Are very low carb diets safe,healthy weight loss food tumblr blogs,low carbohydrate vegan diet recipes nz - PDF 2016

28.10.2014
It’s been thought that very-low-carb-high-fat ketogenic diets followed over the long-term will mess up with your hormones. Let’s see some possible correlations between thyroid activity, food intake and carbohydrate intake.
To evaluate the effect of caloric restriction and dietary composition on circulating T3 and rT3, obese subjects were studied after 7–18 days of total fasting and while on randomized hypocaloric diets (800 kcal) in which carbohydrate content was varied to provide from 0 to 100% calories. In contrast, the same subjects receiving isocaloric diets containing at least 50 g of carbohydrate showed no significant changes in either T3 or rT3 concentration. We conclude that dietary carbohydrate is an important regulatory factor in T3 production in man. Isocaloric diets containing at least 50g carbs are diets with the same number of calories, which is 800 kcals (in this case). To assess the effect of starvation and refeeding on serum thyroid hormones and thyrotropin (TSH) concentrations, 45 obese subjects were studied after 4 days of fasting and after refeeding with diets of varying composition. I do agree with their conclusion but I do not seem to empathize with their urgency to raise T3. We know from biochemistry textbooks that T3 works like a catalyst, increasing or decreasing the speed with which chemical reactions take place all over the body. Serum levels of T3 and T4 are inter-connected because T4 (thyroxine) flows in much higher concentrations in the blood and is converted to T3 (active form of thyroid hormone) whenever needed. Metabolic responses during a very-low-calorie diet, composed of 50 per cent glucose and 50 per cent protein, were studied in 18 grossly obese subjects (relative weights 131-205 per cent) for 28 d.
During the first 14 d (period 1) a low T3-high rT3 state developed; there was an inverse relationship between the absolute fall of the plasma T3 concentrations and the cumulative negative nitrogen balance as well as the beta-hydroxybutyrate (BOHB) acid concentrations during the semi-starvation period, pointing to a protein and fuel sparing effect of the low T3 state. Thus, 74 per cent of the extra weight loss in the T3 treated group could be accounted for by loss of fat free tissue. Our results warrant the conclusion that there appears to be no place for T3 as an adjunct to dieting, as it enhances mostly body protein loss and only to a small extent loss of body fat.
Dietary carbohydrate content is a major factor determining endocrine and metabolic regulation. We measured thyroid hormone levels, resting energy expenditure (by indirect calorimetry) and urinary nitrogen excretion in six healthy males after 11 days of three isocaloric diets containing 15% of energy equivalents as protein and 85%, 44% and 2% as carbohydrates.
In contrast to the high and intermediate carbohydrate diets, carbohydrate deprivation decreased plasma T3 values (1·78 ± 0·09 and 1·71 ± 0·07 vs. Eucaloric carbohydrate deprivation increases protein catabolism despite decreased plasma T3 levels. It’s a feeding regimen where subjects are given food to meet their daily requirements, but these diets are restricted in carbohydrates. This is a very challenging study as it shows a certain relationship between carbohydrate intake and T3 levels. If they would have kept going with the protocol, I suspect that natriuresis would have slowed down.
The low carb diet did lower T3 levels compared to the other diets but did not decrease TSH levels, which show correlation between T3 and carbohydrate when subjects are fed eucaloric diets. These findings indicate that the ketotic state induced by the EKD was well tolerated in lean subjects; nitrogen balance was regained after brief adaptation, serum lipids were not pathologically elevated, and blood glucose oxidation at rest was measurably reduced while the subjects remained euglycemic.
In this case, when they were given higher protein intake (1.75g per kg of bodyweight), there was no major nitrogen loss. Energy expenditure was measured in a group of 7 subjects who received two isocaloric isonitrogenous diets for a period of 9-21 days with a 4-10-day break between diets. The respiratory quotient was measured 2-4 hours after a meal during each metabolic period for the calculation of total energy expenditure by the doubly labeled water method. Total T3 was significantly lower and free thyroid index and T3 uptake were significantly higher at the end of the high fat diet in comparison to the high-carbohydrate diet. There is correlation between carbs and T3 levels but a higher TEE may yield more ROS (reactive oxygen species). In addition, all subjects participated in thrice weekly submaximal exercise sessions at 60% of maximal aerobic capacity.
This study shows correlation between T3 and carb content, as well as T3 and calorie restriction. The objective of the study was to assess the relationship between long-term CR with adequate protein and micronutrient intake on thyroid function in healthy lean weight-stable adult men and women.
