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Magnesium was discovered in 1808 by Sir Humphrey Davy and it is mainly obtained from sea water.
Magnesium is the eighth most abundant element in the Earth crust (the forth in the Earth as a whole) and the ninth in the universe. The elemental Magnesium is very reactive and it is not found naturally on Earth but in compounds. Magnesium is one of the most crucial minerals we are made up of and it is essential that you get your daily dose. 105+ Natural Home Remedies -treat almost any illness with natural ingredients you probably already have in your kitchen. 75+ Natural Beauty Recipes - Get the best hair and skin you've ever had and look younger with these insanely clever natural beauty recipes. DIY Organic Beauty Recipes reveals 50+ step-by-step recipes for making your own all natural, non-toxic, organic beauty products that are fun to make and cheaper than their store bought counterparts. Magnesium is important in more than 300 chemical reactions that keep the body working properly. Magnesium Citrate helps induce a bowel movement and is also highly bioavailable (easily absorbed), so if you suffer from constipation or your levels are low in the body, this is a good choice. Magnesium Lactate is commonly used to treat digestive disorders, but if you have kidney issues or kidney disease, this is not the form you should take.
Magnesium Hydroxide is widely used for the treatment of peptic ulcers and gastric hyperacidity.
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There’s no easy way to check your magnesium levels effectively – magnesium deficiency is often diagnosed by symptoms. But even those symptoms (fatigue, muscle soreness, low testosterone levels, increased inflammation in your heart, and even loss of hearing) only show up in extreme cases, and, as we’ll see, may be tied to other conditions. Statistically speaking, you’re probably deficient – and you had no idea until now(1). Magnesium deficiency is strongly associated with common illnesses like depression(2), migraines(3), impaired glucose tolerance and irregular heart rhythms(4).
Magnesium may be especially important for men, as there is a distinct relationship between magnesium levels and testosterone levels – a 2011 study showed that magnesium supplementation could have a positive impact on men with low testosterone levels(5). On the flip side, there is evidence that getting enough magnesium in your system can help improve your health significantly, possibly helping to safeguard you against type 2 diabetes, cardiovascular disease, and migraine disorders(6).
If that doesn’t convince you to start taking magnesium seriously, who knows what will!
Supplementing your diet with magnesium can be challenging, because in order to be bioavailable it must be bound to another substance.
In addition to this, many magnesium supplements vary in quality, from inexpensive, low-quality products which can have a laxative effect, to higher-quality products with higher levels of absorption in the body. According to the Dieticians Association of Canada, the best dietary sources of magnesium are nuts and seeds, as well as cooked spinach and other leafy greens(7). Magnesium can also be absorbed through the skin – try a nice, relaxing bath with Epsom salts for not only washing away the cares of the day, but restoring your magnesium levels as well!
Magnesium deficiency can occur on its own, but sometimes it is also a symptom of other disorders, such as malabsorption syndromes including Coeliac disease, Crohn’s disease, and steatorrhoea(8). As these may be early indicators of a more serious condition causing your magnesium deficiency.
We will be provided with an authorization token (please note: passwords are not shared with us) and will sync your accounts for you. Conclusion: This study demonstrates a correlation between higher neonatal magnesium levels and decreased risk for long-term abnormal motor exam.
Preterm birth can lead to a wide range of motor and intellectual disabilities affecting up to 35% of survivors (1–4). The neurodevelopmental problems in prematurely born infants are caused by a variety of complex pathophysiological mechanisms (10, 11) with few therapeutic options (12).
Magnesium sulfate administered antenatally has been found to reduce rates of cerebral palsy when given prior to preterm birth (18–20). Our hypothesis was that postnatal magnesium could serve a neuroprotective role in the developing premature brain. To address this hypothesis, we evaluated serum magnesium levels in VLBW infants and compared these with long-term neurodevelopmental outcomes, specifically epilepsy, and abnormal motor exam. This study was approved by the Institutional Review Boards at the University of Utah and Intermountain Healthcare (IH). Data extraction and analysis were performed retrospectively in a cohort of premature infants born at an IH hospital and who were seen in follow-up in the Utah State Department of Health Neonatal Follow-up Program. We queried the EDW using unique identifiers assigned to each of the cohort infants for the period including up to 5 years after birth.
