day four, his temperature was within a normal range.
We knew that the mortality rate for newborns is 30-40%, but it didn’t make what happened any easier. Cyndie was massively sleep deprived and stressed out and didn’t feel this was a good option and that it could hurt the kitten. Over the past day, Opal had started to produce milk and eat a great deal of food, indicating that her milk production was strong. The kittens are just NOT “getting over the hump.” They get mildly better for a day or two and seemingly overnight they get very sick.
She has so much mucus in her, if she eats too fast, she regurgitates her food, then hysterically tries to eat it back up, only to pass it out again. Stool sample showed positive for a type of coccidia I haven't heard of and I will have to report on exactly what this was later since it escapes me just now.
For kittens who've been sick almost every day of their short lives, it really would be remarkable to see them healthy-at last. Bacteria will then start colonising the area, as they will feel very attracted to these remains, and so they will form another layer on top of the broken down food.
And this is the main reason why tonsil stones smell so putrid; in particular, the anaerobic bacteria which create volatile organic compounds are responsible for most of the unpleasant odours. The consistency evolves from a soft gel to a hardened stone, as layers are formed and the materials calcify.
Most people are not aware that their tonsil crypts are full of debris (not necessarily hardened yet in the form of a calcified stone) which is causing their halitosis. Usually, the small size of the crypts and the fact that any small stones that may form are likely to be eventually released and swallowed, guarantees that no large tonsilloliths can ever form. Tonsils are not moisturised as they should be and all these factors are thought to increase the risk of tonsil stone formation. Note that this is very rare (even in severe cases of acid reflux, food particles or acids do not normally reach the tonsillar area). I do have a problem with recurrent throat infections and my tonsil crypts do look enlarged. I think my tonsils are scarred and are collecting stuff in their folds and crannies… Plus I suffer from post nasal drip, although I keep it under control now with regular oral irrigation.
It seems every moment something changes and throws us into a tailspin of fear that this IS FIP.
A few days later, we took in his sister, Latte and the two of them stayed together in Maria’s home. Cyndie had a tough time intervening, but eventually was able to start bottle feeding the neonatal kittens to ensure they were getting some sustenance.
I started a frantic search to find a nursing mama cat we could either rescue (and take any of her kittens, too), or one we could put our four kittens with. Cyndie chose to only give her to the two most critical kittens, a boy who was born first, and a girl who was born last (Opal wouldn’t even clean off the amniotic sack on this kitten, who Cyndie initially feared was dead).
I had to try to draw from my own reserves to help her have faith that tube feeding was the best and possibly last option for Baby G.
I think everyone on Facebook started to feel like all their prayers and hopes were working. For those of you not familiar with Feline Infectious Peritonitis, let's just say that it's a death knell for any cat who gets it.
Yes, they were sick with some sort of horrible URI that wasn't responding to MONTHS of medications, but could it be FIP? In addition, white cells present inside the tonsils will also attack this mix of materials and surround it.
In addition, I have many allergies which cause a lot of mucus to drip down my throat (getting in the tonsils I guess). I am now kind of obsessed with them and always try to get them out so I don’t have bad breath! I have visions of having to euthanize a 10-month old KITTEN because I can't find an answer to what has been slowly robbing him of his neurological function. Tater on the way to the Vet yesterday.On day four of treatment, Tater began to eat on his own.
It's highly contagious, as well, which means I could have put ALL MY CATS, even Bob Dole, at risk for getting this incurable disease. She is so tiny, with such big owl-green-eyes, I can't help but want to do whatever I can to save her life. I never really thought for a minute that my tonsils could be the cause of my bad breath, as the tonsilloliths are not visible. By day five, Tater gained back some of the weight he lost.We brought Tater back to the Vet for a re-check and to discuss what sort of testing we should consider doing.
Tater waiting to see the Vet.Being concerned about FIP, Maria moved Latte into a separate cage. I don’t want to hear the sound of my phone ringing or the tone that indicates I have an incoming text message.
Opal seemed to be feeding them, but no one was really sure since getting close to Opal meant getting clawed.
The bottle feeder who was helping Cyndie had to rush to the hospital because her Mother had a bad gallbladder attack and had to have emergency surgery.
She perked up and they thought she was going to improve so we made plans for them to keep her at the Vet partly so Cyndie could rest and partly so we could be sure Baby G. I asked if she de-wormed him and she said she had a few days ago when she first noticed his belly getting round.
I have now learned how to clean my tonsils properly and my girlfriend has assured me that my breath has improved dramatically since I started applying all your tips. We have a suspicion Tater has Bartonella, which is now called Mycoplasma haemofelis ("feline infectious anemia").


As we found Tater, laying on the pavement, sick and flea infested a few weeks ago.I flat out don't know. We just had to keep her fed and keep an eye on the kittens, but she would do the rest for the next few weeks. The guide was very helpful by the way, not only to get rid of my tonsil stones, but it also opened my eyes about bad breath products, and I also learned a lot about the other causes of my bad breath. This is the first case of FIP I've ever encountered (again, IF that's what is ailing Tater).
I didn’t know must about FIP, but I knew it was fatal and I knew diagnosing it isn’t clear cut, either.Since Sunday morning, Maria and I have been working non-stop.
