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Human herpesvirus 6: A herpes virus that apparently lies dormant in many people, human herpesvirus 6 is most likely to cause problems when the immune system is compromised by disease, as in AIDS patients, or by deliberate immune suppression, as in organ transplant patients. Yoshida, S., Viremia and neutralizing antibody response in infants with exanthem subitum, J. Agut, H., Several groups among human herpesvirus 6 strains can be distinguished by Southern blotting and polymerase chain reaction, J. Salahuddin, Z., Human herpesvirus-6 (Hhv-6, Hblv) in sarcoidosis and lymphoproliferative disorders, J. Ritz, J., Phenotypic and functional deficiency of natural killer cells in patients with chronic fatigue syndrome, J. Cherry, D., Roseola infantum (exanthem subitum), Textbook of Pediatric Infectious Diseases, 2nd ed.
Cassai, E., Characterization of human herpesvirus 6 strains isolated from patients with exanthem subitum with or without cutaneous rash (letter), J. Minamishima, Y., Evidence for putative immediate early antigens in human herpesvirus 6-infected cells, J. Campadellifiume, G., Polyvalent and monoclonal antibodies identify major immunogenic proteins specific for human herpesvirus 7-infected cells and have weak cross-reactivity with human herpesvirus 6, J. Group, The chronic fatigue syndrome: A comprehensive approach to its definition and study, Ann.
Arno, J., Two groups of human herpesvirus 6 identified by sequence analyses of laboratory strains and variants from Hodgkin’s lymphoma and bone marrow transplant patients, J. Yoshida, T., Isolation of human herpesvirus-6 from an infant with Kawasaki disease (letter), Eur.

There have been no reports of human herpesvirus-6 (HHV-6) encephalitis treatment based on both HHV-6 DNA load and the antiviral agent’s concentration in the cerebrospinal fluid (CSF). A 20-year-old male with a hematological malignancy developed HHV-6 encephalitis 15 days after unrelated cord blood transplantation (UCBT).
Serial quantification of HHV-6 DNA in CSF may be useful for successful treatment with GCV in post-transplant HHV-6 encephalitis. References1.Sakai R, Kanamori H, Motohashi K, Yamamoto W, Matsuura S, Fujita A, Ohshima R, Kuwabara H, Tanaka M, Fujita H, Maruta A, Ishigatsubo Y, Fujisawa S. JavaScript is currently disabled, this site works much better if you enable JavaScript in your browser. This publication on HHV-6 is the first and so far only one where a composite picture of the virus is presented. C., Human herpesvirus 7 is a T-lymphotropic virus and is related to but significantly different from herpesvirus 6 and human cytomegalovirus, Proc.
L., A chronic illness characterized by fatigue, neurologic and immunologic disorders, and active human herpes-virus type 6 infection, Ann. L., Prevalence of herpesvirus, human T-lymphotropic virus type I, and treponemal infection in Southeast Asian refugees, J. C., Suppression of human immunodeficiency virus type I replication by human herpesvirus-6, J. A., Detection of human herpesvirus 6 and human papillomavirus 16 in cervical carcinoma, Am. E., The seroepidemiology of human herpesvirus-6 (Hhv-6) from a case-control study of leukaemia and lymphoma, Int.

Long-term outcome of human herpesvirus-6 encephalitis after allogeneic stem cell transplantation. Comprehensive accounts of HHV-6 biology, molecular virology, epidemiology and their association with diseases, including the effects of antiviral drugs, are covered in chapters contributed by leading researchers on HHV-6. The book is an excellent source of reference on HHV-6 for basic researchers, diagnostic virologists, clinicians, medical students and medical librarians since it provides the most current state-of-the-art information on HHV-6.
Magnetic resonance imaging (MRI) of the head revealed abnormal high-intensity signals in the left limbic system on T2-weighted and diffusion-weighted images.
Effect of antivirals on human herpesvirus 6 replication in hematopoietic stem cell transplant recipients. Treatment is experimental at this time, but anti-viral drugs or beta interferon may be tried. Urinary excretion of ganciclovir contributes to improvement of adenovirus-associated hemorrhagic cystitis after allogeneic bone marrow transplantation. Human herpes virus-6 encephalitis in a paediatric bone marrow recipient: successful treatment with pharmacokinetic monitoring and high doses of ganciclovir. Ganciclovir prophylaxis of cytomegalovirus infection and disease in allogeneic bone marrow transplant recipients. Determination of antiviral efficacy against lymphotropic herpesviruses utilizing flow cytometry.

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