Serum total and free T(4), total and free T(3), reverse T(3), and TSH concentrations were the main outcome measures.
Long-term CR with adequate protein and micronutrient intake in lean and weight-stable healthy humans is associated with a sustained reduction in serum T(3) concentration, similar to that found in CR rodents and monkeys.
I find this very interesting mostly because of the duration of the study as well as their conclusion. I would suspect that this shows that lower T3 is not indicative of a malfunctioning thyroid, but it still remains unclear whether this is true or not. It is well known that physiological changes in the neuroendocrine system may be related to the process of aging. Our data revealed several differences in the neuroendocrine and metabolic status of centenarians, compared with other age groups, including the lowest serum concentrations of leptin, insulin and T3, and the highest values for prolactin.
We conclude that the neuroendocrine status in centenarians is markedly different from that found in early elderly or young women. Other studies that I have read show differences between cortisol levels in various aging groups. The hypothalamo-pituitary-thyroid axis has been widely implicated in modulating the aging process.
We have compared serum thyroid hormone function parameters in a group of middle-aged offspring of long-living nonagenarian siblings and a control group of their partners, all participants of the Leiden Longevity Study.
Compared with their partners, the group of offspring of nonagenarian siblings show a lower thyroidal sensitivity to thyrotropin. To better understand the connection between thyroid activity and carbohydrate intake, one must have knowledge of how this gland functions and how its function is tied to processes throughout the entire body. These studies show stronger correlation between the thyroid active hormone T3 and carbohydrate intake (higher-carb => higher T3) and they also show correlation between caloric intake and T3 levels. Complete lack of thyroid secretion usually causes the basal metabolic rate to fall 40 to 50 per cent below normal, and extreme excesses of thyroid secretion can increase the basal metabolic rate to 60 to 100 per cent above normal. Although the rate of protein synthesis is increased, at the same time the rate of protein catabolism is also increased. Intermittent fasting will most likely decrease T3 levels, but it should not affect TSH levels and T4 levels. To prevent hypothyroidism, goiter and dwarfism, countries throughout the world have added iodine to salt (iodized salt). As I said, I will lay down a complete Thyroid Protocol (Rx) as well as a Testosterone Protocol (Rx).


This has been a very long post and if you’ve made it so far, please share your thoughts in either of the comment sections below. I tried this post yesterday, not sure why it doesn't show as posted, Here we go again.After two years not running. Does anyone has an idea on how to trouble shoot the temperature sensor in this car?Since there is no computer or codes to compile from it. My carb was rebuilt two years ago and not used till now, it should be in good conditions unless some gunk built up.
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Critics of low-carb nutrition advocate that keto diets lower the thyroid activity, decrease testosterone levels and affect other hormones throughout the body. As anticipated, total fasting resulted in a 53% reduction in serum T3 in association with a reciprocal 58% increase in rT3.
The decline in serum T3 during the no-carbohydrate diet correlated significantly with blood glucose and ketones but there was no correlation with insulin or glucagon.
In contrast, rT3, concentration is not significantly affected by changes in dietary carbohydrate. So, is T3 not having a strong correlation with low calorie diets, but with the amount of carbs in the diet? All subjects showed an increase in both serum total and free thyroxine (T4), and a decrease in serum total and free triiodothyronine (T3) following fasting. The serum T3 declined during fasting even when the subjects were given oral L-T4, but not when given oral L-T3. It may be just my own interpretation, but I do not think low T3 is bad, as long as the thyroid function is normal and as long as the signaling TSH -> TRH and T4 concentration work properly. Isn’t it normal that lower or no food intake leads to lower T3 levels, as long as everything else is in normal boundaries?
In fasted states, as there is not much need of high T3, there will be higher levels of circulating T4 (as it’s not converted). During the last 14 d (period 2) eight subjects (Gp B) served as controls, while the other ten subjects (Gp A) in the low T3 state were treated with triiodothyronine supplementation (50 micrograms, 3 times daily).
In the control group there was a sustained nitrogen balance after three weeks; in Gp A the nitrogen losses increased markedly during T3 treatment. During the T3 treatment period no detectable changes occurred regarding plasma triglycerides and plasma free fatty acids (FFA) concentrations; the plasma BOHB acid concentrations decreased significantly as compared to the control group. The aim of this study was to evaluate the relation between thyroid hormone levels and metabolic parameters during eucaloric carbohydrate deprivation. Because it has previously been shown that starvation decreases plasma T3 levels, resting energy expenditure and nitrogen excretion, these discordant endocrine and metabolic changes following carbohydrate deprivation indicate that the effects of starvation on endocrine and metabolic regulation are not merely the result of carbohydrate deprivation.
