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The pathway for pain and temperature enters the spinal cord, crosses over to the opposite half of the cord almost immediately (actually within one or two spinal cord vertebral segments), ascends to the thalamus on the opposite side, and then moves on to the cerebral cortex.
The pathway for proprioception and stereognosis (also for the perception of vibration) initially remains on the same side of the spinal cord that it enters, crossing over at the junction between the spinal cord and brain stem. Unlike the other sensory pathways, which cross contralaterally, the spinocerebellar primarily remains ipsilateral.
There are three main connections between the cerebellum and the brain stem—the superior, middle and inferior cerebellar peduncles, which connect with the midbrain, pons, and medulla, respectively. The corticospinal pathway synapses in the anterior horn (motor grey matter) of the spinal cord just prior to leaving the cord. For unknown reasons various disease processes affect different portions of the spinal cord.
Tertiary syphilis (tabes dorsalis) includes propioceptive loss and pain (posterior root irritation), particularly affecting the lower extremities. Use the form below to delete this Figure 01c Neurons View Large Image image from our index. Use the form below to delete this The Nervous System Develops From Embryonic Segments But In Adult image from our index. Use the form below to delete this The Arrangement Of Sensory And Motor Nuclei Cranial Nerves Key image from our index.
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Use the form below to delete this Motor Neurons Carry Can Be Divided Into 3 Distinct Pathways image from our index. To discover images and hq pictures, type your search terms into our powerful search engine box or browse our different categories. As a rule though, there is a tendency for sensory fibers to synapse just prior to crossing over (Fig.
In reality many fibers end in the brain stem and never make it as far as the thalamus, but these will not concern us here. The synaptic areas just prior to this crossing are the nucleus cuneatus and nucleus gracilis (Fig. It partly remains uncrossed until it reaches the level of the brain stem, and partly crosses over at lower levels. The conscious pathway, mentioned above, connects with the thalamus and cerebral cortex, enabling you to describe, for instance, the position of your limb. The spinocerebellar pathways enter the cerebellum via the superior and inferior peduncles (Fig. It extends from the motor area of the cerebral cortex down through the brain stem, crossing over at approximately the same level as the medial lemniscus (at the junction between brain stem and spinal cord), (Figs. This is important, for motor neurons above the level of this synapse (connecting the cerebral cortex and anterior horn) are termed upper motor neurons (UMN), whereas those beyond this level (the peripheral nerve neurons) are termed lower motor neurons (LMN).
18D) leading to lower motor neuron involvement, with weakness, atrophy, fasciculations, fibrillations, and hyporeflexia. 18E) experience sensory and lower motor neuron loss because of peripheral nerve involvement. A lesion of the spinothalamic tract will result in loss of pain-temperature sensation contralateral^, below the level of the lesion.
This is why light touch typically is spared in unilateral spinal cord lesions; there are alternate routes to carry the information.
A lesion of the sensory area of the cerebral cortex may result in a contralateral deficit of all the above sensory modalities.


Thus cerebellar lesions tend to produce ipsilateral malfunctioning, whereas cerebral lesions result in contralateral defects. These terms are actually somewhat misleading and probably would better have been phrased as first order motor neurons (UMN) and second order motor neurons (LMN), as either of these categories may have axons that lie in the upper or lower part of the body.
18A) is characterized by a combination of upper and lower motor neuron signs—muscle weakness, atrophy, fibrillations, and fasciculations combined with hyperreflexia. Nucleus gracilis contains proprioceptive information from the lower part of the body, whereas nucleus cuneatus conveys information from the upper levels. The lesion involves both the anterior horns (motor) of the grey matter (causing a lower motor neuron lesion) as well as the corticospinal tracts (resulting in an upper motor neuron lesion).
