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03.10.2014, admin
The skin is the body’s largest organ, and protects all of the internal organs from external factors such as the sun, sharp objects, and of course infections. Most bacterial skin infections are caused by a single family of bacteria, the Staphylococcus, but infections vary in name, symptoms, and severity quite significantly. The staphylococcus aureus bacteria is carried by roughly 20% of the population, and as many as 40%, so infection is certainly not a definite result of being a carrier, most often the bacteria doesn’t become pathogenic until the skin is cut or grazed, and even then only in very few cases which seems to be more dependent on a weakened immune system. Groups most at risk from contracting a bacterial skin infection are children, the elderly, and people with already suppressed immune systems or diabetes, although in a small number of cases they can occur in healthy individuals. The most common bacterial skin infections noted in US hospitals, a trend that is similar in other developed nations, are cellulitis, impetigo, and folliculitis.
Impetigo, one of the most common bacterial skin infections is most often seen in young children, usually between the ages of two and five.
Cellulitis is a deeper more serious bacterial skin infection affecting the deeper dermis and subcutaneous tissues, which more usually affects the face arms and legs, and almost always occurs due to a break in the skin that leads to infection. Folliculitis is a bacterial infection of the skin at the hair follicles, with swelling, and pustules surrounding the hair, and are often quite painful. Staphylococcal skin infections may rarely be complicated by widespread rashes including toxic shock syndrome and staphylococcal scalded skin syndrome.
Streptococcal skin infections may rarely be complicated by widespread rashes including scarlet fever and toxic shock-like syndrome.
If staphylococcal infections recur, treat carrier sites (nostrils) and identify underlying skin diseases such as eczema, scabies and head lice. Erysipelas, cellulitis and many cases of necrotising fasciitis are most frequently caused by Streptococcus pyogenes, less often by Staphylococcus aureus, enterobacteriae and anaerobes. Normal skin flora includes harmless staphylococci, micrococci, diphtheroids and yeasts with aerobes on the surface and anaerobes within hair follicles. Although Staphylococcus aureus is present in the nostrils of about 30% adults, it becomes invasive resulting in infection if there is an injury or other breach of the skin surface such as dermatitis, scabies or head lice.
Follicular infections: bacterial folliculitis (surface pustules) and furunculosis (deeper boils). Localised staphylococcal infections may be managed using meticulous wound care, antiseptics and topical antibiotics. Toxic shock syndrome (shock in association with fever, rash, swollen peeling hands and feet). Impetigo may be caused by Streptococcus pyogenes, when it is characterised by crusts and ulceration.
Invasive streptococci result in erysipelas (superficial lymphatic invasion) and cellulitis (involvement of subcutis). Necrotising fasciitis is a surgical emergency, most often due to Streptococcus pyogenes and less frequently be due to Staphylococcus aureus, enterobacteriae and other organisms.
Streptococcal toxic shock-like syndrome (shock in association with fever, rash, swollen peeling hands and feet).
Cutaneous Group A streptococcal infections (Streptococcus pyogenes) can result in post-streptococcal glomerulonephritis and should be treated with systemic antibiotics such as penicillin.
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In fact the skin does a remarkable job of protecting the body from infection, most enter the body thru the nose, mouth, ears, eyes, anus, or groin. Milder infections may only exhibit small reddish bumps, compared with more serious infections that may attack the entire skin right thru to it’s deepest levels. Within schools and play centers it is important that mats and other surfaces that children are in contact with are kept clean using hospital grade disinfectant to minimize chances of bacterial transfer. Prevention of contracting skin infections relies to a great extent on cleanliness and avoiding breaks in the skin, and when a break occurs, immediately washing the affected area with soap and water. It manifests in groups of small red bumps which eventually open, becoming crusty, and have a honey colored appearance. The skin around the break will swell and become tender and painful, often showing outward signs of blistering. Two types of folliculitis exist, a superficial infection, and a deeper infection that can extend into the tissue surrounding the follicle resulting in hard painful nodules.
These bacteria provide some protection against pathogens by competitive interference and production of antibiotics. These may not prevent later streptococcal complications due to immunological reactions to superantigens, including the skin diseases hypersensitivity vasculitis and guttate psoriasis. Because these properties are shared by certain bacteria (rickettsiae, chlamydiae), viruses are now characterized by their simple organization and their unique mode of replication. Tell a friend about us, add a link to this page, or visit the webmaster's page for free fun content.
Yet, bacterial skin infections do occur, and can be both painful and disfiguring if not treated early. They are highly contagious, both at play group, and at home, and often children will become reinfected after showing signs of recovery.
