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Herpesvirus: One of a family of double-stranded DNA viruses that cause common infections in humans. Fibroblasts express OvHV-2 capsid protein in vasculitis lesions of American bison (Bison bison) with experimental sheep-associated malignant catarrhal fever. Nelson DD, Taus NS, Schneider DA, Cunha CW, Davis WC, Brown WC, Li H, O'Toole D, Oaks JL.SourceDepartment of Veterinary Microbiology and Pathology, Washington State University, Pullman, WA 99164-7040, USA.
American bison (Bison bison) are particularly susceptible to developing fatal sheep-associated malignant catarrhal fever (SA-MCF) caused by ovine herpesvirus-2 (OvHV-2), a ?-herpesvirus in the Macavirus genus. Epstein-Barr Virus Infection Induces Aberrant TLR Activation Pathway and Fibroblast-Myofibroblast Conversion in Scleroderma.


Farina A, Cirone M, York M, Lenna S, Padilla C, McLaughlin S, Faggioni A, Lafyatis R, Trojanowska M, Farina GA.Source1] Rheumatology Section, Department of Medicine, Boston University School of Medicine, Boston, MA, USA [2] Institute Pasteur-Fondazione Cenci Bolognetti, Department of Experimental Medicine University of Rome Sapienza, Rome, Italy. Scleroderma (SSc) is a complex and heterogeneous connective tissue disease mainly characterized by autoimmunity, vascular damage and fibrosis that mostly involve the skin and lungs. Epstein-Barr Virus Latent Membrane Protein 2 Effects on Epithelial Acini Development Reveal Distinct Requirements for the PY and YEEA motifs.
Epstein-Barr virus (EBV) is a gamma herpesvirus associated with numerous cancers including the epithelial cancers nasopharyngeal carcinoma (NPC) and gastric carcinoma. All members of this family are double-stranded DNA viruses with large complex genomes that replicate in the nucleus of the cell.


Because replication occurs inside the host nucleus, herpesviruses can use the host's transcription machinery and DNA repair enzymes to support a large genome with complex arrays of both essential or so-called dispensable genes.
The dispensable genes enhance the cellular environment for virus production, defend the virus from the host immune system and promote cell to cell spread. The large numbers of dispensable genes are in reality required for a productive in vivo infection.



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