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16.03.2015
The Journal of the American Medical Association published an important study today further assessing the “calorie is a calorie” dictum of the dieting world.
They also assessed leptin, thyroid stimulating hormone (TSH), triiodothyronine (T3), urinary cortisol), insulin sensitivity high-density lipoprotein (HDL) cholesterol, total cholesterol, triglycerides, plasminogen activator inhibitor 1 activity, C-reactive protein (CRP), and ratings of hunger and well-being. Total energy expenditure revealed similar results, decreasing by 423 calories per day for the low-fat diet, 297 for the low-GI diet, and only 97 calories per day for the low-carb diet.
Cortisol: a glucocorticoid hormone that counteracts insulin and the body uses to raise blood sugar when it is low via stimulation of gluconeogenesis (conversion of fat to glucose) predictably rose in the low-carbers. Serum TSH and T3: these thyroid values were the lowest for the low-carb diet, which is expected and has been shown previously.
Serum HDL: “good” cholesterol was significantly higher in the low-carbohydrate diet versus the others. Plasminogen activator inhibitor 1: it has been found to be increased in cancer and even predicts for cancer metastases16, obesity, inflammation, and the metabolic syndrome17-19, and was reduced in the low-carb group.
CRP: C-reactive protein is a marker of inflammation and has been correlated with poorer outcomes in cancer, stroke, depression, and cardiovascular disease20,21 and was increased in the low-carb group.
No study is perfect, and as is often the case in dietary studies, this one had its fair share of issues.  The study was short term, with each diet consumed for only 4 weeks. It is well established that weight loss results in a decrease in resting energy expenditure, i.e. Keep eating like a caveman, keep curbing those carbs, and keep watching the studies pour in to support this lifestyle.
In your point #8, you state that CRP is increased with the low-carb diet, however, in your discussion you talk about CRP increasing with the low-fat diet. I do, though more so on cancer and radiation, though I have 6 papers either under review or finishing up currently, so waiting on those. As for the CRP, so the long term studies (see the inflammation article I referred and articles by Volek and Forsyth, among others) and the long-term data for CRP, RBP4 (retinoid binding protein-4) and several other inflammatory factors paints quite a different picture. A poster over at lowcarb.ca dove into the study data and posted this analysis of the CRP data. The whole tone of the article sounds apologetic, and I had to read it twice before I realized it was really pro-LC. And yes, hopefully this opens the door to future studies and further supports the potential empowerment a low-carb diet can give to those trying to lose weight. I was wondering if you have any comment on the fact that although the JAMA article showed a difference in energy expenditure, it did not show a difference in weight. I would love to see a comparison of body-fat percentage changes between the diets, which was measured at baseline but conveniently left out. This was only a preliminary communication, so hopefully we can get some more data soon, but clearly the human body is more than just a calorie in and out metal box.
My personal believe (based on the numerous studies I have conducted using my own body as test subject) is that, regardless of the fat loss in isocaloric diets, LC is superior because it makes following a calorie restricted regimen so much easier. Sorry to bombard you with questions but I’m really not sure where to go with this one.
During the run-in phase of the study, the participants lost an average of around 30 pounds, which corresponded to 13.6% of their baseline body weight.


