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Signs herpes outbreak is coming

The connection between chronic genital herpes (HSV-2) and HIV risk has grown ever stronger.
There is a strong biological rationale that genital ulcers open a portal through the protective outer mucosal layers, These inflammatory zones, in addition, contain a concentration of the types of immune cells infected by HIV (see, for example, Donaghy et al. At ISSTDR, the HIM (Health in Men) gay male cohort from Sydney led off the conference with unsettling findings (Fengyi et al.
The herpes lesion biopsy shows a loss of the outer skin layer and invasion by immune cells. AdvertisementThe accumulation of STDs in high-risk populations may confuse the observed associations.
The need for successful, continual HSV-2 suppression to protect against HIV transmission was stressed by another report at ISSTDR (Mark et al.
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A 2006 meta-analysis of previous studies noted that preexisting HSV-2 infection tripled the risk of acquiring HIV in men and women generally (Freeman et al. Still, the HIM results imply that you have to test for and treat HSV-1 as well as HSV-2 (not to mention other STDs) to have an effect on HIV transmission. The trial's main goal is to see whether acyclovir reduces HIV acquisition, but it will also check on medication adherence and HSV-2 suppression. The Body and its logos are trademarks of Remedy Health Media, LLC, and its subsidiaries, which owns the copyright of The Body's homepage, topic pages, page designs and HTML code.

The reverse is also quite likely true: genital herpes coinfection can increase infectiousness of persons who already have HIV.
HIM observed a significant association between HSV-1 and HIV infection, but not between HSV-2 and HIV. But there weren't enough HIV cases within this subpopulation to make the results statistically significant. Here, daily acyclovir showed no significant benefit over placebo in reducing levels of either genital HIV or genital HSV-2.
Twenty-five HSV-2+ adults had their genital secretions checked four times a day for 60 days. The chances of statistically significant results are enhanced by the larger trial population, which greatly helps to break down the results by adherence. General Disclaimer: The Body is designed for educational purposes only and is not engaged in rendering medical advice or professional services. University of Washington researchers reported two years ago that genital tract HIV levels increased during periods of HSV-2 reactivation and were related to the quantity of herpes virus present (Baeten et al. The big surprise this summer emerged from the initial large studies of herpes suppression, in both HIV positive and HIV negative women from northern Tanzania. There's a chance that the trial will confirm that acyclovir is not the right drug for this task. The 95% CI is the range within which the ratios are likely to occur 95% of the time, due to the impact of chance fluctuations.

The information provided through The Body should not be used for diagnosing or treating a health problem or a disease. AIDS 2007) published at the time of the conferences calculated that 42% of the HIV contracted by Ugandan women and 65% of that contracted by Zimbabwean women could have been avoided in the absence of HSV-2. At ISSTDR, the same group reported on the effect of the anti-herpes drug valacyclovir on 20 otherwise untreated Peruvian HIV positive women. The HIV negative study is the first to test whether a continuous anti-herpes regimen would reduce the likelihood of contracting HIV in people already infected with genital herpes (Watson-Jones et al.
The greater number of people in a trial, the narrower the 95% CI, and the more trustworthy the observed results. Both plasma and cervical HIV levels were moderately lower than in women taking placebo, by 45% and 56%, respectively (Baeten et al. Nonetheless, its role in genital ulcer disease is thought to be growing, especially in such populations as MSM and college students (Xu et al. The answer from this study was no: among the 821 women enrolled, the rate of contracting HIV was practically the same whether they received acyclovir (400 mg twice a day) or placebo (above 4 per 100 person-years). A study of 136 HIV positive Burkina Faso women published similar results earlier this year (Nagot et al.

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