However, there is increasing prevalence of methicillin-resistant strains of staphylococci so antibiotics should not be prescribed for trivial reasons.
Streptococcal wound infections are painful with surrounding well-demarcated erythema and oedema.
Fever and chills are other symptoms, and patients with fever generally require urgent medical attention. Physicians might consider that Morgellons and Bartonella are in some way intertwined.??Morgellons sufferers report an array of symptoms happening just under the skin, such as the emergence of fibers and filament clusters, specks, particles and sand shaped granules, yet physicians are unaware of this phenomena because they are not using enlargement devices. Group A streptococci are uniformly sensitive to penicillin and improvement is usually seen within hours of treatment.
These are the proteins that form the capsid (protein coat); there may also be a few enzymes or regulatory proteins involved in assembling the capsid around newly synthesized viral nucleic acid, in controlling the biochemical mechanisms of the host cell, and in lysing the host cell when new virions have been assembled.
An unaided visual exam using the naked eye is no longer appropriate to an emerging illness involving so many artifacts. Erysipelas and cellulitis may recur in the same site and prolonged courses of penicillin are sometimes required prophylactically.
No one trusts the judgment of a mechanic who refuses to look under the hood of a car. A more thorough approach needs to be taken using equipment such as dermascopes and microscopes, as this is the only way to identify the microscopic fibers that are the defining symptom of Morgellons, and which separates it from all other diseases.


For certain viruses the RNA is replicated by a viral enzyme (transcriptase) contained in the virion, or produced by the host cell using the viral RNA as a messenger. Using a dermascope can also prevent misdiagnosing diseases that have some similar symptoms, such as Lyme (although some patients may have both). In other viruses a reverse transcriptase contained in the virion transcribes the genetic message on the viral RNA into DNA, which is then replicated by the host cell. Microscopic enlargement needs to be at minimum 60x-200x in order to see the fibers clearly. As evidenced in the photos in our galleries, there is something clearly wrong with the skin of Morgellons sufferers. When these proteins assemble to form the capsid, part of the host cell membrane is pinched off to form the envelope of the virion.Some viruses have only a few genes coding for capsid proteins. Most doctors currently believe that the fibers patients come to see them about are 'textile' in nature.
If you take a look at the galleries of fibers in urine, stool, blood, skin, and hair, you will see that these filaments are invading the body systemically and cannot possibly be textile. Some envelope proteins facilitate viral entry into the cell, and others have directly pathogenic effects.Some viruses do not produce rapid lysis of host cells, but rather remain latent for long periods in the host before the appearance of clinical symptoms.
Click here to view other particles in the skin as well as other unidentified artifacts.?• ?Randy Wymore, Director of OSU-CHS Center for the Investigation of Morgellons Disease and Associate Professor of Pharmacology and Physiology had the fibers analyzed at the Tulsa police department's forensic laboratory. The Morgellons particles didn't match any of the 800 fibers on their database, nor the 85,000 known organic compounds. In the lentiviruses, it was formerly mistakenly believed that virus was inactive during this period.
For those insisting the CDC study of Morgellons laid down the law for definitive answers about Morgellons, I'd suggest you listen to Doctor Randy Wymore speak more in depth about the study and its inherent flaws and bias. The true situation is that lentiviruses are rapidly replicating and spawning dozens of quasi-species until a particularly effective one overruns the ability of the host's immune system to defeat it.
Many clinicians refer to this condition as delusional parasitosis or delusional infestation and consider the filaments to be introduced textile fibers.
For many years all forms of latency were thought to be identical, but now it has been discovered that there are different types with basic and important distinctions.In viral latency, most of the host cells may be protected from infection by immune mechanisms involving antibodies to the viral particles or interferon.
In contrast, recent studies indicate that MD is a true somatic illness associated with tickborne infection, that the filaments are keratin and collagen in composition and that they result from proliferation and activation of keratinocytes and fibroblasts in the skin. Previously, spirochetes have been detected in the dermatological specimens from four MD patients, thus providing evidence of an infectious process. (2013) has revealed the presence of Borrelia in Morgellons lesions in everyone tested. Cytotoxic lymphocytes may also act as antigen-presenting cells to better coordinate the immune response. Containment of virus in mucosal tissues is far more complex, involving follicular dendritic cells and Langerhans cells.Some enveloped RNA viruses can be produced in infected cells that continue growing and dividing without being killed.
It is the subject of considerable debate within the medical profession and is often labeled as delusions of parasitosis or dermatitis artefacta.