I feared Tater was going to die a few days ago, but it's Thursday and he's still with us.I just found out if he has FIP he can live for a few MONTHS like this so any hope I had of it not being the wet form of FIP is dashed again. I’m doing research, trying to understand what to do, what to test for, if there’s new research, anything we can do to save Tater’s life. When Cyndie looked at the little kitten, she realized the other two siblings were MUCH larger and clearly doing much better. Olson.I've cried a river of tears and had to force myself to go into the foster room-a place that once only meant joy to me-to witness seeing my dear foster kitten wobble across the room, trying not to fall over, but flopping to the floor if he tries to go too far.
I found it ironic that the costs for the cremation would be more than what we spent for her Vet care. Granted my life sucks right now, but having to face this would put me over the edge and into a dangerous place.
He is reluctant or unable to eat, so Sam and I have to zip him into a “cat bag” so I can syringe feed him every day and give him his meds-which may or may not do a thing.
Tissue Warning.The Vet didn't feel we needed to do tests just yet, but to continue with treatment though she did agree to change Tater's antibiotic to Doxycycline which would fend off the Bartonella, if that's what he's been battling.
There are no conclusive tests that will tell us more than just levels of exposure to the Coronavirus if he has FIP or if he has Bartonella.For now she wants to stay the course and see how he does. I already have many little tin boxes of ashes and these two babies could rest with my cats, never to be forgotten. Tater is NOT out of the woods, BUT he is also NOT close to death as he was just one week ago today. I am trying to be gentle, loving, sweet, not miss his mouth and squirt the warm fluid all over us.
Sunday before his Vet visit.E-mails poured in, comments on Facebook were all supporting Tater and Maria, saying prayers, wishing us all well, sharing their stories of losing kittens to this unforgiving disease. He thinks we're going to give him the painful eye drops again or shove a pill down his throat. Today we opted to do a bile acid test, in the hopes that Fred has a very rare condition called a Portosystemic Shunt. Fred's neurologist felt it was worth investigating and it IS treatable if it's causing the problems. Olson.They wanted to look again, with a second ultrasound, at Fred's mesenteric lymph nodes. There are whisperings of advancements in the treatment or cure of FIP, but so far the only hope is one study says they saved 20% of a small number of FIP cats who had the dry form. Not really because now they're seeing a faint “glow” around his kidneys which could mean LYMPHOMA, but of course it's not definitive. Nothing seems to be at this point.Tomorrow, April 23rd, they will do an MRI of Fred's brain.
Back home eating normally.Some say that if FIP occurred in puppies this would have been cured 20 years ago.
Cats have been “second class” animals for far too long.I’m glad that UCDavis is doing a major study and Vets like Dr. Tater’s belly is full of fluid-so much so that an xray couldn’t even see his organs through the liquid. If those tests are normal they will look again at the fluid to see if there is toxoplasmosis or cryptococcous-even though his blood showed no signs of it-they can some times find it in the spinal fluid. Those two things we CAN treat for and cure, but lymphoma and FIP are a death sentence for Fred.
We were going to run a titer, but after doing more reading, it was clear we didn’t have time for the results and it wouldn’t really tell us anything. We could run a test on the protein levels in the fluid and that would be a better indicator, but what is the point? We may never know what is happening so we'll do what most vets do-give Fred steroids and hope the inflammation reduces and he gets some function back in his limbs, that he has better quality of life for some amount of time. I must help Maria, who has to face the difficult question of having to help Tater pass away when he gets too sick. It's the ONLY drug known to POSSIBLY, in some very few cases, give cats with the dry form of FIP a greater chance to live longer and more comfortably. I found out about a rescuer who is using a different method, who swears she is having success turning kittens around who have FIP. Some cats have lived over a year with the therapy and one cat has lived over 5 years.Excuse my rambling, awful post.
I'm in a very bad place, terrified, hoping that somehow, some way, Fred gets a miracle and we can cure him.
It feels very unlikely right now, but foolish me, I will hold onto the few strands of hope I have left. I know it’s not traditional western medicine, it’s eastern medicine which is based on 4000 years of study.
If Fred has to die, he will die at home with his brother, Sam and I by his side, if we have anything to say about it.
I can’t disregard that and I’ve had success using different treatments for upper respiratory and skin ailments. Tater is going to die, anyway, so I want to try.The problem is getting the medicine to Maria in time. They only got about 1cc out of him-not nearly enough to make a difference.After another endless number of texts, calls and e-mails, Maria opted to take Tater home. If he continues to be uncomfortable, she’ll bring him back to the vet to get a shot of Depo (steroids).
If this person says she’s helped quite a few kittens with FIP maybe there is a bizarre chance.
We're doing our best, all we can think of…but it may not be enough.The problem is NO ONE KNOWS. The stress of not knowing, of trying to make the BEST choice for Tater, has gotten to Maria and myself.
We’re trying so hard to keep him from coming in the door.If we could get Tater to live until tomorrow, perhaps this stuff will work or we’ll have to say farewell to a very sweet little guy.
After the stress of two weeks of being sick and stressed out of my mind about flying before I flew to Salt Lake for BlogPaws, then the stress of what to do with Opal’s kittens and now the troubles with Tater…it just feels like way too much, but I can’t give up for Tater’s sake.



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