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According to a recent study, the genetic variability of a worldwide sample of type I herpes simplex viral strains (HSV-1) provides independent support for the Out-of-Africa hypothesis for human origins—and along with it, added credibility for the biblical account of humanity’s beginnings.
To better grasp the relationship between virulence and genetic make up, the researchers sequenced and compared the entire genomes (consisting of about 152,000 base pairs, or genetic letters) for 31 different HSV-1 strains collected from around the world.
The geographic distribution and time scale for the origin of the various viral strains corresponds closely to the results of human genetic variability studies that track the origin and spread of humanity around the world. Anthropologists interpret the results of these studies as support for the Out-of-Africa hypothesis. Though often presented and discussed within the context of the evolutionary paradigm, this model has profound biblical implications.
The agreement between the genetic variability pattern of the HSV-1 strains and human genetic variability provides independent support for the Out-of-Africa model (and consequently for the biblical account of humanity’s beginnings).
As researchers continue to study our origin using the human genome and the genomes of human surrogates, such as HSV-1, the scientific credibility of Genesis 1 and 2 continues to advance. Reasons to Believe is a ministry devoted to integrating science and faith and to demonstrating how the latest science affirms our faith in the God of the Bible. The mission of local chapters is to strengthen and equip fellow believers for productive dialogue with doubters and skeptics. Get equipped with powerful new reasons to believe and enjoy content-rich apologetic material delivered to you, wherever you are.
Herpes simplex type 1: A herpes virus that causes cold sores and fever blisters in and around the mouth. Sign up to stay informed with the latest womens health updates on MedicineNet delivered to your inbox FREE!
Genital herpes is a disease caused by the herpes simplex virus (HSV), of which there are two types.


HSV-2 cannot survive long on a non-living surface, so there is no real risk of getting it from a toilet seat or hot tub, for example.
This journal is a member of and subscribes to the principles of the Committee on Publication Ethics. This time of the year a lot of attention is focused on why some viral strains can be so deadly while others elicit a relatively minor response.
The scientists discovered that the viral strains clustered into six groups that corresponded to different parts of the world.
Briefly, this model (also called the replacement model) maintains that modern humans evolved recently (about 100,000 years ago) in East Africa from a small hominid population and then migrated around the world to replace pre-existing hominids. If humanity’s genesis happened in the way Scripture describes it, then genetic diversity patterns observed among people groups around the world would be very similar to those discovered by anthropologists. Your donation helps our ministry take this life-changing message to skeptics around the world while encouraging and strengthening the faith of Christians. When it's active, it travels to the surface of the infected area (skin or mucous membrane) and makes copies of itself. One reason may be that the virus can infect a woman's genitals more easily than it can a man's. HSV-1 can also spread from the mouth to the genitals during oral sex (fellatio, cunnilingus, analingus). But just as HSV-1 can infect the genitals and cause genital herpes, HSV-2 can pass from one person's genitals to another person's mouth, resulting in oral herpes. Following a lytic infection of the peripheral epithelium, HSV-1 enters sensory neurons and is trafficked by retrograde axonal transport to the sensory ganglia.
Researchers have also been able to trace the origin and migration patterns of humanity using several other types of human pathogens and parasites.


This is called "shedding" because these new viruses can, at this time, rub off on another person. Genital herpes is more common among blacks than it is among whites, and it becomes more common as people age. A latent infection then ensues where gene expression from the viral genome is limited to the LATs. In the past, HSV-1 was not known to cause genital herpes, but that is changing, especially among people who begin having sex at a young age. Then the virus travels back down the nerve to a ganglion (mass of nerve tissue), usually at the base of the spine, where it lies dormant for a while. Although a PNS neuron is depicted, it is generally accepted that the events surrounding the formation of a latent infection in neurons of the PNS and the CNS (the focus of this paper) are similar. The tegument is surrounded by a lipid membrane containing many viral glycoproteins that are important for attachment and penetration (2) of cell membranes. After penetration of the virion into the cell cytoplasm it has been seen in EM studies that the tegument can drift away from the capsids. Alternatively a cellular protein (Zhangfei) may inhibit the ability of Vp16 to activate the IE genes.44 With lack of gene expression or DNA replication the viral genome becomes progressively covered with nucleosomes and forms stable chromatin (6). Long-term HSV vectors utilize this promoter by inserting a complementary DNA for the gene to be expressed in front of the LAT promoter.



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