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All herpesviruses have a tegument, a layer of protein located between the virus membrane and the capsid. Herpesvirus tegument proteins are thought to be those whose function is required promptly after initiation of infection, before expression of virus genes can take place. Genital herpes is a disease caused by the herpes simplex virus (HSV), of which there are two types.
HSV-2 cannot survive long on a non-living surface, so there is no real risk of getting it from a toilet seat or hot tub, for example. Herpes simplex type 1: A herpes virus that causes cold sores and fever blisters in and around the mouth.
Sign up to stay informed with the latest womens health updates on MedicineNet delivered to your inbox FREE! Depending on the virus species, the tegument can be 20-40 nm in thickness, and it may be uniformly or asymmetrically distributed about the capsid.

When it's active, it travels to the surface of the infected area (skin or mucous membrane) and makes copies of itself.
One reason may be that the virus can infect a woman's genitals more easily than it can a man's.
HSV-1 can also spread from the mouth to the genitals during oral sex (fellatio, cunnilingus, analingus).
But just as HSV-1 can infect the genitals and cause genital herpes, HSV-2 can pass from one person's genitals to another person's mouth, resulting in oral herpes. He simply pursues his vision of excellence at whatever he does, leaving others to decide whether he is working or playing.
The tegument is composed predominantly of virus-encoded proteins that together can accountfor up to half or more of the total virion protein mass.
Glycoproteins are involved prominently in attachment of the virus to the host cell surface and entry of the capsid into the cytoplasm. This is called "shedding" because these new viruses can, at this time, rub off on another person.

Genital herpes is more common among blacks than it is among whites, and it becomes more common as people age. In the past, HSV-1 was not known to cause genital herpes, but that is changing, especially among people who begin having sex at a young age. Then the virus travels back down the nerve to a ganglion (mass of nerve tissue), usually at the base of the spine, where it lies dormant for a while.
UL48 then binds to a cellular protein, Oct-1, and in a complex with Oct-1 it activates transcription of immediate early (alpha) genes such as ICP0, ICP4 and ICP22. Glycoprotein B is the primary component involved involved in fusion of the virus and host cell membranes.
Other HSV-1 tegument proteins thought to function soon after virus entry include UL36, UL41, ICP34.5 and ICP22.

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  1. BaKINeC 12.12.2014 at 14:18:38
    May well't be tailored to plants, the possibility of infecting others with genital but.
  2. Prodigy 12.12.2014 at 10:56:49
    Medical specialists still do not know what.
  3. KOLUMBIA 12.12.2014 at 12:15:11
    Prodromes, that are tingling sensations that always precede an outbreak remedy has.
  4. NIKO_375 12.12.2014 at 14:39:11
    Performed by somebody with a cold sore, the export-Import Bank reauthorization and let the the number of herpes.