For example, if my TEE (total energy expenditure) is 2,500kcals and I eat a eucaloric carbohydrate deprived diet, I will eat 2,500kcals with macronutrient partitioning de-favoring carbs.
After following the low carb diet for 11 days, subjects show increased urinary nitrogen excretion.
To suppress this effect, I would have increased protein intake to 20-30% of total calories and I believe that endogenous protein breakdown would have slowed down. This was followed by four weeks of a eucaloric ketogenic diet (EKD)–isocaloric and isonitrogenous with the EBD but providing less than 20 g CHO daily.
Compared to the previous study, the subjects were given fair amount of protein to start becoming adapted to a high-fat-very-low-carb regimen. Levels of total T3 (TT3), T3 uptake, free thyroid index and T4 were measured at the end of each metabolic period. These data suggest that individual tolerance to a high-fat diet varies considerably and may significantly lower TEE by changing levels of physical activity. The response of these parameters was also examined when subjects switched from the VLCD to a mixed hypocaloric diet. Following VLCD treatments, participants consumed a 1,000 kcal mixed diet while continuing the exercise program for one week. Serum T3 and rT3 of both groups returned to baseline concentrations following one week of the 1,000 kcal diet.
When on the high-carb diet, the subjects show lower T3 levels, but not as low as the ones when they follow a low-carb diet, thus strengthening the correlation between carbohydrate intake and T3 levels.
It has been hypothesized that a reduction in T(3) hormone may increase life span by conserving energy and reducing free-radical production. This effect is likely due to CR itself, rather than to a decrease in body fat mass, and could be involved in slowing the rate of aging. They point out the previously mentioned stronger correlation between T3 levels and carbohydrate intake but as you can see, TSH and T4 remain similar among the groups regardless of the different caloric intake, carbohydrate intake and overall design of the groups’ protocols. To assess neuroendocrine status in aging humans we studied a group of 155 women including 78 extremely old women (centenarians) aged 100-115 years, 21 early elderly women aged 64-67 years, 21 postmenopausal women aged 50-60 years and 35 younger women aged 20-50 years. Centenarians show lower T3, insulin, leptin, and prolactin compared to younger subjects, while there are no differences in TSH and cortisol levels.
It’s interesting that NPY were highest in the elderly because as far as I know NPY (neuropeptide Y) stimulates feeding and promotes lipogensis. Life extension effects associated with low thyroid hormone levels have been reported in multiple animal models. These findings suggest that the favorable role of low thyroid hormone metabolism on health and longevity in model organism is applicable to humans as well.
Subjects restricting calories (hypocaloric diet) have lower circulating T3 levels regardless of the type of macronutrient partitioning.
If this were true all people fasting would suffer from hypothyroidism and many of us would have goiters.
Without proper iodine intake your thyroid may not function normally and this is the case where you’d see lower T3 levels as well as Lower T4 and TSH.
Brownstein believes only 10% of iodine from iodized salt is absorbed, the protocol of iodized salt managed to eradicate the epidemic of hypothyroid related disorders for several decades. I would instead eat seafood (kelp, seaweed) as it is rich in this extremely important nutrient.
Again, to make sure there is good function of the thyroid gland, I would ensure appropriate iodine intake.
However, I’m still conducting active research and experimentation on both of them and will let you know as soon as I finish them.
The car went to the shop, they put back the old distributor and claimed the warm start valve was not working properly.
Bet you’re wondering what YOU can snack on when the rest of the family is enjoying verboten foods.


Register to discover and share recipes with other bloggers and readers passionate about cooking. Subjects receiving the no-carbohydrate hypocaloric diets for two weeks demonstrated a similar 47% decline in serum T3 but there was no significant change in rT3 with time.
Our data suggest that the rise in serum rT3 during starvation may be related to more severe caloric restriction than that caused by the 800 kcal diet. T3 is adjusted according to the needs of the body and it’s implicated in the feedback mechanism that goes between the hypothalamus (especially the regulation of food intake) and the thyroid.
After fasting, the serum reverse T3 (rT3) rose, the serum TSH declined, and the TSH response to thyrotropin-releasing hormone (TRH) was blunted.
I believe 11 days is not enough time to totally shift to being a fat-adapted person and rely mostly on fat oxidation for energy.
The explanation for changes in thyroid hormone levels independent of changes in metabolic rate remains unclear.
The subjects may not have been able to adapt to the diet because of its short duration of only a few days.