Physicians might consider that Morgellons and Bartonella are in some way intertwined.??Morgellons sufferers report an array of symptoms happening just under the skin, such as the emergence of fibers and filament clusters, specks, particles and sand shaped granules, yet physicians are unaware of this phenomena because they are not using enlargement devices. Their corresponding spinal cord pathways are termed fasciculus gracilis (graceful, like a ballerina's legs) and fasciculus cuneatus (cunning, being closer to the brain). An unaided visual exam using the naked eye is no longer appropriate to an emerging illness involving so many artifacts. The fasciculus gracilis and fasciculus cuneatus are collectively termed the posterior columns (Fig. No one trusts the judgment of a mechanic who refuses to look under the hood of a car. A more thorough approach needs to be taken using equipment such as dermascopes and microscopes, as this is the only way to identify the microscopic fibers that are the defining symptom of Morgellons, and which separates it from all other diseases. Using a dermascope can also prevent misdiagnosing diseases that have some similar symptoms, such as Lyme (although some patients may have both). However, it is vital to remember the sites of contralateral crossing over of the various pathways and also the location of the pathways within the white matter as seen in cross section (Fig. A lesion of the posterior columns results in a decrease in conscious proprioception and stereognosis (also vibration sense) ipsilaterally below the level of the lesion. Microscopic enlargement needs to be at minimum 60x-200x in order to see the fibers clearly. As evidenced in the photos in our galleries, there is something clearly wrong with the skin of Morgellons sufferers.
Most doctors currently believe that the fibers patients come to see them about are 'textile' in nature.
If you take a look at the galleries of fibers in urine, stool, blood, skin, and hair, you will see that these filaments are invading the body systemically and cannot possibly be textile.
Click here to view other particles in the skin as well as other unidentified artifacts.?• ?Randy Wymore, Director of OSU-CHS Center for the Investigation of Morgellons Disease and Associate Professor of Pharmacology and Physiology had the fibers analyzed at the Tulsa police department's forensic laboratory. The Morgellons particles didn't match any of the 800 fibers on their database, nor the 85,000 known organic compounds. For those insisting the CDC study of Morgellons laid down the law for definitive answers about Morgellons, I'd suggest you listen to Doctor Randy Wymore speak more in depth about the study and its inherent flaws and bias. Many clinicians refer to this condition as delusional parasitosis or delusional infestation and consider the filaments to be introduced textile fibers. In contrast, recent studies indicate that MD is a true somatic illness associated with tickborne infection, that the filaments are keratin and collagen in composition and that they result from proliferation and activation of keratinocytes and fibroblasts in the skin. Previously, spirochetes have been detected in the dermatological specimens from four MD patients, thus providing evidence of an infectious process. (2013) has revealed the presence of Borrelia in Morgellons lesions in everyone tested. It is the subject of considerable debate within the medical profession and is often labeled as delusions of parasitosis or dermatitis artefacta. This view is challenged by recent published scientific data put forward between 2011-2013 identifying the filaments found in MD as keratin and collagen based and furthermore associated with spirochetal infection.
The novel model of the dermopathy put forward by those authors is further described and, in particular, presented as a dermal manifestation of the multi-system disease complex borreliosis otherwise called Lyme disease. The requirements for a diagnosis of delusional disorder from a psychiatric perspective are clarified and the psychological or psychiatric co-morbidity that can be found with MD cases is presented.
Management of the multisytem disease complex is discussed both in general and from a dermatological perspective.
Despite evidence demonstrating that an infectious process is involved and that lesions are not self-inflicted, many medical practitioners continue to claim that this illness is delusional. We present relevant clinical observations combined with chemical and light microscopic studies of material collected from three patients with Morgellons disease.
Our study demonstrates that Morgellons disease is not delusional and that skin lesions with unusual fibers are not self-inflicted or psychogenic. Proliferative stages of bovine digital dermatitis demonstrate keratin filament formation in skin above the hooves in affected animals.
The multifactorial etiology of digital dermatitis is not well understood, but spirochetes and other coinfecting microorganisms have been implicated in the pathogenesis of this veterinary illness. Morgellons disease is an emerging human dermopathy characterized by the presence of filamentous fibers of undetermined composition, both in lesions and subdermally. While the etiology of Morgellons disease is unknown, there is serological and clinical evidence linking this phenomenon to Lyme borreliosis and coinfecting tick-borne agents. Although the microscopy of Morgellons filaments has been described in the medical literature, the structure and pathogenesis of these fibers is poorly understood.
In contrast, most microscopy of digital dermatitis has focused on associated pathogens and histology rather than the morphology of late-stage filamentous fibers.
The most effective antibiotics have included tetracyclines (doxycycline, minocycline), macrolides (Zithromax, Biaxin), Septra DS, and quinolone drugs (Cipro, Levaquin, Avelox, and Factive).