This view is challenged by recent published scientific data put forward between 2011-2013 identifying the filaments found in MD as keratin and collagen based and furthermore associated with spirochetal infection. It is also possible for the DNA of some viruses to be incorporated into the host cell DNA, producing a carrier state.
The novel model of the dermopathy put forward by those authors is further described and, in particular, presented as a dermal manifestation of the multi-system disease complex borreliosis otherwise called Lyme disease.
These are almost always retroviruses, which are called proviruses before and after integration of viral DNA into the host genome.Few viruses produce toxins, although viral infections of bacteria can cause previously innocuous bacteria to become much more pathogenic and toxic. Other viral proteins, such as some of the human immunodeficiency virus, appear to be actively toxic, but those are the exception, not the rule.However, viruses are highly antigenic. The requirements for a diagnosis of delusional disorder from a psychiatric perspective are clarified and the psychological or psychiatric co-morbidity that can be found with MD cases is presented. Management of the multisytem disease complex is discussed both in general and from a dermatological perspective. In patients with hemolytic anemia or sickle cell disease it causes aplastic crisis; it can also cause acute arthritis.
Persistent infection in immunocompromised patients can lead to chronic bone marrow failure.
This has the most rapid onset of the hepatitis viruses affecting humans; transmission is easier than for the hepatitis B and C viruses, but infection generally does not persist. Despite evidence demonstrating that an infectious process is involved and that lesions are not self-inflicted, many medical practitioners continue to claim that this illness is delusional.
Progression of HIV-2 infection to AIDS is generally slower and less extreme than that of HIV-1.
We present relevant clinical observations combined with chemical and light microscopic studies of material collected from three patients with Morgellons disease.
The virus is believed to induce permanent infection and has a propensity toward a subset of T lymphocytes called the CD4 cells. Our study demonstrates that Morgellons disease is not delusional and that skin lesions with unusual fibers are not self-inflicted or psychogenic. The infected cells become dysfunctional and eventually the host's immune system is overwhelmed or exhausted; death ensues, usually as a result of infection.
Proliferative stages of bovine digital dermatitis demonstrate keratin filament formation in skin above the hooves in affected animals. The multifactorial etiology of digital dermatitis is not well understood, but spirochetes and other coinfecting microorganisms have been implicated in the pathogenesis of this veterinary illness.
HTLV-1 causes chronic infection and is associated with adult T-cell leukemia and a type of myelopathy. Morgellons disease is an emerging human dermopathy characterized by the presence of filamentous fibers of undetermined composition, both in lesions and subdermally.
While the etiology of Morgellons disease is unknown, there is serological and clinical evidence linking this phenomenon to Lyme borreliosis and coinfecting tick-borne agents.
Serotype A viruses are subject to major antigenic changes (antigenic shifts) as well as minor gradual antigenic changes (antigenic drift) and cause widespread epidemics and pandemics. Although the microscopy of Morgellons filaments has been described in the medical literature, the structure and pathogenesis of these fibers is poorly understood. In contrast, most microscopy of digital dermatitis has focused on associated pathogens and histology rather than the morphology of late-stage filamentous fibers.
RSV can cause a wide variety of respiratory disorders ranging from a mild cold to serious or even fatal disease of the lung in the very young and very old.


It regularly produces an outbreak of infection each winter and virtually disappears in the summer months. The most effective antibiotics have included tetracyclines (doxycycline, minocycline), macrolides (Zithromax, Biaxin), Septra DS, and quinolone drugs (Cipro, Levaquin, Avelox, and Factive). The most severe infections in children are in the very young, especially those who are preterm, immunologically compromised, or suffering from a congenital heart defect or preexisting lung disorder. Often the skin lesions and associated symptoms resolve once the above antibiotics are used in a comprehensive treatment plan" ??Horowitz, Richard (2013-11-12).
Adults at risk for infection include parents and others who are repeatedly exposed to young children, for example, pediatric nurses and day care attendants. Why Can't I Get Better?: Solving the Mystery of Lyme and Chronic Disease (Kindle Locations 2662-2666).
The course of infection tends to be milder in adults than in children and about 15 per cent of affected adults have no symptoms. Still, 100% of the patients, that were felt to genuinely haveMorgellons Disease, have large microscopic-to small macroscopic fibersvisible under their outer layer of skin. These fibers are not associated withscabs or open lesions, nor are they under scarred tissue. The idea thatMorgellons fibers are mere fuzz and lint, simply sticking to the lesions andscabs, is not possible based on the observations that were just described.These fibers are under "normal-appearing" areas of skin. The photographs indicate that the particles are very active, and are growing "tendrils," fibers," "ruffles," protrusions," and are creating connections between themselves. These connecting "fibers" appear to be creating translucent "crystal clear" objects that give the appearance of "crystal fibers."                                                                              —Fungihomeworld.
