Medicine for bacterial skin infections,how to remove the odor in armpit,how to get rid of discoloration from acne naturally inside,best way to get rid of sweat stains on hats 2014 - PDF Review

admin | What Cures Bv | 27.01.2014
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Sign up to stay informed with the latest skin care and conditions updates on MedicineNet delivered to your inbox FREE! Contact dermatitisContact dermatitis is a rash that is brought on either by contact with a specific chemical to which the patient is uniquely allergic or with a substance that directly irritates the skin. Oral candidiasis One form of oral candidiasis, thrush, appears as white to gray patches (pseudomembranes) on the tongue, soft palate, gingiva, oropharynx, and buccal mucosa.
Chronic hyperplastic candidiasis (candidal leukoplakia) consists of irregular, white, persistent plaques on the tongue or mucous membranes that are difficult to remove; this form of candidiasis occurs especially in male smokers.
Candidal intertrigo Candida infection may occur in any skin fold, causing soreness and itching. Candidal vulvovaginitis and balanitis Most women with candidal vulvovaginitis have no underlying disease, but candi-dal vulvovaginitis may accompany diabetes mellitus and HIV infection. Candidal paronychia and nail infection Maceration of the tissue surrounding the nail, typically caused by excessive moisture, may cause paronychia, which is characterized by erythema, swelling, and pain of the nail fold with loss of the cuticle [see Figure 6].
Figure 5 Prominent satellite lesions of discrete vesicles are seen in a patient with candidiasis. Figure 6 In a Candida paronychia, seen on this patient’s thumb, the nail fold becomes red, swollen, and painful. Scrapings from cutaneous or mucous membrane lesions may be mixed with KOH solution and examined under the microscope for budding yeasts with pseudohyphae. Oral candidiasis For oral candidiasis, topical nystatin suspension, 200,000 to 400,000 units three to five times a day, is usually effective; an alternative treatment is clotrimazole troches. Candidal intertrigo and balanitis Candidal intertrigo and balanitis respond to a topical azole cream, such as miconazole or clotrimazole.
Candidal vulvovaginitis Treatment of vulvovaginitis includes a topical azole in the form of a cream, suppository, or ointment, administered intravaginally, typically once daily for 7 days. Candidal paronychia Patients with candidal paronychia should keep their fingers dry; when wet work is unavoidable, patients should use cotton liners under rubber gloves. Tinea versicolor (pityriasis versicolor) Because the term tinea traditionally refers to dermatophyte infection, some clinicians prefer the term pityriasis, which means scaling, for this yeast infection. Malassezia folliculitis (Pityrosporum folliculitis) In folli-culitis, inflammation of the hair follicle causes red papules and pustules that surround individual hairs. In patients with tinea versicolor, KOH preparations of scrapings from the lesions demonstrate pseudohyphae and yeasts, which resemble spaghetti and meatballs.
Figure 7 Tinea versicolor appears on the chest of this patient as oval, hypopigmented, finely scaling macules. Figure 8 Vesicopustules or bullae of impetigo rupture quickly and leave an erythematous base covered with a thin, seropurulent exudate. Simple treatment of tinea versicolor and Malassezia folliculi-tis involves applying selenium sulfide shampoo from the chin to the waist and from the shoulders to the wrist, allowing the shampoo to dry, and then washing it off after 10 to 15 minutes.
Initially a vesicular infection of the skin, impetigo rapidly evolves into pustules that rupture, with the dried discharge forming honey-colored crusts on an erythematous base [see Figure 8]. The usual cause is Staphylococcus aureus, but sometimes, Streptococcus pyogenes (group A streptococci) is also present; occasionally, S.
Ecthyma (from the Greek word ekthyma, meaning pustule) is a deeper infection than impetigo. Cellulitis and erysipelas are acute, spreading infections of the skin caused by streptococci of groups A, B, C, and G.
Diagnosis Cutaneous findings include rapidly expanding erythema and swelling of the skin [see Figure 9], sometimes accompanied by proximal streaks of redness, representing lymphangitis, and tender, enlarged regional lymph nodes. Figure 9 Erythema, edema, and sharp demarcation of the lesion from the normal surrounding skin characterize facial erysipelas. On the face, the typical location is on one or both cheeks, with a butterfly pattern of erythema and swelling. The diagnosis is largely clinical; in a typical case, cultures are unnecessary and usually unrewarding. Treatment Treatment consists of elevation of the affected area to help reduce edema and administration of systemic antibiotic therapy. In patients with leg cellulitis, treatment of tinea pedis is useful in preventing further episodes, which are likely to cause permanent lymphatic damage and can lead to lymphedema and further risk of infection. Acne develops when cells and natural oils begin to block up tiny hair follicles in the skin.
