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Oral herpes is easy to diagnose when a patient has visible sores or ulcers, but early stages of herpes on the face and mouth as well as on the genitals are harder to diagnose. Herpes should be distinguished from herpes zoster, chicken pox, impetigo, and drug eruptions. Herpetic sores are classified as heat sores in TCM because the lesions are red and follow febrile disease conditions. As with all treatment protocols, be sure to tailor and modify the basic treatments below to fit your patient's specific constitution, root cause, and symptoms. Treatment principle: Drain Spleen and Stomach damp heat, nourish yin, drain deficient heat . Cash, checks and credit cards are also gratefully accepted if you prefer to donate in person. Pain, emotional upheaval, fatigue, people who are burned out, folks who want to try esoteric acupuncture. Thirdly, this is an oversimplified but essentially accurate explanation of how several antiviral drugs work. A herpes simplex virus attaches to a susceptible host cell (seen on the right), fusing its envelope with the cell membrane, and releasing naked capsids that deliver viral DNA into the nucleus of the host cell, where it initiates the synthesis of viral DNA.


Acyclovir molecules entering the cell are converted to acyclovir monophosphate by virus induced thymidine kinase enzyme. Host-cell enzymes add two more phosphates to form acyclovir triphosphate, which is transported to the nucleus. Shown in red is the cleavage of phosphate from the acyclovir triphosphate by the herpes simplex's own enzymes, the herpes simplex's DNA polymerase enzyme incorporates the acyclovir monophosphate into the growing DNA strand as if it were 2-deoxyguanosine monophosphate (a "G" base). Further elongation of the chain is impossible because acyclovir monophosphate lacks the attachment point necessary for the insertion of any additional nucleotides.
During active disease phases there are blisters on the skin which contain the infectious virus. The acyclovir (as do other antiviral drugs) is mistakenly incorportated into the growing DNA strand. The acyclovir does not have the proper structure to add any additional "A"'s, or "G"'s, or "C"'s, or "T"'s to it, and so the DNA strand cannot continue to elongate or grow.
If I swallow my acyclovir drugs, isn't acyclovir going into all of my cells, and blocking the growth (or replication) of all my cell's DNA, not just the cells infected with virus? Accompanying symptoms may include high fever and swelling of tenderness of the lymph nodes.


Crusting of the lesions begins after three to four days of onset and complete healing within three weeks.
Viruses are transported along the sensory nerves to the nerve cell bodies where they go dormant and reside as long as the host is alive. They may come out of dormancy because of a variety of triggers (one of which is immunosuppressant drugs). Therefore, only those cells that are infected with the virus will have this specific enzyme and convert the original acyclovir into the acyclovir that can be incorporated as a "G" base. When it becomes active the virus multiplies, travels along the axon of the neuron to the nerve terminals in the skin and releases the virus particles.



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    Simpler than antiviral medicines that and you will note that.
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    And the right way to forestall passing the two and I might find some genital areas.