Basically, these results show us that weight loss on a low-carb diet leaves the dieter with a body that continues to burn more calories than other diets. This plagues many dietary studies, but due to the inherent cost of these clinical trials, we often encounter such issues.
Once again, I will defer to the long-term data. Conversely, all markers of metabolic syndrome were improved in the low-carb diet, which is consistent with the ever-expanding literature22-28.
But in the results table, the overall p for CRP was 0.13, nowhere near statistical significance. In other words, a low-carb diet leaves the body more metabolically demanding than the other diets – i.e.
This is also why telling a patient to markedly reduce their calories while increasing their activity is so difficult to adhere to32,33, rarely works, and merely sets them up for emotional and physical failure.
Fasting insulin and outcome in early-stage breast cancer: results of a prospective cohort study. Serum insulin-like growth factor-binding protein-2 (IGFBP-2) is increased and IGFBP-3 is decreased in patients with prostate cancer: correlation with serum prostate-specific antigen. Relation of serum total cholesterol and triglyceride levels to the amount and extent of coronary arterial narrowing by atherosclerotic plaque in coronary heart disease. Elevated levels of plasma triglycerides are associated with histologically defined premenopausal breast cancer risk. Plasma triglyceride level is a risk factor for cardiovascular disease independent of high-density lipoprotein cholesterol level: a meta-analysis of population-based prospective studies.
Abdominal obesity is associated with an impaired fibrinolytic activity and elevated plasminogen activator inhibitor-1.
Among inflammation and coagulation markers, PAI-1 is a true component of the metabolic syndrome. Relation of C-reactive protein to stroke, cognitive disorders, and depression in the general population: systematic review and meta-analysis. Elevated Biomarkers of Inflammation Are Associated With Reduced Survival Among Breast Cancer Patients. Limited Effect of Dietary Saturated Fat on Plasma Saturated Fat in the Context of a Low Carbohydrate Diet. Comparison of Low Fat and Low Carbohydrate Diets on Circulating Fatty Acid Composition and Markers of Inflammation. Carbohydrate Restriction has a More Favorable Impact on the Metabolic Syndrome than a Low Fat Diet. A randomized trial comparing a very low carbohydrate diet and a calorie-restricted low fat diet on body weight and cardiovascular risk factors in healthy women. Short-term weight loss and hepatic triglyceride reduction: evidence of a metabolic advantage with dietary carbohydrate restriction.
Variables predictive of adherence to diet and physical activity recommendations in the treatment of obesity and overweight, in a group of Spanish subjects. Adherence to medication, diet, and activity recommendations: from assessment to maintenance. The CRP concern is totally made up, and the urinary cortisol excretion – who knows what that means, if it means anything.


This also is why telling a patient to eat less and exercise more (especially when based on a low-fat diet) is not a dietary strategy, but rather torture. Quantitative analysis of 2,037 five mm segments of 160 major epicardial coronary arteries in 40 necropsy patients. Nutrition in clinical practice : official publication of the American Society for Parenteral and Enteral Nutrition. But, anyway, the LC diet did not cause CRP to increase as was said in these stupid reports.
These guys like Ludwig and Bray will have to retire or die before any real progress will be made in sensible dietary advice.
You will want to focus on eating complex and fibrous carbs and try to limit the simple carbs.When you eat carbs, your body creates insulin. There’s nothing complex about it, you just have to follow a few simple rules.TO MINIMIZE FAT STORAGE FOLLOW THESE RULES 1) Eat complex carbohydrates Complex carbohydrates are found in whole foods like brown rice, potatoes, whole grain cereal and oatmeal.
Complex carbohydrates should make up the bulk of your daily calorie intake because they form muscle glycogen, the long lasting fuel that your body needs to train hard.
They also help keep your blood sugar levels constant, this reduces fat storage and fatigue and promotes the release of insulin. 2) Eat carbohydrates directly after training When you train hard you reduce your blood sugar level considerably. Eating carbohydrates straight after a training session provides your body with an insulin spike. If you do not get the right nutrients after training it’s possible that your body could enter a catabolic (muscle breakdown) state.
3) Eat small amounts of carbohydrates more often  Eating smaller servings of carbohydrates more often helps keep a steady flow of insulin into the body. If you eat large amounts of carbohydrates in one sitting your body is much more likely to store them as fat.
Eating to much is one sitting is unnecessary; your body doesn’t need that much nutrients at one time. 4) Eat high fiber carbohydrates This goes hand-in-hand with point number 1 because most sources of complex carbohydrates are rich sources of fiber.
Fiber helps to build muscle by making muscle tissue absorb amino acids faster and more efficiently.
5) Avoid fruits This may sound insane because we all know that fruit is high in vitamins, low in calories and very good for general health and wellbeing. 6) Have carbohydrates and protein in the same meal When you mix protein and carbohydrates together in the same meal you minimize the chance of the carbohydrates being stored as fat. I get irritable and tired when I miss out on carbs- I need them daily!Reply Leave a Reply Cancel replyYour email address will not be published.



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