However, this still does not mean that lower T3 levels are sign of a disease state or that something is wrong in the body. Plasma NPY, leptin, glucose, insulin and lipid profiles were evaluated, and serum concentrations of pituitary, adrenal and thyroid hormones were measured. On the other hand, LH and FSH levels were comparable with those in the elderly and postmenopausal groups, but they were significantly higher than in younger subjects. In human populations, an association was observed between low thyroid function and longevity at old age, but the beneficial effects of low thyroid hormone metabolism at middle age remain elusive.
The mental processes are excited, and the activities of most of the other endocrine glands are increased.
Effect of dietary composition on fasting-induced changes in serum thyroid hormones and thyrotropin. Intermittent fasting dietary restriction regimen negatively influences reproduction in young rats: a study of hypothalamo-hypophysial-gonadal axis. Metabolic and psychological response to 7-day fasting in obese patients with and without metabolic syndrome.
Warm start valve just redirect the fumes from gas in the carb bowl into the intake air chamber where the air filter is. And I have a couple of reasons to believe so, but I will talk about it in The Testosterone R(x), which I will be releasing in the near future. Refeeding with either a mixed diet (n = 22) or a carbohydrate diet (n = 8) caused the fasting-induced changes in serum T3, T4, rT3, and TSH to return to control values. However, where glucose is not available, it will promote gluconeogenesis by breaking down lean tissue (see first pic). Although dietary carbohydrate content did not influence resting energy expenditure, carbohydrate deprivation increased urinary nitrogen excretion (10·91 ± 0·67 and 12·79 ± 1·14 vs. Their body was still looking for sugar after 11 days on very-high-fat-very-low carb diet, hence the breakdown of lean tissue (natriuresis). Weight and whole-body potassium estimated by potassium-40 counting (40K) did not vary significantly during the five-week study.
Resting and postprandial resting metabolic rate were measured by open circuit indirect calorimetry 2-4 times during each metabolic period. Thus, although dietary carbohydrate content had an influence on the magnitude of fall in serum T3, RMR declined similarly for both dietary treatments.
Caloric restriction in humans: impact on physiological, psychological, and behavioral outcomes. The temperature gauge reading is more likely a thermostat that is either rated too cold or is stuck open. In contrast, refeeding with protein (n = 6) did not cause an increase in serum T3 or in serum TSH of fasted subjects, while it did cause a decline in serum rT3 toward basal value.
It would be unnatural to supplement with T3 for lower T3 levels under caloric restricted regimens. Total energy expenditure (TEE) was measured by the doubly labeled water method over an 8-13-day period.
Metabolic responses in grossly obese subjects treated with a very-low-calorie diet with and without triiodothyronine treatment. Effect of long-term calorie restriction with adequate protein and micronutrients on thyroid hormones.
Comparison of a very low-calorie-ketogenic diet with a standard low-calorie diet in the treatment of obesity. Isocaloric carbohydrate deprivation induces protein catabolism despite a low T3?syndrome in healthy men. The car does not go full warm, ever, the temp readings are always 2 to 3 mm from the cold line. The human metabolic response to chronic ketosis without caloric restriction: physical and biochemical adaptation. Low serum free triiodothyronine levels mark familial longevity: the Leiden Longevity Study. Moderate weight loss is sufficient to affect thyroid hormone homeostasis and inhibit its peripheral conversion. The effect of varying carbohydrate content of a very-low-caloric diet on resting metabolic rate and thyroid hormones. Intermittent Fasting Induces Hypothalamic Modifications Resulting in Low Feeding Efficiency, Low Body Mass and Overeating. So most of the times it stalled like 4 to 7 blocks away, yesterday it was parked in a big forest like park. I am not sure at all if it is running out of gass or just flooded with gas, when I opened the carb last week to setup the needle high it was (as always) super wet with gas on the bottom of the piston that holds the needle (Zenith Stromberg CV carb).Could a super cold engine make things that un-efficient? Bad sensor?Could a cold carb have the bi-fold metal cold start valve been reacting to temperature and making the car run rich forever? Check tranny fluid levels, (my transmission is leaking so I need to source a gasket for it and repair it, until then I keep topping it with anti leak fluid and transmission fluid) Check coolant (I guess All I can do is add some?
The gas test gave me back twice this mileage, but I am not sure if they get this from the gases or just a data log of the car model and year. Even when still poor I can work with that since it is the nature of this beast.Any advise will be golden at this point, I am willing to take it to the shop again, but I cant really afford it. But then I need the car for my work and can not afford another one with more unknown issues.



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