Some people do fine using one acne cream, although many need a combination of approaches to control their teen acne. Mersch received his Bachelor of Arts degree from the University of California, San Diego, and prior to entering the University Of Southern California School Of Medicine, was a graduate student (attaining PhD candidate status) in Experimental Pathology at USC. Fitzpatrick's Color Atlas & Synopsis of Clinical Dermatology Klaus Wolff, Richard Allen Johnson, Dick Suurmond Copyright 2005, 2001, 1997, 1993 by The McGraw-Hill Companies. Gram stains of the same specimen are easier to evaluate because they disclose very large, oval, gram-positive cocci that may demonstrate budding or pseudohyphal formation. Prolonged topical therapy with creams or solutions of various azole preparations, such as clotrimazole, is often necessary to eradicate the infection.
Repeating this regimen after 1 week is usually effective; reap-plication once every few weeks as necessary should prevent relapses, which are otherwise common. Erysipelas involves the superficial dermis, especially the dermal lymphatics, and cellulitis affects the deeper dermis and subcutaneous fat. Needle aspiration of the lesion yields an isolate in about 5% of specimens, as do blood cultures in febrile patients.
Other measures to diminish the frequency of future attacks include control of edema by diuretics or mechanical means, such as elastic stockings, and, for those with frequent episodes, prophylactic antibiotics. There he was involved in research in radiation biology and received the Huisking Scholarship. This rash is tends to be weepy and oozy and affects the parts of the skin which have come in direct contact with the offending substance.
He obtained his BA degree in bacteriology, his MA degree in microbiology, and his MD at the University of California, Los Angeles. Prose, Leonard Kristal Copyright 2008, 1998, 1990, 1975, by the McGraw-Hill Companies, Inc.
Predisposing factors in adults include diabetes mellitus, use of systemic or local corticosteroids, use of broad-spectrum antibiotics, use of radiotherapy or chemotherapy, and impaired cell-mediated immunity, especially from HIV infection. Candidiasis of the tongue can also take the form of median rhomboid glossitis, a diamond-shaped area of atrophic papillae in the central portion of the lingual surface.
Commonly affected areas include the groin, inframammary regions, and folds of the abdominal pan-nus. With chronic colonization, nail involvement may occur, producing yellowish discoloration and separation of the nail plate from the nail bed (onycholysis).
These organisms are much larger than bacteria and are much easier to see on Gram stain than on KOH preparation. An alternative to suppositories is treatment with a single oral dose (150 mg) of fluconazole, which is at least as effective as topical therapy and is often preferred by patients. The lesions are small, discrete macules that tend to be darker than the surrounding skin in light-skinned patients and hypopigmented in patients with dark skin. The yeast form prevails in fol-liculitis and is easily seen on Gram stain of purulent material from a pustule, appearing as a large, oval, gram-positive coccus that is much larger than bacteria.


With tinea versicolor, scaling resolves promptly, but the pigmentary changes may take weeks to months to disappear. Impetigo characteristically occurs on skin damaged by previous trauma, such as abrasions or cuts. Erysipelas has an elevated, sharply demarcated border, but differences in the clinical appearances of erysipelas and cellulitis are unimportant and often unclear. The cuta-neous surface may resemble the skin of an orange (peau d’or-ange) because the hair follicles remain tethered to the deeper structures, keeping their openings below the surrounding superficial edema and creating the characteristic dimpling of the skin.
Systemic symptoms, such as fever, headache, and confusion, can accompany these infections; sometimes, such symptoms precede by hours any cutaneous findings on examination. The easiest approach is the administration of oral penicillin or eryth-romycin, 250 mg twice daily.
When the body's immune cells move in to attack the bacteria, the results of the battle are the classic symptoms of acne -- swelling, redness, and pimples. The oral retinoid Accutane works differently by reducing the amount of oil secreted by glands in the skin. Physicians might consider that Morgellons and Bartonella are in some way intertwined.??Morgellons sufferers report an array of symptoms happening just under the skin, such as the emergence of fibers and filament clusters, specks, particles and sand shaped granules, yet physicians are unaware of this phenomena because they are not using enlargement devices. Common examples of allergic contact dermatitis are poison ivy, poison sumac, poison oak (same chemical, different plant) and reactions to costume jewelry containing nickel.Diaper rashThis is a common type of irritant contact dermatitis that occurs in most infants and some adults who wear diapers when feces and urine are in contact with skin for too long.
