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The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Cold sores -- also called fever blisters -- are a painful infection caused by the herpes simplex virus (HSV).
Although cold sores generally are not serious, the infection may be life-threatening for anyone who has AIDS or whose immune system is depressed by other disorders or medications.
The infection from a cold sore may cause blindness if it spreads to the eye, and meningitis or encephalitis if it spreads to the brain.
Cold sores are usually caused by the herpes simplex virus (HSV) type 1, which is transmitted by such forms of contact as kissing an infected person or sharing eating utensils, towels, or razors. Vesicular stomatitis in a wild-caught juvenile Grauer’s gorilla (Gorilla beringei graueri).
Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions.


Please try searching for the product or category you are seeking or contact us for assistance. These blisters are usually caused by HSV type 1, but can less commonly be due to HSV type 2. Gross lesions, histopathologic examination, transmission electron microscopy, and molecular screening confirmed human herpesvirus type 1 (HSV-1) as the etiologic agent.
Use of trade names is for identification only and does not imply endorsement by any of the groups named above.
They may show up anywhere on your body, but are most likely to appear on the outside of your mouth and lips, your nose, cheeks, or fingers.
A person with a cold sore who performs oral sex on another person can give that person genital lesions with HSV-1. Blisters form, then break and ooze; a yellow crust or a scab develops and eventually sloughs off, revealing new skin underneath.


After the first infection, many people develop antibodies and never have another cold sore. Similarly, a person who performs oral sex on another person with genital HSV-2 lesions can develop oral lesions. The virus may be triggered by certain foods, stress, fever, colds, allergies, sunburn, and menstruation. B) Section of oral mucosa adjacent to a vesiculo-ulcerative lesion exhibits epithelial cell necrosis, cytoplasmic swelling, nuclear chromatin margination (sometimes with discrete Cowdry type A inclusions), and multinucleated syncytia typical of herpesviral cytopathic effects (hematoxylin and eosin stain). C) Electron micrograph of the same lesion demonstrates intranuclear, unenveloped virions ?100 nm in diameter that are budding through the nuclear membrane to form enveloped virions ?140 nm in diameter; morphologic features of both are compatible with a herpesvirus.



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