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Genistein is classified as a phytoestrogen which are plant-derived nonsteroidal compounds that possess estrogen-like biological activity.
Many of the often-cited genotoxic effects of genistein, including apoptosis, cell growth inhibition, topoisomerase inhibition and others become less obvious. In consideration of levels of dietary genistein uptake and bioavailability we have defined in vitro concentrations of genistein >5 microM as non-physiological, and thus "high" doses. Disclaimer : The above statements have not been evaluated by the Food and Drug Administration. Your use of this website constitutes acknowledgement and acceptance of our Terms & Conditions.

Nonfermented soy foods, such as tofu, contain higher levels of the genistein glycosides, while fermented soy foods, such as tempeh and miso, contain higher levels of the aglycone. Genistein has been found to have both weak estrogenic and weak anti-estrogenic effects.Genistein has weak estrogenic activity as measured in in vivo and in vitro assays.
It is a scavenger of reactive oxygen species and inhibits lipid peroxidation.It also inhibits superoxide anion generation by the enzyme xanthine oxidase.
Recent cellular, epigenetic and microarray studies are beginning to decipher genistein effects that occur at dietarily relevant low concentrations. However, we don't know whether genistein (or even soy isoflavones generally) are responsible for this effect.

In vivo,its estrogenic activity is one-third that of glycitein and four times greater than that of daidzein. In addition, genistein, in animal experiments, has been found to increase the activities of the antioxidant enzymes superoxide dismutase, glutathione peroxidase, catalase and glutathione reductase. In toxicology, the well accepted principle of "the dose defines the poison" applies to many toxicants and can be invoked, as herein, to distinguish genotoxic versus potentially beneficial in vitro effects of natural dietary products such as genistein.

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