Acute atrophic candidiasis especially follows antibiotic therapy and causes painful, red, denuded lesions of the mucous membranes; the tongue may have ery-thematous areas with atrophic filiform papillae.
The lesions are patches of bright erythema accompanied by maceration and an irregular, scalloped border, beyond which papules and pustules (satellite lesions) commonly form [see Figure 5]. Male sexual partners of women with candidal vulvovaginitis—especially male sexual partners who are uncircumcised—may develop balani-tis, characterized by erythema, pustules, and erosions on the glans of the penis. Dentures should be cleaned carefully with an effective disinfectant, such as chlorhexidine.
They often coalesce to form large patches of various colors (versicolor) ranging from white to tan [see Figure 7].
Biopsies of these lesions show organisms around and within the hair follicle, with accompanying neutrophilic inflammation.
Topical azoles, such as ke-toconazole, miconazole, and clotrimazole, are also effective, but the expense of these drugs makes their use impractical except for small or isolated lesions. Exposed areas are most commonly involved, typically the extremities or the areas around the mouth and nose.
In this disorder, known as bullous impetigo, superficial, fragile, and flaccid vesiculopustules form and then rupture, with the exu- date drying into a thin, brown, varnishlike crust. For treatment of sparse, nonbullous lesions, topical mupirocin ointment applied three times daily for 7 days is as effective as oral antimicrobials. Louis University School of Medicine, he completed his Internal Medicine residency and Rheumatology fellowship at the University of California, Irvine. An unaided visual exam using the naked eye is no longer appropriate to an emerging illness involving so many artifacts.
In chronic atrophic candidiasis, contamination of dentures with Candida causes painful, red, and sometimes edematous lesions with a shiny, atrophic epithelium and well-demarcated borders where the dentures contact the mucous membranes.
For patients who have difficulty applying a topical agent because of physical disabilities or other factors, oral ketoconazole or fluconazole in a single 400 mg dose is an effective alternative. Impetigo is usually a disease of young children and is more frequent in hot, humid climates than in temperate ones. Sometimes, the vesiculopustules are not apparent, and erythematous erosions are the only evident disturbance. Ecthyma commonly occurs in patients with poor hygiene or malnutrition or patients who have had skin trauma.
Instead of penicillin, many clinicians prescribe an anti-staphylococcal agent—either a first-generation cephalosporin or a penicillinase-resistant penicillin—because of concerns about S. No one trusts the judgment of a mechanic who refuses to look under the hood of a car. A more thorough approach needs to be taken using equipment such as dermascopes and microscopes, as this is the only way to identify the microscopic fibers that are the defining symptom of Morgellons, and which separates it from all other diseases. This infection most commonly involves the upper trunk, but the arms, axillae, abdomen, and groin may also be affected. Because these yeasts form part of the normal cutaneous flora, growth of the organism on cultures from scrapings of the skin surface is not very helpful diagnostically. The lesions, which are often multiple and are most common on the lower extremities, begin as vesicles that rupture, creating circular, erythematous lesions with adherent crusts. Edema from any cause, including venous insufficiency, hypoalbuminemia, and lymphatic damage, is a predisposing factor. Additional circumstances in which organisms other than streptococci are likely to be responsible for cases of cellulitis include immunodeficiency, penetrating trauma, immersion injuries in freshwater or saltwater, granulocytopenia, and animal bites or scratches.
Using a dermascope can also prevent misdiagnosing diseases that have some similar symptoms, such as Lyme (although some patients may have both).
Some patients with these predisposing factors have angular cheilitis (perleche), characterized by erythema and fissuring of the corners of the mouth. Culture of the yeast from the pus of folliculitis, however, is definitive, but it requires special media, such as Sabouraud agar with olive oil, to provide the necessary lipids for growth. Beneath the scabs, which may spontaneously slough, are ulcers that leave a scar when healing occurs. Infection commonly occurs on skin that has been permanently damaged by burns, trauma, radiotherapy, or surgery.
