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Phase 2 The internalized EB, within a membrane-bound compartment, immediately begins differentiation into the reticulate body (RB).
Phase 3 As the RBs replicate, the inclusion grows as well to accommodate the increasing numbers of organisms. Phase 1Gonorrhea starts as a surface infection of the mucous membranes found in the urethra, endocervix and anus. Phase 2Attachment of bacteria to the microvillus of non-ciliated columnar epithelial cells is required for colonization during infection. Phase 3The bacteria enter the columnar cells by the process called parasite-directed endocytosis in which the bacteria are engulfed by the columnar cell membrane, forming a vacuole. Phase 4The vacuole is subsequently carried to the basement membrane of the host columnar cell, where the bacteria multiply after being released into the subepithelial tissue by the process of exocytosis. Phase 1 After successful infection of a basal cell, the virus expresses E1 and E2 proteins, which are for replicating and maintaining the viral DNA as a circular episome. Phase 2 Basal stem cells in the epithelial basement layer can maintain papillomavirus genomes for decades. Phase 3 The viral oncogenes E6 and E7 promote cell growth by inactivating the tumor suppressor proteins p53 and pRb. L1 A single viral protein, known as L1, is necessary and sufficient for formation of a 60 nanometer capsid composed of 72 star-shaped capsomers (see figure). L2 The papillomavirus capsid also contains a viral protein known as L2, which is less abundant. The 33-year-old has starred in films like "Ishq Vishk", "Welcome to Sajjanpur", "Vivah" and "Satyagraha". Please do not use derogatory comments in reply to the stories published on Bollywood, India-Forums.
So kindly, refrain from creating an unhealthy atmosphere for all, and keep your comments constructive.


Also, please use the Rate It button if you want to just thank the author for posting the story. Disclaimer: All Logos and Pictures of various Channels, Shows, Artistes, Media Houses, Companies, Brands etc. EB's have rigid outer membranes that help them survive for limited amounts of time outside of their host cells. RB's are the intracellular forms of Chlamydia that are non-infectious but are metabolically active. Through unknown mechanisms, RBs begin a differentiation program back to the infectious EBs, which are released from the host cell to initiate a new round of infection.
Attachment of the bacterium is aided by fimbriae, which are short hair-like appendages, or pili. The mucous membrane is destroyed resulting in the accumulation of Neisseria gonorrhoeae and neutrophils in the subepithelial connective tissue. HPV can remain in the infected person's cells for an indefinite time--most often in a latent state, but occasionally producing symptoms or disease if the immune system weakens.
The expression of the viral late genes, L1 and L2, is exclusively restricted to the host basal cells as they begin to divide and become increasingly differentiated in the upper layers of the epithelium.
It is thought that the HPV virion infects the basal cells of epithelial tissues through micro-abrasions.
Although not clear how L2 is arranged within the virion, it is known to perform several important functions, including facilitating the packaging of the viral genome into nascent virions as well as the infectious entry of the virus into new host cells. Fimbriae are primarily composed of oligomeric pilin proteins that are used to attach the bacterium to the cells of the mucous membrane surface. Virions can then be sloughed off in the dead squames of the host epithelium and the viral lifecycle continues. Interactions between L1 and sulfated sugars on the cell surface promote initial attachment of the virus.


The virus is able to get inside from the cell surface via interaction with a specific receptor, likely via the alpha-6 beta-4 integrin and transported to membrane-enclosed vesicles called endosomes.
Each T cell is uniquely equipped to deal with all types of pathogens such as a virus, bacteria or toxin. Once the EB attaches to a susceptible host cell, it mediates its own internalization through pathogen-specified mechanisms that allow for the recruitment of actins with subsequent engulfment of the bacterium. The outer membrane protein PII then helps the bacterium tightly bind and invade the host cell. The papillomavirus genome is a double-stranded circular DNA molecule 8,000 base pairs in length. The capsid protein L2 disrupts the membrane of the endosome, allowing the viral genome to escape and traffic, along with L2, to the cell nucleus.
According to published data, the inclusion has no interactions with the endocytic pathway and apparently inserts itself into the exocytic pathway as it retains the ability to intercept sphingomyelin-containing vesicles.
It is packaged within the L1 shell along with cellular histone proteins, which serve to wrap and condense DNA. There are three types of professional APCs which include: B cells, macrophages and dendritic cells. Examples of non-professional APCs include: fibroblast, thymic epithelial cells, thyroid epithelial cells, glial cells, pancreatic beta cells and vascular endothelial cells.



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