Asymmetric sensorineural hearing loss causes 2014,earring making supplies india 2014,hearing loss brain tumour boy - PDF Review

1Assistance Professor, Otolaryngology Department, Hamadan University of medical Sciences, Besat Hospital, Hamadan, Iran.
Methods: This case-study research was done on 95 hepatitis-B patients as the case group and 97 normal cases as the control group. Conclusion: The results show that hepatitis-B patients are more prone to hearing loss and that hepatitis B disease can cause hearing loss. Hearing loss is a known complication of viral infections such as coxsackievirus, cytomegalovirus, herpes, influenza, measles, and mumps.
Hearing loss due to viral infection can be unilateral or bilateral, and ranges from partial to severe hearing loss that will often be permanent.4 Viral hepatitis has been shown to be associated with various extrahepatic manifestations. One hundred consecutive patients with hepatitis B (HBS-Ag positive) between August 2005 and October 2009 were chosen for this study. Figure 1: Mean Frequency-specific results in PTA (dB) of the patients with hepatitis B (+HBS-Ag) and the control groups (-HBS Ag) considering the average thresholds in both ears.
Table 1: Mean pure tone average (dB) results of the patients with hepatitis B and control groups.
The known etiologies of acquired sensorineural hearing loss include acoustic trauma, physical trauma, ototoxicity, genetic predisposition, infections, Meniere’s disease, aging, and autoimmune diseases.
The present study demonstrates the association between hepatitis B and sensorineural hearing loss (SNHL) and a need is felt to conduct further studies involving the temporal bones in hepatitis-B patients. 2Otolarygologist, Otolaryngology Department, Baheshty University of Medical Sceinces, Taleghani Hospital. The difference between two groups was statistically significant with p-value less than 0.05. This study suggests that hepatitis B prophylaxis is important in decreasing hepatitis-B involvement and therefore, hearing loss.

The patients were compared with one hundred cases who had undergone hepatitis B examination as the control group. This study was directly reviewed and approved by the ethics committee at Hamedan University of Medical Sciences. Ninety-five patients and 97 non-hepatic B (negative HBS-Ag) cases were enrolled in the study. Autoimmune inner ear disease was first described in 1979 and the disease has become more widely recognized over the last decade. It is suggested that physicians ask for HBS-Ag test for the presence of hepatitis B in patients with bilateral SNHL with unknown etiology, and be aware that hepatitis B prophylaxis is valuable in decreasing hepatitis B involvement and therefore hearing loss. 3Pathologist, Pathology Department, Hamadan University of Medical Sciences, Besat Hospital.
This study was performed to investigate the relation between positive HBS-Ag (hepatitis B disease) and hearing loss.
Polyarteritis nodosa is a life threatening necrotizing vasculitis that affects medium-sized arteries, and its association with hepatitis B virus is well-documented. These two groups underwent complete clinical history, physical and audiologic examinations. Systemic vasculitis such as polyarteritis nodosa may rarely result in a rapidly progressive hearing loss.
A case of PAN with positive Hbs Ag cured by immunosuppressive and antiviral therapies: a case report. Use of the auditory brainstem response testing in the clinical evaluation of the patients with diabetes mellitus.
Cases with history of trauma to the ear or skull, ear infection or tumors, autoimmune diseases and other known etiology hearing loss were excluded from both groups.

In this study, none of the patients had polyarteritis nodosa, vaccination for hepatitis B, or any treatment for hepatitis that affect their hearing thresholds.
Pure-tone audiometry (PTA) and pure tone average of the right and left ears (average between 250, 500, 1000, 2000, 4000 and 8000 Hz) along with speech discrimination score (SDS) were performed for the two groups by a professional audiologist using AC 40, Interacostics, Assen, Danmark.
This means that hepatitis B alone can affect hearing threshold by inner ear involvement although none of the patients had conventional hearing loss (>30 dB). Also, tympanometry was performed to evaluate the acoustic reflex by Amplaid 775, Milan, Italy.
The acoustic reflex thresholds and SDS were normal in both ears for the patient and control groups. In this study, SDS in the audiometric test did not show retrocochlear involvement, but auditory brainstem response (ABR) assessment would have helped to distinguish not only the central pathways but also the brainstem neural integrity.15 Therefore, future direction of study should be to assess the HBS-Ag positive patients with ABR, otoacoustic emission (OAE), and electrocochleography (ECOG) testing for better evaluation of the inner ear, cochlear nerve, brain stem and cerebral neural pathways. Average pure tone of the two groups exhibited a significant difference (p=0.01) as shown in Table 2. Audiological assessment was performed before starting any medical treatment for hepatitis B patients (newly diagnosed as HBS-Ag positive) and hearing loss was considered if the threshold was more than 20 dB. There was no marked pattern for hearing loss in the patient group, although losses were frequently asymmetric.

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