Patients often get worse shortly after therapy, with further extension of the cellulitis, higher fever, greater toxicity, and increased white blood cell counts, presumably because rapid killing of the organisms releases potent enzymes, such as strep-tokinase and hyaluronidase, that cause many of the clinical features. Microscopic enlargement needs to be at minimum 60x-200x in order to see the fibers clearly. As evidenced in the photos in our galleries, there is something clearly wrong with the skin of Morgellons sufferers.
Other contributing conditions are maceration from excessive salivation or licking, poorly fitting dentures, and a larger fold from diminished alveolar ridge height. For extensive lesions, these antibiotics are preferred to topical therapy, and they are the treatment of choice for bullous impetigo. Treatment should be with an oral antistaphylococcal agent, such as dicloxacillin or cephalexin. Most doctors currently believe that the fibers patients come to see them about are 'textile' in nature. If you take a look at the galleries of fibers in urine, stool, blood, skin, and hair, you will see that these filaments are invading the body systemically and cannot possibly be textile.
Click here to view other particles in the skin as well as other unidentified artifacts.?• ?Randy Wymore, Director of OSU-CHS Center for the Investigation of Morgellons Disease and Associate Professor of Pharmacology and Physiology had the fibers analyzed at the Tulsa police department's forensic laboratory. The Morgellons particles didn't match any of the 800 fibers on their database, nor the 85,000 known organic compounds.
For those insisting the CDC study of Morgellons laid down the law for definitive answers about Morgellons, I'd suggest you listen to Doctor Randy Wymore speak more in depth about the study and its inherent flaws and bias. Acupuncture , heat , electrical stimulation channels of energy , body's internal organs, correct imbalances digestion, absorption, and energy production activities, circulation of their energy .
Many clinicians refer to this condition as delusional parasitosis or delusional infestation and consider the filaments to be introduced textile fibers.
In contrast, recent studies indicate that MD is a true somatic illness associated with tickborne infection, that the filaments are keratin and collagen in composition and that they result from proliferation and activation of keratinocytes and fibroblasts in the skin. Previously, spirochetes have been detected in the dermatological specimens from four MD patients, thus providing evidence of an infectious process. (2013) has revealed the presence of Borrelia in Morgellons lesions in everyone tested.


It is the subject of considerable debate within the medical profession and is often labeled as delusions of parasitosis or dermatitis artefacta. This view is challenged by recent published scientific data put forward between 2011-2013 identifying the filaments found in MD as keratin and collagen based and furthermore associated with spirochetal infection. The novel model of the dermopathy put forward by those authors is further described and, in particular, presented as a dermal manifestation of the multi-system disease complex borreliosis otherwise called Lyme disease.
The requirements for a diagnosis of delusional disorder from a psychiatric perspective are clarified and the psychological or psychiatric co-morbidity that can be found with MD cases is presented. Management of the multisytem disease complex is discussed both in general and from a dermatological perspective.
Despite evidence demonstrating that an infectious process is involved and that lesions are not self-inflicted, many medical practitioners continue to claim that this illness is delusional. We present relevant clinical observations combined with chemical and light microscopic studies of material collected from three patients with Morgellons disease.
Our study demonstrates that Morgellons disease is not delusional and that skin lesions with unusual fibers are not self-inflicted or psychogenic.
Proliferative stages of bovine digital dermatitis demonstrate keratin filament formation in skin above the hooves in affected animals.
The multifactorial etiology of digital dermatitis is not well understood, but spirochetes and other coinfecting microorganisms have been implicated in the pathogenesis of this veterinary illness. Morgellons disease is an emerging human dermopathy characterized by the presence of filamentous fibers of undetermined composition, both in lesions and subdermally. While the etiology of Morgellons disease is unknown, there is serological and clinical evidence linking this phenomenon to Lyme borreliosis and coinfecting tick-borne agents. Although the microscopy of Morgellons filaments has been described in the medical literature, the structure and pathogenesis of these fibers is poorly understood.
