Asymmetric hearing loss definition legal,ringing in one ear when i yawn quotes,remedies ringing ears 900 - 2016 Feature

Author: admin, 14.09.2015. Category: Cure Ringing Ears

Classic stigmata of congenital syphilis SNHL, interstitial keratitis, Hutchinson teeth (notched incisors), mulberry molars, Clutton joints (bilateral painless knee effusions), nasal septal perforation & saddle deformity& frontal bossing. Case reports of dizziness in and around high-strength (?3T) magnets have prompted investigations into the effects of high-strength magnetic fields on human balance and cognitive function.
The best explanation for these effects of magnetic vestibular stimulation (MVS) is that they are due to a static magneto-hydrodynamic (MHD) force, called the Lorentz force, which occurs in a magnetic field due to the presence of normal ionic currents into hair cells within the ion-rich endolymph of the labyrinth.
Skeletal findings osteochondritis& periostitis of long bones SNHL, interstitial keratitis, Hutchinson teeth (notched incisors), mulberry molars, Clutton joints (bilateral painless knee effusions), nasal septal perforation & saddle deformity& frontal bossing. The influence of strong magnetic fields on vestibular function in rats and mice has been identified by circling behavior after magnetic field exposure, an effect that does not occur after prior labyrinthectomy (1, 2).


The MHD force produces a pressure in the endolymph that is sensed by the cupula of the lateral semicircular canal (SCC), producing a horizontal nystagmus (3).
Quantitative analysis of this behavior suggests that resting utricular hair cell current is primarily responsible for generating the MHD force (4, 9).
Because the utricle is close to the opening of the lateral SCC, the pressure from the Lorentz force it generates can deflect the cupula of the lateral SCC. This magnetic field-induced nystagmus requires only the presence of a static magnetic field; it is not a result of image acquisition.
Individuals with unilateral vestibular hypofunction (UVH) have asymmetric remaining vestibular function and should have a similar MVS response to those with intact function, but proportional to the residual function of their remaining utricle and SCCs.


The goal of the present study was to investigate MVS in humans with UVH to explore further the mechanisms involved in MVS and suggest a potential clinical use for MVS in evaluating the function of individual structures within the labyrinth.



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