Often the skin lesions and associated symptoms resolve once the above antibiotics are used in a comprehensive treatment plan" ??Horowitz, Richard (2013-11-12). Why Can't I Get Better?: Solving the Mystery of Lyme and Chronic Disease (Kindle Locations 2662-2666). Still, 100% of the patients, that were felt to genuinely haveMorgellons Disease, have large microscopic-to small macroscopic fibersvisible under their outer layer of skin.
These fibers are not associated withscabs or open lesions, nor are they under scarred tissue. The idea thatMorgellons fibers are mere fuzz and lint, simply sticking to the lesions andscabs, is not possible based on the observations that were just described.These fibers are under "normal-appearing" areas of skin.
The photographs indicate that the particles are very active, and are growing "tendrils," fibers," "ruffles," protrusions," and are creating connections between themselves. These connecting "fibers" appear to be creating translucent "crystal clear" objects that give the appearance of "crystal fibers."                                                                              —Fungihomeworld. 5mar2010                 ?                   ?        Although Morgellons has no known "cure" at this time, we believe recognizing and identifying the disease gives a patient something to go on so they can start becoming proactive with their health. Often these visits are out-of-pocket expenditures as patients escalate up the food chain to specialists not within their health plans, bankrupting themselves unnecessarily. Having a diagnosis will allow sufferers to make informed decisions about their next healthcare choices. There is no silver bullet when it comes to Morgellons. A holistic approach working with diet and pH, plus supplements to rebuild the body's systems is one avenue towards proactive change and symptom reduction. Fibers and particles in various states of emergence from the skin. This mass exodus happens after coconut oil has been applied to my skin and usually takes 3-4 hours after application to see these kind of results. Fibers under the skin can clearly be seen in these photos as well as in urine and stool, plus the lab doing DNA testing has noted that they are seeing fibers in stool samples. I truly do not understand how this could be misconstrued as a mental illness—and anti-depressants and anti-psychotics are not a fix. This community is looking for a way to repair the health of the body.  A quick Google search revealed that only a quarter of the dermatologists in the United States even own a dermascope. Plus, a scope that only enlarges 10 or 15 times, is not going to be appropriate for diagnosis of Morgellons. The fact that our doctors are supposedly experts and authorities, and considered the last word in the eyes of most of the public, yet are clearly not equipped to diagnose a new disease such as Morgellons, reflects poorly on the medical profession.?You have power to move the Morgellons cause along, if only you will participate. We need your support and input as health "authorities" to bring credibility to the phenomenon? happening.
If you think there are not enough people sick with this disease to warrant your time, think again and heed a call from those of us who are looking at the skin of our friends and families and are finding fibers in them. As of yet, this "healthy" population is asymptomatic, but we believe they are carrying the seeds of this disease in their systems. Imagine a world in which every patient going for a physical with their GP or dermatologist had 3 minutes devoted to examination of the skin with a scope—and what kind of data could be gathered from this! That's the kind of world I would like to live in. One final note before I step off my soapbox is to reinforce the oath you took on becoming a doctor.
DOP in patients records have led to loss of their legal rights, hospitalization against their will and in some cases children being removed from their parents.
Also, be cautious as to how you phase things as in "I'm having crawling sensations on my skin." instead of "I'm infested with bugs!" I'd refrain from using the term "Morgellons" until you sense you can trust your practitioner.
Click the links to see extensive documentation of Morgellons fibers in Urine, Stool, Blood, Skin, Hair Above: Fiber is magnified 200x. At the time this was taken, my urine pH was very low (acidic) and I was having a hard time bringing it up to alkaline for two days.
When I checked my skin I saw many more fibers then usual and many were still under the skin. I asked myself what would cause such fiber growth, and made the connection to an acidic system.
It it my personal wish to see physicians actually begin to use magnification so that they may understand the phenomena their patients are experiencing. Photo submitted courtesy of oddiyfuqoro NA will? Above: This particle is one of the classic forms I find being expelled from the body.
The Morgellons particles didn't match any of the 800 fibers on their database, nor the 85,000 known organic compounds. For those insisting the CDC study of Morgellons laid down the law for definitive answers about Morgellons, I'd suggest you listen to Doctor Randy Wymore speak more in depth about the study and its inherent flaws and bias. Often the skin lesions and associated symptoms resolve once the above antibiotics are used in a comprehensive treatment plan" ??Horowitz, Richard (2013-11-12).



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