5mar2010                 ?                   ?        Although Morgellons has no known "cure" at this time, we believe recognizing and identifying the disease gives a patient something to go on so they can start becoming proactive with their health.
Often these visits are out-of-pocket expenditures as patients escalate up the food chain to specialists not within their health plans, bankrupting themselves unnecessarily. Having a diagnosis will allow sufferers to make informed decisions about their next healthcare choices. There is no silver bullet when it comes to Morgellons. A holistic approach working with diet and pH, plus supplements to rebuild the body's systems is one avenue towards proactive change and symptom reduction. Fibers and particles in various states of emergence from the skin. This mass exodus happens after coconut oil has been applied to my skin and usually takes 3-4 hours after application to see these kind of results. Fibers under the skin can clearly be seen in these photos as well as in urine and stool, plus the lab doing DNA testing has noted that they are seeing fibers in stool samples.
I truly do not understand how this could be misconstrued as a mental illness—and anti-depressants and anti-psychotics are not a fix. This community is looking for a way to repair the health of the body.  A quick Google search revealed that only a quarter of the dermatologists in the United States even own a dermascope.
Plus, a scope that only enlarges 10 or 15 times, is not going to be appropriate for diagnosis of Morgellons.
The fact that our doctors are supposedly experts and authorities, and considered the last word in the eyes of most of the public, yet are clearly not equipped to diagnose a new disease such as Morgellons, reflects poorly on the medical profession.?You have power to move the Morgellons cause along, if only you will participate.
We need your support and input as health "authorities" to bring credibility to the phenomenon? happening. If you think there are not enough people sick with this disease to warrant your time, think again and heed a call from those of us who are looking at the skin of our friends and families and are finding fibers in them.
As of yet, this "healthy" population is asymptomatic, but we believe they are carrying the seeds of this disease in their systems. Imagine a world in which every patient going for a physical with their GP or dermatologist had 3 minutes devoted to examination of the skin with a scope—and what kind of data could be gathered from this! That's the kind of world I would like to live in. One final note before I step off my soapbox is to reinforce the oath you took on becoming a doctor. DOP in patients records have led to loss of their legal rights, hospitalization against their will and in some cases children being removed from their parents. Also, be cautious as to how you phase things as in "I'm having crawling sensations on my skin." instead of "I'm infested with bugs!" I'd refrain from using the term "Morgellons" until you sense you can trust your practitioner. Click the links to see extensive documentation of Morgellons fibers in Urine, Stool, Blood, Skin, Hair Above: Fiber is magnified 200x.
At the time this was taken, my urine pH was very low (acidic) and I was having a hard time bringing it up to alkaline for two days.
When I checked my skin I saw many more fibers then usual and many were still under the skin.
I asked myself what would cause such fiber growth, and made the connection to an acidic system.
It it my personal wish to see physicians actually begin to use magnification so that they may understand the phenomena their patients are experiencing. Photo submitted courtesy of oddiyfuqoro NA will? Above: This particle is one of the classic forms I find being expelled from the body. The Morgellons particles didn't match any of the 800 fibers on their database, nor the 85,000 known organic compounds. For those insisting the CDC study of Morgellons laid down the law for definitive answers about Morgellons, I'd suggest you listen to Doctor Randy Wymore speak more in depth about the study and its inherent flaws and bias. It is composed of eight segments, including a nucleoprotein (which folds its nucleic acids and helps them to be transcribed), a hemagglutinin (which helps it to enter cells), a neuraminidase (which helps it to bud out of infected cells), several transcriptases (which make copies of the viral RNA), a matrix protein (which supports the outer membrane), and several nonstructural proteins. The virus mutates frequently and causes annual disease outbreaks, some of which (pandemics) affect millions of people.
It can be treated with antiviral drugs and prevented with annual vaccinations that target its frequently evolving antigens. Often the skin lesions and associated symptoms resolve once the above antibiotics are used in a comprehensive treatment plan" ??Horowitz, Richard (2013-11-12). Viruses suspected of causing tumors in humans include Epstein-Barr virus (associated with Burkitt lymphoma), hepatitis B virus (associated with hepatocellular carcinoma), papilloma virus (associated with carcinoma of the cervix), and human herpesvirus 8 (associated with Kaposi sarcoma).



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