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Signs herpes outbreak is coming

Herpes viruses are known to infect skin cells as well as cells lining the cervix and the genital tract. As part of their investigation of Akt’s role in herpes infections, the researchers took laboratory cultures of those human cell types and mixed them for 15 minutes with four different drugs known to inhibit Akt. The paper "HSV activates AKT to trigger calcium release and promote viral entry: novel candidate target for treatment and suppression" was published online by The FASEB Journal. Herpes is a viral disease that causes painful blisters on the genitals and the surrounding areas.
Scientists, according to Science News, have created the vaccine by attacking a viral surface protein called glycoprotein D (gD-2), The herpes virus uses this protein to enter host cells.

The finding, published online by The FASEB Journal, could lead to new drugs for treating or suppressing herpes virus infections. Herold and her colleagues had previously shown that infection by the herpes viruses depends on calcium released within the cells. By deleting gD-2 from the viral genome, researchers have rendered the virus unable to infect cells. In this study, they found that calcium release occurs because the viruses activate a critical cell-signaling molecule called Akt at the cell membrane.
In addition, the vaccine triggers the body’s own immune cells to produce antibodies that recognize different viral targets.

By contrast, cells not pretreated with the Akt inhibitors were readily infected on exposure to the virus. The research was supported by grants from the National Institutes of Health (AI-061679) and the Center for AIDS Research at Einstein and Montefiore Medical Center (AI-51519).

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