We collected all total serum magnesium levels (n = 223) drawn on a cohort of 75 very-low birth weight (VLBW) infants during their initial, post-birth hospitalization (Table 1).

We examined the correlation between postnatal magnesium levels and neurodevelopmental outcomes in premature infants.
Our study raises the possibility that there may be a broader window of opportunity for magnesium administration in premature infants to help improve neurodevelopmental outcomes.
Animal model data show a neuroprotective role for magnesium against injury in the developing CNS (25–27). Limitations of this study included sample size, retrospective data collection, and missing follow-up for some infants.
Our pilot findings raise the possibility that magnesium levels during a critical developmental time window could affect neurological outcome.
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Save a fortune AND your families health with these super simple homemade cleaning products that work BETTER than store bought! You can get magnesium from your diet, but sometime supplementation is needed if magnesium levels are too low.
While not every doctor has extensive backgrounds in nutrition, they will often be able to help you determine if your magnesium deficiency is a symptom of a larger, more systemic problem. This means that you will not need to remember your user name and password in the future and you will be able to login with the account you choose to sync, with the click of a button.
This page doesn't support Internet Explorer 6, 7 and 8.Please upgrade your browser or activate Google Chrome Frame to improve your experience. Magnesium supplementation has been suggested to have neuroprotective function in the developing brain.
Average total serum magnesium level in the neonate during the period of prematurity was the main independent variable assessed, tested using a Wilcoxon rank-sum test.
Larger studies are needed to evaluate the hypothesis that higher neonatal magnesium levels can improve long-term neurodevelopmental outcomes. Very-low birth weight (VLBW) infants with birth weight less than 1500 g have elevated rates of cerebral palsy, epilepsy, autism, intellectual disability, and behavioral problems (5, 6).
Further, the complications of preterm birth are now also recognized to damage both gray matter and axon tracts and to lead to impaired neurodevelopment (13–17). Magnesium has also been found in small studies to improve neurodevelopmental outcomes in term infants with birth asphyxia (21–24). Magnetic resonance imaging (MRI) data suggest that longer exposure of premature infants to the extrauterine environment results in increasing impairments of CNS connectivity (28). We recorded magnesium levels during the initial hospitalization, for the time period when the infants were still premature (less than 37 weeks gestation). Data were anonymously collected and analyzed with no identifying information, and a waiver of informed consent was obtained. Selected demographic, birth, laboratory, and outcome characteristics of the excluded patients n = 27 (no magnesium levels drawn).
First, for the outcome of the abnormal motor exam, data from the Utah State Department of Health Neonatal Follow-up Program were obtained. Abnormal motor exam, which was defined as the presence of cerebral palsy, hypotonia, or spasticity, was assessed between 20 and 36 months after birth. Strip scatterplots of average magnesium levels (x-axis) and neurodevelopmental outcomes, for abnormal motor exam (A), and epilepsy (B).
While prenatal magnesium administration has been associated with a decreased risk of cerebral palsy (18–20), our study demonstrates that postnatal serum magnesium levels in VLBW infants may be associated with improved neurodevelopmental outcomes.
That is, perhaps magnesium supplementation could also be considered in premature infants and not only in mothers at risk for preterm birth. Further, in term infants with birth asphyxia a potentially protective role has been suggested for magnesium (21–24). While the presence of an abnormal neuro-motor exam can be determined by age 20 months as was done in this study, a more extensive longitudinal study with objective scoring, such as using the Bailey Scale of Infant Development, would provide more reliable outcomes data. So you have to ask yourself, “What am I using it for?” Do I need it to help me with a bodily function or am I just low in Magnesium?
Our objective was to determine whether higher neonatal serum magnesium levels were associated with better long-term neurodevelopmental outcomes in very-low birth weight infants.
While survival rates have improved dramatically for premature infants (7), neurodevelopmental outcomes have not (8). Further, several animal model studies suggest that magnesium could play neuroprotective roles in the developing vertebrate CNS (25–27).