In contrast, most microscopy of digital dermatitis has focused on associated pathogens and histology rather than the morphology of late-stage filamentous fibers. The most effective antibiotics have included tetracyclines (doxycycline, minocycline), macrolides (Zithromax, Biaxin), Septra DS, and quinolone drugs (Cipro, Levaquin, Avelox, and Factive). Often the skin lesions and associated symptoms resolve once the above antibiotics are used in a comprehensive treatment plan" ??Horowitz, Richard (2013-11-12). Why Can't I Get Better?: Solving the Mystery of Lyme and Chronic Disease (Kindle Locations 2662-2666). Specialization, Materia Medica, internal medicine, diagnosis , examine, refer ,investigations .health shops, pharmacies, prescription, complex remedies, labelled ,individualized ,mental, migraines, asthma, eczema, hay fever, glandular fever, chronic fatigue syndrome. Still, 100% of the patients, that were felt to genuinely haveMorgellons Disease, have large microscopic-to small macroscopic fibersvisible under their outer layer of skin. These fibers are not associated withscabs or open lesions, nor are they under scarred tissue. Free-radical, toxin-, inflammatory, degenerative diseases, Nutrients, minerals, enzymes, vitamins, amino acids. The idea thatMorgellons fibers are mere fuzz and lint, simply sticking to the lesions andscabs, is not possible based on the observations that were just described.These fibers are under "normal-appearing" areas of skin.
The photographs indicate that the particles are very active, and are growing "tendrils," fibers," "ruffles," protrusions," and are creating connections between themselves. These connecting "fibers" appear to be creating translucent "crystal clear" objects that give the appearance of "crystal fibers."                                                                              —Fungihomeworld. 5mar2010                 ?                   ?        Although Morgellons has no known "cure" at this time, we believe recognizing and identifying the disease gives a patient something to go on so they can start becoming proactive with their health. Often these visits are out-of-pocket expenditures as patients escalate up the food chain to specialists not within their health plans, bankrupting themselves unnecessarily. Having a diagnosis will allow sufferers to make informed decisions about their next healthcare choices. There is no silver bullet when it comes to Morgellons. A holistic approach working with diet and pH, plus supplements to rebuild the body's systems is one avenue towards proactive change and symptom reduction. Fibers and particles in various states of emergence from the skin. This mass exodus happens after coconut oil has been applied to my skin and usually takes 3-4 hours after application to see these kind of results.
Fibers under the skin can clearly be seen in these photos as well as in urine and stool, plus the lab doing DNA testing has noted that they are seeing fibers in stool samples. I truly do not understand how this could be misconstrued as a mental illness—and anti-depressants and anti-psychotics are not a fix.
This community is looking for a way to repair the health of the body.  A quick Google search revealed that only a quarter of the dermatologists in the United States even own a dermascope.
Plus, a scope that only enlarges 10 or 15 times, is not going to be appropriate for diagnosis of Morgellons.
The fact that our doctors are supposedly experts and authorities, and considered the last word in the eyes of most of the public, yet are clearly not equipped to diagnose a new disease such as Morgellons, reflects poorly on the medical profession.?You have power to move the Morgellons cause along, if only you will participate. We need your support and input as health "authorities" to bring credibility to the phenomenon? happening. If you think there are not enough people sick with this disease to warrant your time, think again and heed a call from those of us who are looking at the skin of our friends and families and are finding fibers in them. As of yet, this "healthy" population is asymptomatic, but we believe they are carrying the seeds of this disease in their systems.
Imagine a world in which every patient going for a physical with their GP or dermatologist had 3 minutes devoted to examination of the skin with a scope—and what kind of data could be gathered from this! That's the kind of world I would like to live in. One final note before I step off my soapbox is to reinforce the oath you took on becoming a doctor. DOP in patients records have led to loss of their legal rights, hospitalization against their will and in some cases children being removed from their parents. Also, be cautious as to how you phase things as in "I'm having crawling sensations on my skin." instead of "I'm infested with bugs!" I'd refrain from using the term "Morgellons" until you sense you can trust your practitioner.
Click the links to see extensive documentation of Morgellons fibers in Urine, Stool, Blood, Skin, Hair Above: Fiber is magnified 200x. At the time this was taken, my urine pH was very low (acidic) and I was having a hard time bringing it up to alkaline for two days.
When I checked my skin I saw many more fibers then usual and many were still under the skin. I asked myself what would cause such fiber growth, and made the connection to an acidic system. It it my personal wish to see physicians actually begin to use magnification so that they may understand the phenomena their patients are experiencing. Photo submitted courtesy of oddiyfuqoro NA will? Above: This particle is one of the classic forms I find being expelled from the body.
The Morgellons particles didn't match any of the 800 fibers on their database, nor the 85,000 known organic compounds. For those insisting the CDC study of Morgellons laid down the law for definitive answers about Morgellons, I'd suggest you listen to Doctor Randy Wymore speak more in depth about the study and its inherent flaws and bias. Often the skin lesions and associated symptoms resolve once the above antibiotics are used in a comprehensive treatment plan" ??Horowitz, Richard (2013-11-12).



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