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By age 40, nearly 90% of adults have been exposed to the herpes simplex virus-1 (HSV-1), which causes cold sores. Until now, research has generally concentrated on keeping HSV1 inactive — and preventing cold sores from ever showing up.
Understanding how to override the microRNA could allow scientists to activate the virus and then kill it using acyclovir.
The theory is that by activating the virus, then preventing it from returning to hibernation, which is when researchers think it gains strength, it can be completely eradicated.
Cullen and his colleagues, as well as several biotech firms, are already investigating such treatments and how best to deliver them. The patient will see the swelling at the fever blister site along with the development of little, hard and pimple-like protuberances that are highly painful. The tiny hard elevations now swell, burst open and coalesce to make up a huge open up ulcer. In this stage, the ulcer becomes dry and will scab in the areas where the lesion is not kept moist by the saliva from the oral cavity. In the healing stage, a number of scabs are formed on the lesion which subsequently flakes off. While still in the final stage of their life cycle the fever blister can tend to come back. Pay attention and be careful as the cold sore virus is very infectious from the first irritation the complete absence of the fever blister signs and symptoms. Cold sores are not just restricted to the mouth and lips but can also occur on nose, eyes and even hands. Hello, I Have a cold sore on my upper lip, I had a headache a few days ago, that was sevier, along with a cold that im trying to get over, Anyhow, I Got some Abreva, and that seemed to make it 10 times worse, so it spread a little, I Also got some of the Lypsyl, Extreme cold sore relief, And it seems to be working, Is there anything else that i can do to keep getting it to go away, Also how long until its not contagious Anymore! Herpes of the oral form is infection by “herpes simplex virus” and is an infection which causes sores on the gums, lips, roof of the mouth, inside of cheeks, as well as tongue. Starting at the site of infection, this virus can move to the nerve tissue in the spine known as “dorsal root ganglion”. As soon as encountered by stresses, either physical or emotional, this virus can reactivate and develop new symptoms and sores.


But the Duke researchers took the opposite tack: figuring out precisely how to switch the virus from latency to its active stage.
Cullen believes that a drug could be developed to block the microRNA that suppress HSV-1 into latency; once it's active, acyclovir can be used to destroy the virus permanently. It will scab over with a brownish crush and is accompanied by itching or burning sensation.
The blisters eventually rupture as painful open sores, develop a yellowish membrane before healing, and disappear within 3 - 14 days. Rarely, the infection may be accompanied by difficulty in swallowing, chills, muscle pain, or hearing loss. For those who do, however, cold sores are a painful and permanent nuisance, always erupting in the same location, at the original site of infection on the lips or mouth.
In its latent stage, HSV-1 produces a single molecular product, called latency-associated transcript RNA, or LAT RNA. Cullen suggests that this new research may also eventually be applied to other latent viruses, such as herpes simplex virus-2 (HSV-2), which causes genital herpes, or the chicken pox virus, which causes shingles in adults. Fever, running nose and head pains are other common symptoms associated with this point of the stage.
The fever blisters can easily spread from one person to another or from one part of the body of the infected person to another site in the same patient.
A day later I started getting dried cracks on the lower lip and tingling sensation on upper lip. Worse yet, once you're infected with the virus that causes them, you're stuck with it for good. Once HSV-1 enters the body it hunkers down for life, most of the time hiding dormant in the cranial nerves near the spine. Unlike most messenger RNA, LAT RNA doesn't produce proteins, so scientists have never been able to determine LAT RNA's exact function.
Cullen warns that some patients, especially those suffering genital herpes, may have to take acyclovir on a regular basis (HSV-2 is a hardier virus), but for people with HSV-1, the virus could be eradicated with just one dose.
Ulcer stage which is also known as weeping stage is the most painful and contagious of all the stages.


But landmark research reported today by microbiologists at Duke University may offer the potential for a cure. The virus can be triggered by outside stress, such as exposure to sunlight, a fever or emotional distress.
But by inserting the LAT RNA into mice, Cullen found that it breaks down into even smaller strands called microRNA. Before it becomes visible, the skin starts getting tightened in the area where the cold sore is about to be formed. The skin may remain slightly pink or red.The symptoms like headaches, fever and edematous lymph glands start healing by now. After it's active and a cold sore appears, it's treatable with the drug acyclovir, marketed under the name Zovirax, which kills replicating HSV-1.
Researchers then discovered that it was the microRNA that blocked production of the protein that activates HSV-1. The lymphatic nodules are quite well edematous as felt under the surface of the lower jaw which might be quite painful. Itching and irritation of the cold sore are still experienced even though the healing process continues.
But the mystery has been how to eliminate the virus while it's hiding, before it produces unsightly symptoms.
The infection can spread even at this stage so the patient should avoid any kind of physical contact. This marks the first stage of the cold sores as the virus is heading towards the outer surface layer of the neural tissue cells. Healing continues underneath the new skin which is a constant source of tingling and redness present in the area for another week or more.



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