We posited that magnesium could help protect connectivity development of premature infants, and that higher magnesium levels throughout the premature period could be neuroprotective. The objective of our study was to determine whether higher neonatal magnesium levels are associated with improved long-term neurological outcomes. Five patients were lost to follow-up; 27 patients had incomplete or missing data and were not included in analysis (Table 2). Of note, diagnoses of seizures at any time during the NICU hospitalization were excluded, as was the diagnosis of febrile seizures.

A standardized neurological motor exam was performed by a developmental pediatrician or pediatric neurologist when the infant was between 20 and 36 months of age. Wilcoxon Rank-Sum tests were used to compare magnesium levels for the outcomes of seizures and of composite abnormal motor exams. On average infants had their magnesium levels checked three times, but the number of magnesium levels checked ranged from 1 to 17 times (Table 3).
The outcome of epilepsy was assessed for by following patient outcomes for up to 5 years after birth. Specifically, higher serum magnesium levels in preterm infants were associated with lower rates of an abnormal motor exam (spasticity, cerebral palsy, or hypotonia). Use of magnesium sulfate would have to be balanced with concerns for potential adverse side-effects in premature infants (29, 30). There are multiple potential mechanism(s) by which magnesium could exert neuroprotective effects. We excluded 32 infants from our analysis because of incomplete data, including a lack of magnesium levels. 500,000 births each year are premature, while worldwide it is estimated that 12.9 million infants yearly are born before 37 weeks gestation (9, 37, 38). Neurodevelopmental and functional outcomes of extremely low birth weight infants in the National Institute of Child Health and Human Development Neonatal Research Network, 1993-1994.
The effect of chronic or intermittent hypoxia on cognition in childhood: a review of the evidence.
Cognitive, educational, and behavioural outcomes at 7 to 8 years in a national very low birthweight cohort.
Motor development in very preterm and very low-birth-weight children from birth to adolescence: a meta-analysis. Despite efforts to reduce preterm birth, the rate has remained relatively stable over the last few decades and was 11.7% in the United States in 2011 (9). Serum magnesium levels were those drawn during the initial hospitalization up through the end of the premature period; defined as less than 37 weeks gestation. We also performed logistic regression analyses that included birth weight and magnesium level. Another issue requiring further study will be research on the mechanism(s) of potential neuroprotection.
The small sample size also led to limitations on performing multivariate regression analyzes (34). This significant burden of prematurity, with its attendant risks for adverse neurodevelopmental outcomes, warrants further investigations into potential neuroprotective roles for magnesium after preterm birth.
Sources of financial support: RGF was funded in part by NIH grant U10 HD053124 from the Eunice Kennedy Shriver National Institute of Child Health and Human Development.
IH is a large, vertically integrated not-for-profit health care system in the Intermountain West encompassing 23 hospitals including the single children’s hospital. Second, for the outcome of epilepsy, we followed the infants for up to 5 years after birth using the EDW.
For multivariate logistic regression analysis we modeled birth weight and magnesium levels by analysis of quartiles. While they showed a trend toward lower magnesium levels associated with increased risk for abnormal motor outcome or for epilepsy, the sample size was underpowered and did not show statistically significant differences (Table 5).
Another bias could arise because timing of magnesium level blood draws were not evenly distributed in the different infants; and were not distributed evenly across the hospitalization.
EASC was funded in part by NIH grant K23 HD061910 from the Eunice Kennedy Shriver National Institute of Child Health and Human Development. Antenatal, perinatal, and follow-up data were extracted for each patient in the cohort from the Enterprise Data Warehouse (EDW) maintained by IH. We defined epilepsy as any encounter that had a record of the patient having had a seizure and for which the patient was placed on an anti-epileptic drug.
JLB was funded in part by NIH grants K08 DA024753 and DP2 MH100008, from the March of Dimes Foundation, and from the Primary Children’s Medical Center Foundation. The outcome of epilepsy excluded seizures that occurred solely during the NICU hospitalization; and febrile seizures.
Pharmacokinetics and pharmacodynamics of serum magnesium can be affected by both endogenous and iatrogenic factors, including medications and calcium metabolism.
In fact, normative premature infant magnesium levels, and effects of maternal magnesium administration on neonatal levels, are under active study (35, 36), and are important subjects for future research.

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