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Research, where brain imaging was used to identify which parts of the brain behave abnormally in tinnitus, has in turn allowed other tinnitus researchers to focus their attention on these areas.
Professor Richard Tyler is an Action on Hearing Loss funded scientist at the University of Iowa in the USA.
Cochlear implants work by electrically stimulating the nerves in the ear and it has been shown that this electrical stimulation can sometimes reduce tinnitus.
The ultimate aim of this project is to inform the design of a cheap and minimally invasive cochlear implant for the treatment of tinnitus and to get cochlear implant companies to invest in the development of such a device.
Their results showed that these optimal settings varied for different people, suggesting cochlear implants for tinnitus will have to be set up individually for each patient.
The researchers also found that the duration of stimulation was important in the effectiveness of the treatment – it became more effective after longer periods of stimulation. Already, several cochlear implant companies have expressed interest in this research and are funding Professor Tyler to continue his work in the hope that an implant to treat tinnitus will be available within the next five years. A gene therapy cure for inherited deafness has been successfully tested and could be offered to patients in under five years.
Researchers restored the hearing of deaf mice by injecting a virus carrying the healthy gene into their inner ears. The research offers new hope to children born with defective genes that cause "profound" hearing loss, leaving them with a permanent handicap that may affect their education and employment prospects. The treatment was tested on two strains of mutant mouse representing different forms of TMC1-related deafness in humans.
The other mouse strain, called "Beethoven", had a less common form of TMC1 deafness caused by only one copy of the paired genes not working. Deaf mice with the recessive defect fully regained their ability to hear after treatment with the TMC1 gene. Placed in a "startle box" and exposed to abrupt, loud noises, they responded in just the same way as healthy mice with no hearing problems. Both TMC1 and TMC2 code for proteins at the tip of sensory hairs in the inner ear that when stimulated convert sound vibrations into nerve signals. A defective TMC1 gene is enough to cause deafness on its own, but the scientists showed that gene therapy with TMC2 could compensate for its loss.


An engineered virus called adeno-associated virus 1, or AAV1, was used to insert the corrective genes.
He added that other forms of genetic deafness may also respond to the same kind of treatment. Overall, severe to profound hearing loss in both ears affects one to three children in every 1,000 born.
Dr Margaret Kenna, a specialist in genetic hearing loss at Boston Children's Hospital, said: "Current therapies for profound hearing loss like that caused by the recessive form of TMC1 are hearing aids, which often don't work very well, and cochlear implants. A spokesman for the National Deaf Children's Society (NDCS) charity said: " There are many different causes of childhood deafness, including genetics, premature birth, infection and illness and we welcome all medical discoveries that contribute to our understanding of these conditions. This website and associated newspapers adhere to the Independent Press Standards Organisation's Editors' Code of Practice. You simply stick the RFID stickeronto your item and then look around with the cellphone looking glass and it circles the location of the item.
CHICAGO (AP) — A 2-year-old girl born without a windpipe now has a new one grown from her own stem cells, the youngest patient in the world to benefit from the experimental treatment.
Images from the web about 2 year old girl gets windpipe made from stem cells, hope you like them. Find us on FacebookRecent CommentsAHMED on Burma ‘ends attacks in Kachin’wisnel on Obama visit to Kenya?
For about 600,000 people, this perception of hearing noise, such as ringing, hissing or roaring, where there’s no outside source for the sound, is so bad that it seriously affects their quality of life and is associated with a high occurrence of depression and sleep disorders.
But progress has been made in recent years in uncovering what lies behind tinnitus, in particular by UK charity Action on Hearing Loss, which, following its merger with Deafness Research UK, is the only UK charity dedicated to funding research into hearing loss. In Australia, research funded by Action on Hearing Loss shows tinnitus is caused by over-activity in the part of the brain that is involved in hearing and that this is caused by damage to the inner ear. His current area of research is to explore whether a cochlear implant could be designed to treat tinnitus. Professor Tyler has been investigating this in more detail to find out what proportion of people who suffer from tinnitus could be helped by such an implant; and to discover which settings would best benefit t tinnitus sufferers. The researchers performed tests to identify the best possible cochlear implant settings for reducing tinnitus.


They also showed that, for some people, an implant with a very short electrode was able to suppress the tinnitus sounds – this is an important breakthrough, as a short electrode is less likely to damage any residual hearing and, potentially, allows an implant to be used to treat a wider range of people, including those who suffer from tinnitus with no associated hearing loss.
For some, the suppression of tinnitus even persisted for a length of time after electrical stimulation stopped.
The scientists focused on one, TMC1, that accounts for between 4% and 8% of cases and plays a central role in hearing by coding for vital inner ear protein.
Children with this kind of "recessive" genetic defect go profoundly deaf from a very young age, usually around two years.
This "dominant" defect causes children to go deaf gradually from about the age of 10 to 15. Their hearing was partially restored, and the treatment was successful at the cellular and brain level. A genetic switch sequence known as a "promoter" was also used to ensure the genes were only activated in the inner ear hair cells. If you have a complaint about the editorial content which relates to inaccuracy or intrusion, then please contact the editor here.
It will display arrows telling you to look to the right, left, up, down, until you get to the location where your keys are located. All I can say is, I can’t wait for this to be real I lose my keys more than enough times around my house. I manage the Charity's social media, blog and e-newsletter and help with promoting digital campaigns.
Well, this futuristic viewing glass is going to use an RFID sticker to track down your lost items.



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