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Encephalitis is inflammation of the brain; from Greek enkephalos ("brain") and itis ("inflammation").
Among children, a large number of acute encephalitides are of the type known as demyelinating encephalitis, which may develop as a complication of such viral diseases as measles or chicken pox or as a result of vaccination against such viral diseases as smallpox.
Symptoms common to most types of encephalitis are fever, headache, drowsiness, lethargy, coma, tremors, and a stiff neck and back. Characteristic neurological signs include uncoordinated and involuntary movements, weakness of the arms, legs, or other portions of the body, or unusual sensitivity of the skin to various types of stimuli.
Our patient was an immunocompetent 72-year-old woman with a personal history of arterial hypertension and high cholesterol levels which were treated with diet alone. Diagnosis is important since the condition may result in severe sequelae if not treated early. Herpes simplex encephalitis (HSE) is a neurologically devastating illness associated with substantial morbidity and mortality in adults and older children.
The correct recognition of HSE, and distinguishing it from other forms of encephalitis, brain abscess or mimicking disorders early in the course of evaluation facilitates treatment decisions when neurologic complications occur.
The combination of clinical characteristics, magnetic resonance imaging of the head, and polymerase chain reaction (PCR) analysis of cerebrospinal fluid for herpes simplex type 1 DNA is a sensitive and specific way of diagnosing HSE for proper treatment.
Whether prolonged therapy with oral antiviral treatment after standard intravenous acyclovir offers any advantage from the standpoint of neurologic morbidity or mortality is unproven. PCR-negative typical cases and PCR-positive atypical cases of HSE can occur and require treatment with acyclovir. JavaScript is currently disabled, this site works much better if you enable JavaScript in your browser.

It may come as no surprise that large, energy-rich countries like the United States use the most energy, but how much per person? This map includes energy consumption data from the International Energy Agency and includes electricity, as well as fossil and gathered fuels. In some cases, the inflammation affecting the brain may involve adjoining structures as well; there is inflammation of the brain and of the spinal cord in encephalomyelitis and of the brain substance and of the meninges (the membranes covering the brain) in meningoencephalitis.
Although the influenza virus was suspected, the causative agent of sleeping sickness was never established. It is so-called because damage is not done to the nerve cell body but to the insulation (myelin sheath) surrounding the nerve fibres.
The symptoms, signs, and an examination of the cerebrospinal fluid can usually establish the presence of encephalitis, but they do not necessarily establish the cause, which often remains unknown.
The symptoms remaining after recovery from the acute phase of brain inflammation vary considerably, depending on the type of encephalitis and on the age and general health of the patient. Registrese en Elsevier y obtendra: informacion relevante, maxima actualizacion y promociones exclusivas.
Ramirez-Bermudez et al.4 described 83 patients diagnosed with viral encephalitis whose main symptoms were neuropsychiatric alterations. Considering that there is no evidence of the benefits of acyclovir administered prophylactically, further studies are needed to assess this indication in patients with a history of herpes virus encephalitis or those undergoing intracranial surgery at locations where the virus can persist or disseminate. Encephalitis is most often caused by the direct or indirect action of an infective organism and sometimes by such noninfective agents as chemicals. This situation makes specific treatment difficult, and even when the causative virus is known, there may be no drugs effective against it.

Many individuals are weak and ebilitated after an attack but recover with no serious aftereffects. Axial T1-weighted and coronal T2-weighted MR images show a hyperintense lesion on the right temporal lobe and insular cortex. Although treatment with acyclovir at high doses was not started until 20 days after surgery, the patient progressed favorably. Median time to symptom onset after surgery is 6 days,9 which together with the non-specific symptoms in encephalitis can resemble the most typical postoperative complications. These findings resembled those described in the literature.4 Subsequent measurements of PCR in CSF confirmed the diagnosis.
This is an old but elegant study not likely to be repeated which examines whether viral isolates of the trigeminal ganglion and brain in the same patient are identical or not. Laboratory analysis performed 15 days after surgery revealed moderate hyponatraemia, which was attributed to a syndrome of inappropriate antidiuretic hormone secretion.
Twenty days after the surgery, she started to present visual hallucinations and seizures predominantly affecting the left side of the body.

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